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cardiac autonomic function in patients with myasthenia gravis:analysis of the heart-rate variability in the time-domain

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Neuroimmunology and Neuroinflammation 2015年第1期2卷 21-25页

作者： Sherifa Ahmed Hamed Khaled Osama Mohamad Mohamad Adam Department of Neurology and Psychiatry Assiut University Hospital71516 AssiutEgypt Department of Cardiology Assiut University Hospital71516 AssiutEgypt

Aim:Myasthenia gravis(MG)is a neuromuscular transmission disorder caused by acetylcholine receptor *** autonomic dysfunctions were rarely reported in patients with *** cardiac abnormalities were variable and reported in patients at severe stages of the disease and with *** investigated cardiac functions in patients with MG using Ambulatory 24‑h electrocardiographic Holter‑***:This study included 20 patients with MG with a mean age of 28.45±8.89 years and duration of illness of 3.52±1.15 *** standard Holter reports include data for heart‑rate,ventricular ectopies(VEs),supraventricular ectopies(SVEs),heart-rate variability(HRV),ST,QT,atrial fibrillation and T‑wave ***:VEs,SVEs and ST‑T changes were reported in 55%,40%and 20%of patients *** with healthy subjects(n=20),HRV components including SDNN,SDANN,SDNN Index,RMS‑SD and pNN50(P=0.001 for all)were reduced in patients indicating sympathetic and parasympathetic autonomic *** abnormalities were reported in 30-60%of *** significant correlations were identified between SDNN,RMS‑SD,pNN50,and duration of ***:Depressed HRV may be an early manifestation of autonomic neuropathy in patients with MG even in milder stages of the *** information is useful in rating disease progression and the efficacy of therapeutic interventions.

关键词： cardiac autonomic function heart-rate variability myasthenia gravis

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Short-term exposure to ambient ozone associated with cardiac arrhythmias in healthy adults

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Global Health Journal 2022年第1期6卷 6-18页

作者： Lingyan Liu Yutong Zhu Hongbing Xu Yang Wang Tong Wang Qian Zhao Yi Zhang Jie Chen Shengcong Liu Tieci Yi Rongshan Wu Shuo Liu Xiaoming Song Jianping Li Wei Huang Department of Occupational and Environmental Health Sciences School of Public HealthPeking UniversityBeijing 100191China Peking University Institute of Environmental Medicine Peking UniversityBeijing 100191China Key Laboratory of Molecular Cardiovascular Sciences of Ministry of Education Health Science CenterPeking UniversityBeijing 100191China Department of Prevention and Health Care Hospital of Health Science CenterPeking UniversityBeijing 100191 China Institute for Risk Assessment Sciences University Medical Centre UtrechtUniversity of UtrechtP.O.Box 801253508 TC Utrechtthe Netherlands Department of Cardiology Peking University First HospitalBeijing 100034China

Objective The exact biological mechanism whereby exposure to ambient ozone(O3)may contribute to clinical onset of cardiovascular events remains *** this study,we aim to examine the impacts of O3 exposure on cardiac arrhythmias and potential pathways involved through autonomic dysfunction and myocardial *** Seventy-three non-smoking healthy adults were followed with 4 repeated measurements of 24-hour ambulatory arrhythmias,heart rate variability,ST-segment deviation,and blood pressure(BP)in Beijing,China,2014‒*** additive mixed models coupled with distributed lag nonlinear models were constructed to evaluate the associations and potential interlinks between O3 exposure and outcome *** During the study period,24-hour average concentrations of ambient O3 were 47.4µg/m3(ranging from 1.0 to 165.9µg/m3).Increased risks of premature ventricular contraction and ventricular tachycardia were associated with interquartile range increases in O3 exposure during the last 5 days before each participant's clinic visit,with relative risks of 2.14(95%confidence interval[CI]:1.95 to 2.32)and 5.47(95%CI:3.51 to 7.43),*** analyses further showed that sympathetic activation,parasympathetic inhibition,and elevated BP levels,as well as heightened risks of ST-segment depression could mediate up to 47.74%of the risks of arrhythmias attributable to O3 *** Our results suggest that short-term exposure to ambient O3 could prompt the genesis of arrhythmias partially through worsening autonomic function and myocardial burden.

关键词： Ozone cardiac arrhythmias cardiac autonomic function Myocardial injury Panel study

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