检索结果-南通市图书馆

Meta analysis of application of acupuncture combined with drug anesthesia in anorectal surgery

Journal of Hainan Medical University 2020年第18期26卷 40-45页

作者： Jia-Bin Liang Yue Liu Xin-Jun Liu Yang Wei Wei-Chuan Kuang Yan-Qing Lu Department of Acupuncture and rehabilitation Guangzhou University of Chinese Medicine 510405 guangdong provincial Second Traditional Chinese Medicine hospital Department of Acupuncture and Rehabilitation 510095

Objective:To evaluate the effectiveness and safety of acupuncture combined drug anesthesia and simple drug anesthesia in anorectal ***:The randomized controlled trials of acupuncture combined with drug anesthesia was evaluated using Cochrane systematic review *** search scope was China National Knowledge Internet,wanfang database,China Biomedical Literature,Pubmed and *** deadline was December 31,*** randomized controlled trials of acupuncture combined with anesthesia that met the inclusion criteria were *** terms of trial quality evaluation,Cochrane risk bias was used for ***,RevMan 5.2 software was used for statistical ***:This study finally included 15 articles,a total of 1321 *** heterogeneity of significant rate was good,and the combined effect amount OR was 3.55(95%CI[2.06,6.12]).The combined effect quantitative test(Z=4.57,P<0.00001)showed that there was a statistical significance,indicating that acupuncture combined drug anesthesia was better than simple anesthesia in anorectal *** heterogeneity of adverse reactions was good,and the combined effect test(Z=8.88,P<0.00001)showed that there was a statistical significance,indicating that the adverse reaction rate of acupuncture combined with drug anesthesia was lower than that of simple drug anesthesia in the anorectal ***:Compared with simple drug anesthesia,acupuncture combined with drug anesthesia can improve the anesthesia effect and reduce the occurrence of adverse reactions in anorectal surgery.

关键词： Acupuncture anesthesia Anorectal surgery Randomized control Review Systematic review

来源：

维普期刊数据库评论

在线全文

维普期刊数据库

学校读者我要写书评

暂无评论

Nanotopographic micro-nano forces finely tune the conformation of macrophage mechanosensitive membrane protein integrinβ_(2)to manipulate inflammatory responses

引用

Nano Research 2023年第7期16卷 9715-9729页

作者： Yuanlong Guo Yong Ao Chen Ye Ruidi Xia Jiaomei Mi Zhengjie Shan Mengru Shi Lv Xie Zetao Chen hospital of Stomatology Sun Yat-sen University and Guangdong Provincial Key Laboratory of StomatologyGuangzhou 510055China Department of Microbiology Zhongshan School of MedicineSun Yat-sen UniversityGuangzhou 510080China

Finely tuning mechanosensitive membrane proteins holds great potential in precisely controlling inflammatory *** addition to macroscopic force,mechanosensitive membrane proteins are reported to be sensitive to micro-nano ***β_(2),for example,might undergo a piconewton scale stretching force in the activation ***-aspect-ratio nanotopographic structures were found to generate nN-scale biomechanical *** with the advantages of uniform and precisely tunable structural parameters,it is fascinating to develop low-aspect-ratio nanotopographic structures to generate micro-nano forces for finely modulating their conformations and the subsequent mechanoimmiune *** this study,low-aspect-ratio nanotopographic structures were developed to finely manipulate the conformation of integrinβ_(2).The direct interaction of forces and the model molecule integrinαXβ_(2)was first *** was demonstrated that pressing force could successfully induce conformational compression and deactivation of integrinαXβ_(2),and approximately 270 to 720 pN may be required to inhibit its conformational extension and *** low-aspect-ratio nanotopographic surfaces(nanohemispheres,nanorods,and nanoholes)with various structural parameters were specially designed to generate the micro-nano *** was found that the nanorods and nanohemispheres surfaces induce greater contact pressure at the contact interface between macrophages and nanotopographic structures,particularly after cell *** higher contact pressures successfully inhibited the conformational extension and activation of integrinβ_(2),suppressing focal adhesion activity and the downstream PI3K-Akt signaling pathway,reducing NF-κB signaling and macrophage inflammatory *** findings suggest that nanotopographic structures can be used to finely tune mechanosensitive membrane protein conformation changes,providing an effective strategy for precisely modulating inflammat

关键词： nanotopographic structures micro-nano forces mechanosensitive membrane proteins protein conformational changes inflammatory responses

来源：

维普期刊数据库评论

在线全文

维普期刊数据库

学校读者我要写书评

暂无评论

Enhanced osteogenesis of titanium with nano-Mg(OH)_(2) film and a mechanism study via whole genome expression analysis

引用

Bioactive Materials 2021年第9期6卷 2729-2741页

作者： Mengyu Yao Shi Cheng Guoqing Zhong Jielong Zhou Hongwei Shao Limin Ma Chang Du Feng Peng Yu Zhang Department of Biomedical Engineering School of Materials Science and EngineeringSouth China University of TechnologyGuangzhou510641China Department of Orthopedics Guangdong Provincial People’s HospitalGuangdong Academy of Medical SciencesGuangzhouGuangdong510080China Shantou University Medical College Shantou515041GuangdongChina

Titanium(Ti)has been the most widely used orthopedic implant in the past ***,their inert surface often leads to insufficient osteointegration of Ti *** solve this issue,two bioactive Mg(OH)_(2) films were developed on Ti surfaces using hydrothermal treatment(Ti-M1#and Ti-M2#).The Mg(OH)_(2) films showed nano-flake structures:sheets on Ti-M1#with a thickness of 14.7±0.7 nm and a length of 131.5±2.9 nm,and on Ti-M2#with a thickness of 13.4±2.2 nm and a length of 56.9±5.6 *** films worked as Mg ions releasing *** the gradual degradation of Mg(OH)_(2) films,weakly alkaline microenvironments will be established surrounding the modified *** from the sustained release of Mg ions,nanostructures,and weakly alkaline microenvironments,the as-prepared nano-Mg(OH)_(2) coated Ti showed better in vitro and in vivo ***,Ti-M2#showed better osteogenesis than Ti-M1#,which can be ascribed to its smaller ***,whole genome expression analysis was applied to study the osteogenic mechanism of nano-Mg(OH)_(2) *** both coated samples,most of the genes related to ECM-receptor interaction,focal adhesion,and TGF-β pathways were upregulated,indicating that these signaling pathways were activated,leading to better ***,cells cultured on Ti-M2#showed markedly upregulated BMP-4 gene expression,suggesting that the nanostructure with Mg ion release ability can better activate BMP-4 related signaling pathways,resulting in better ***-Mg(OH)_(2) films demonstrated a superior osteogenesis and are promising surface modification strategy for orthopedic applications.

关键词： Titanium Surface modification Osteogenesis Whole genome analysis

来源：

维普期刊数据库评论

在线全文

维普期刊数据库

学校读者我要写书评

暂无评论

IGF2BP3-induced activation of EIF5B contributes to progression of hepatocellular carcinoma cells

引用

Oncology Research 2022年第2期30卷 77-87页

作者： XIAOYIN LI QIAN WANG HONGFENG LIANG SHISHENG CHEN HAIWEN CHEN YAOYONG LU CHANGFU YANG Department of Oncology(Section 3) Gaozhou People’s HospitalGaozhouChina South China Research Center for Acupuncture and Moxibustion Medical College of Acu-Moxi and RehabilitationGuangzhou University of Chinese MedicineGuangzhouChina The Second Affiliated hospital of Guangzhou University of Chinese Medicine(guangdong provincial hospital of Chinese Medicine) GuangzhouChina Affiliated Nanfang hospital(Branch Tai He hospital)of Southern Medical University GuangzhouChina

In this study,we investigated the functional role of eukaryotic initiation factor 5B(EIF5B)in hepatocellular carcinoma(HCC)and the underlying *** analysis demonstrated that the EIF5B transcript and protein levels as well as the EIF5Bcopy number were signiﬁcantly higher in the HCC tissues compared with the non-cancerous liver ***-regulation of EIF5B signiﬁcantly decreased proliferation and invasiveness of the HCC ***,EIF5B knockdown suppressed epithelial-mesenchymal transition(EMT)and the cancer stem cell(CSC)***-regulation of EIF5B also increased the sensitivity of HCC cells to 5-ﬂuorouracil(5-FU).In the HCC cells,activation of the NF-kappa B signaling pathway and IkB phosphorylation was signiﬁcantly reduced by EIF5B ***2BP3 increased the stability of the EIF5B mRNA in an m6A-dependent *** data suggested that EIF5B is a promising prognostic biomarker and therapeutic target in HCC.

关键词： EIF5B NF-jB Epithelial-mesenchymal transition Cancer stem cells N6-methyladenosine Hepatocellular carcinoma

来源：

维普期刊数据库评论

在线全文

维普期刊数据库

学校读者我要写书评

暂无评论

Multi-platform omics analysis reveals molecular signature for COVID-19 pathogenesis,prognosis and drug target discovery

引用

Signal Transduction and Targeted Therapy 2021年第5期6卷 1535-1545页

作者： Yuming Li Guixue Hou Haibo Zhou Yanqun Wang Hein Min Tun Airu Zhu Jingxian Zhao Fei Xiao Shanwen Lin Dongdong Liu Dunrong Zhou Lang Mai Lu Zhang Zhaoyong Zhang Lijun Kuang Jiao Guan Qiushi Chen Liyan Wen Yanjun Zhang Jianfen Zhuo Fang Li Zhen Zhuang Zhao Chen Ling Luo Donglan Liu Chunke Chen Mian Gan Nanshan Zhong Jincun Zhao Yan Ren Yonghao Xu State Key Laboratory of Respiratory Disease National Clinical Research Center for Respiratory DiseaseGuangzhou Institute of Respiratory Healththe First Affiliated Hospital of Guangzhou Medical UniversityGuangzhouChina BGI-Shenzhen ShenzhenChina The Sixth Affiliated hospital of Guangzhou Medical University Qingyuan People's HospitalQingyuanGuangdongChina HKU-Pasteur Research Pole School of Public HealthLi Ka Shing Faculty of MedicineThe University of Hong KongHong Kong SARChina Department of Infectious Diseases Guangdong Provincial Key Laboratory of Biomedical ImagingGuangdong Provincial Engineering Research Center of Molecular ImagingThe Fifth Affiliated HospitalSun Yat-sen UniversityZhuhaiGuangdongChina Yangjiang People's hospital YangjiangGuangdongChina Yangjiang Center for Disease Control and Prevention YangjiangGuangdongChina Guangzhou Eighth People's hospital of Guangzhou Medical University GuangzhouGuangdongChina Guangzhou Customs District Technology Center GuangzhouChina

Disease progression prediction and therapeutic drug target discovery for Coronavirus disease 2019(COVID-19)are particularly important,as there is still no effective strategy for severe COVID-19 patient ***,we performed multi-platform omics analysis of serial plasma and urine samples collected from patients during the course of *** analyses of these omics data revealed several potential therapeutic targets,such as ANXA1 and *** changes in plasma indicated dysregulation of macrophage and suppression of T cell functions in severe patients compared to those in non-severe ***,we chose 25 important molecular signatures as potential biomarkers for the prediction of disease *** prediction power was validated using corresponding urine samples and plasma samples from new COVID-19 patient cohort,with AUC reached to 0.904 and 0.988,*** conclusion,our omics data proposed not only potential therapeutic targets,but also biomarkers for understanding the pathogenesis of severe COVID-19.

关键词： pathogenesis urine drug

来源：

维普期刊数据库评论

在线全文

维普期刊数据库

学校读者我要写书评

暂无评论

Infection of wild-type mice by SARS-CoV-2 B.1.351 variant indicates a possible novel cross-species transmission route

引用

Signal Transduction and Targeted Therapy 2022年第1期7卷 258-269页

作者： Ting Pan Ran Chen Xin He Yaochang Yuan Xiaohui Deng Rong Li Haiping Yan Shumei Yan Jun Liu Yiwen Zhang Xiantao Zhang Fei Yu Mo Zhou Changwen Ke Xiancai Ma Hui Zhang Institute of Human Virology Key Laboratory of Tropical Disease Control of Ministry EducationGuangdong Engineering Research Center for Antimicrobial Agent and ImmunotechnologyZhongshan School of MedicineSun Yat-sen University510080 GuangzhouGuangdongChina Center for Infection and Immunity Study School of MedicineShenzhen Campus of Sun Yat-sen University518107 ShenzhenGuangdongChina Department of Gastroenterology The Eighth Affiliated HospitalSun Yat-sen University518033 ShenzhenGuangdongChina Sun Yat-sen University Cancer Center State Key Laboratory of Oncology in South ChinaCollaborative Innovation Center for Cancer Medicine510060 GuangzhouGuangdongChina guangdong provincial People's hospital Guangdong Academy of Medical Sciences510080 GuangzhouGuangdongChina guangdong provincial Center for Disease Control and Prevention 511430 GuangzhouGuangdongChina National Guangzhou Laboratory Bio-Island510320 GuangzhouGuangdongChina

COVID-19 is identified as a zoonotic disease caused by SARS-CoV-2,which also can cross・transmit to many animals but not *** modifications of SARS-CoV-2 or mice enable the mice susceptible to viral *** neither is the natural situation,they are currently utilized to establish mouse infection *** we report a direct contact transmission of SARS-CoV-2 variant B.1.351 in wild-type *** SARS-CoV-2(B.1.351)re plicated efficiently and induced significant pathological changes in lungs and tracheas,accompanied by elevated proinflammatory cytokines in the lungs and ***,the receptor-binding domain(RBD)of SARS-CoV-2(B.1.351)spike protein turned to a high binding affinity to mouse angiotensin-converting enzyme 2(mACE2),allowing the mice highly susceptible to SARS-CoV-2(B.1.351)*** work suggests that SARS-CoV-2(B.1.351)expands the host range and therefore increases its transmission route without adapted *** the wild house mice live with human populations quite closely,this possible transmission route could be potentially *** addition,because SARS-CoV-2(B.1.351)is one of the major epidemic strains and the mACE2 in laboratory-used mice is naturally expressed and regulated,the SARS-CoV-2(B.1.351)/mice could be a much convenient animal model system to study COVID-19 pathogenesis and evaluate antiviral inhibitors and vaccines.

关键词： elevated utilized adapted

来源：

维普期刊数据库评论

在线全文

维普期刊数据库

学校读者我要写书评

暂无评论

Design of single-phased magnesium alloys with typically high solubility rare earth elements for biomedical applications:Concept and proof

引用

Bioactive Materials 2023年第4期22卷 180-200页

作者： Dong Bian Xiao Chu Jin Xiao Zhipei Tong He Huang Qinggong Jia Jianing Liu Wenting Li Hui Yu Yue He Limin Ma Xiaolan Wang Mei Li Tao Yang Wenhan Huang Chi Zhang Mengyu Yao Yu Zhang Zhigang Xu Shaokang Guan Yufeng Zheng Medical Research Center Guangdong Provincial People’s HospitalGuangdong Academy of Medical SciencesGuangzhou510080China Department of Orthopedics Guangdong Provincial People’s HospitalGuangdong Academy of Medical SciencesGuangzhou510080China School of Materials Science and Engineering Zhengzhou UniversityZhengzhou450001China Academy for Advanced Interdisciplinary Studies Peking UniversityBeijing100871China School of Materials Science and Engineering Peking UniversityBeijing100871China ERC for Revolutionizing Metallic Biomaterials North Carolina A&T State UniversityGreensboroNC27411USA

Rare earth elements(REEs)have been long applied in magnesium alloys,among which the mischmetal-containing WE43 alloy has already got the CE mark approval for clinical application.A considerable amount of REEs(7 wt%)is needed in that multi-phased alloy to achieve a good combination of mechanical strength and corrosion ***,the high complex RE addition accompanied with multiple second phases may bring the concern of biological ***-phased Mg-RE alloys with simpler compositions were proposed to improve the overall performance,i.e.,“Simpler alloy,better performance”.The single-phased microstructure can be successfully obtained with typical high-solubility REEs(Ho,Er or Lu)through traditional smelting,casting and extrusion in a wide compositional range.A good corrosion resistance with a macroscopically uniform corrosion mode was guaranteed by the homogeneously single-phased *** bimodal-grained structure with plenty of sub-grain microstructures allow us to minimize the RE addition to<1 wt%,without losing mechanical *** single-phased Mg-RE alloys show comparable mechanical properties to the clinically-proven Mg-based *** exhibited similar in-vitro and in-vivo performances(without local or systematic toxicity in SD-rats)compared to a high purity *** addition,metal elements in our single-phased alloys can be gradually excreted through the urinary system and digestive system,showing no consistent accumulation of RE in main organs,i.e.,less burden on *** novel concept in this study focuses on the simplification of Mg-RE based alloys for biomedical purpose,and other biodegradable metals with single-phased microstructures are expected to be explored.

关键词： Biodegradable magnesium Rare earth Bimodal grain structure Mechanical property Corrosion resistance

来源：

维普期刊数据库评论

在线全文

维普期刊数据库

学校读者我要写书评

暂无评论

SPDEF suppresses head and neck squamous cell carcinoma progression by transcriptionally activating NR4A1

引用

International Journal of Oral Science 2021年第4期13卷 370-380页

作者： Yanting Wang Xianyue Ren Weiyu Li Ruoyan Cao Suyang Liu Laibo Jiang Bin Cheng Juan Xia hospital of Stomatology Sun Yat-sen UniversityGuangzhouChina guangdong provincial Key Laboratory of Stomatology GuangzhouChina Guanghua School of Stomatology Sun Yat-sen UniversityGuangzhouChina

SAM pointed domain containing E26 transformation-specific transcription factor(SPDEF)plays dual roles in the initiation and development of human ***,the biological role of SPDEF in head and neck squamous cell carcinoma(HNSCC)remains *** this study,the expression level of SPDEF and its correlation with the clinical parameters of patients with HNSCC were determined using TCGA-HNSC,GSE65858,and our own clinical ***8,colony formation,cell cycle analysis,and a xenograft tumor growth model were used to determine the molecular functions of SPDEF in ***-qPCR,dual luciferase reporter assay,and rescue experiments were conducted to explore the potential molecular mechanism of SPDEF in *** with normal epithelial tissues,SPDEF was significantly downregulated in HNSCC *** with HNSCC with low SPDEF mRNA levels exhibited poor clinical *** SPDEF inhibited HNSCC cell viability and colony formation and induced G0/G1 cell cycle arrest,while silencing SPDEF promoted cell proliferation in *** xenograft tumor growth model showed that tumors with SPDEF overexpression had slower growth rates,smaller volumes,and lower *** could directly bind to the promoter region of NR4A1 and promoted its transcription,inducing the suppression of AKT,MAPK,and NF-κB signaling ***,silencing NR4A1 blocked the suppressive effect of SPDEF in HNSCC ***,we demonstrate that SPDEF acts as a tumor suppressor by transcriptionally activating NR4A1 in *** findings provide novel insights into the molecular mechanism of SPDEF in tumorigenesis and a novel potential therapeutic target for HNSCC.

关键词： inhibited SPD NR4A1

来源：

维普期刊数据库

同方期刊数据库评论

在线全文

学校读者我要写书评

暂无评论

Stabilization of MYC G-quadruplex DNA by ruthenium(II)complex overcomes imatinib resistance in chronic myeloid leukemia cells harboring T315I mutation

引用

Genes & Diseases 2023年第2期10卷 399-402页

作者： Yuening Sun Xin Chen Siyan Liao Aochu Liu Huan Zhou Liling Jiang Wa Ding Wenjie Mei Jinbao Liu Xianping Shi Guangzhou Municipal and guangdong provincial Key Laboratory of Protein Modification and Degradation State Key Laboratory of Respiratory DiseaseAffiliated Cancer Hospital of Guangzhou Medical University School of Basic Medical SciencesGuangzhou Medical UniversityGuangzhouGuangdong 510120China guangdong Province Engineering Technology Center for Molecular Probe and Biomedicine Imaging Guangdong Pharmaceutical UniversityGuangzhouGuangdong 510006China

The key pathogenesis of chronic myeloid leukemia(CML)is the formation of BCR-ABL fusion gene,encoding a 210 kDa Bcr-Abl tyrosine kinase,which is crucial for the occurrence and development of ***(IM)is the first targeted anticancer drug approved by FDA for the treatment of CML;however,some patients,especially those in accelerated phase and blastic phase,develop primary or secondary drug resistance to ***,the most challenging resistance is caused by T315I mutation of Bcr-Abl,which represents approximately 15%–20%of all acquired mutations and renders cell resistant to a variety of tyrosine kinase inhibitors.1,2 Thus,there is an urgent need to develop novel strategies to overcome Bcr-Abl T315I-meidated IM resistance.

关键词： myeloid overcome resistance

来源：

维普期刊数据库评论

在线全文

维普期刊数据库

学校读者我要写书评

暂无评论

Stress increases MHC-I expression in dopaminergic neurons and induces autoimmune activation in Parkinson’s disease

引用

Neural Regeneration Research 2021年第12期16卷 2521-2527页

作者： Bao-Yan Wang Yong-Yi Ye Chen Qian Hong-Bo Zhang Heng-Xu Mao Long-Ping Yao Xiang Sun Guo-Hui Lu Shi-Zhong Zhang The National Key Clinical Specialty The Engineering Technology Research Center of Education Ministry of ChinaGuangdong Provincial Key Laboratory on Brain Function Repair and RegenerationDepartment of NeurosurgeryZhujiang HospitalSouthern Medical UniversityGuangzhouGuangdong ProvinceChina Department of Neurosurgery the Second Affiliated Hospital of Guangzhou Medical UniversityGuangzhouGuangdong ProvinceChina Department of Neurosurgery First Affiliated Hospital of Nanchang UniversityNanchangJiangxi ProvinceChina

The expression of major histocompatibility complex class I(MHC-I),a key antigen-presenting protein,can be induced in dopaminergic neurons in the substantia nigra,thus indicating its possible involvement in the occurrence and development of Parkinson’s ***,it remains unclear whether oxidative stress induces Parkinson’s disease through the MHC-I *** the present study,polymerase chain reaction and western blot assays were used to determine the expression of MHC-I in 1-methyl-4-phenylpyridinium(MPP+)-treated SH-SY5Y cells and a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine(MPTP)-induced Parkinson’s disease mouse *** findings revealed that MHC-I was expressed in both *** detect whether the expression of MHC-I was able to trigger the infiltration of cytotoxic T cells,immunofluorescence staining was used to detect cytotoxic cluster of differentiation 8(CD8)+T cell infiltration in the substantia nigra of MPTP-treated *** results indicated that the presentation of MHC-I in dopaminergic neurons was indeed accompanied by an increase in the number of CD8+T ***,in MPTP-induced Parkinson’s disease model mice,the genetic knockdown of endogenous MHC-I,which was caused by injecting specific adenovirus into the substantia nigra,led to a significant reduction in CD8+T cell infiltration and alleviated dopaminergic neuronal *** further investigate the molecular mechanisms of oxidative stress-induced MHC-I presentation,the expression of PTEN-induced kinase 1(PINK1)was silenced in MPP+-treated SH-SY5Y cells using specific small interfering RNA(siRNA),and there was more presentation of MHC-I in these cells compared with control siRNA-treated *** together,MPP+-/MPTP-induced oxidative stress can trigger MHC-I presentation and autoimmune activation,thus rendering dopaminergic neurons susceptible to immune cells and *** may be one of the mechanisms of oxidative stress-induced Parkinson’s disease,and implies the potential

关键词： antigen presentation autoimmune CD8+T cell dopaminergic neuron major histocompatibility complex class I mitochondria neuroinflammation oxidative stress Parkinson’s disease PINK1

来源：

维普期刊数据库

同方期刊数据库评论

在线全文

学校读者我要写书评

暂无评论

欢迎您,

建议与咨询留下您的常用邮箱和电话号码，以便我们向您反馈解决方案和替代方法

时间限定

文献类型

馆藏选择

核心期刊

语言

文献类型

帮助

文字说明：

检索规则说明：

检索范例：

分类表

所选分类

限定检索结果

文献类型

馆藏范围

日期分布

学科分类号

主题

机构

作者

语言

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

请选择保存的检索档案：

请选择收藏分类：

通借通还

欢迎您,

建议与咨询 留下您的常用邮箱和电话号码，以便我们向您反馈解决方案和替代方法

时间限定

文献类型

馆藏选择

核心期刊

语言

文献类型

帮助

文字说明：

检索规则说明：

检索范例：

分类表

所选分类

限定检索结果

文献类型

馆藏范围

日期分布

学科分类号

主题

机构

作者

语言

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

在线全文

请选择保存的检索档案： 新增检索档案 确定 取消

请选择收藏分类： 新增自定义分类 确定 取消

通借通还

建议与咨询留下您的常用邮箱和电话号码，以便我们向您反馈解决方案和替代方法

请选择保存的检索档案：

请选择收藏分类：