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The Second International Conference on Selenium in the Environment and Human Health

作者： Prokisch J Attila S Beata B Peter E Judit P zsofia m mohsen Z Institute of Bio- and Environmental Energetics University of Debrecen Centre for Agricultural Sciences and EngineeringB(o|¨)sz(o|¨)rmenyiut 138.H-4032DebrecenHungary Bionanoferm Ltd.Egyetemter 1 H-4032DebrecenHungary Department of Dairy Science Faculty of AgricultureKafr El-Sheikh UniversityEgypt

Introduction Bioavailability and toxicity of selenium(Se) are strongly related to the chemical form(Hartikainen, 2005).Organic Se forms are less toxic,but are significantly important in functional foods,food and feed supplements because of their increased *** is applied for the industrial production of selenium-methionine from inorganic Se(Rayman,2004).Recent interest in the field of nanotechnology has stimulated research into the chemical synthesis of Se *** on the reduction of selenite to elemental Se by sulphate reducing,Se respiring bacteria was published for first time in the 1990s(Tomeiet al.,1995).

关键词： Selenium and Its Testing for Feed and Food Applications Lactomicrosel Novel Fermentation Technology for Production of Selenium Nanospheres

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Perianal disease,small bowel disease,smoking,prior steroid or early azathioprine/biological therapy are predictors of disease behavior change in patients with Crohn's disease

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World Journal of Gastroenterology 2009年第28期15卷 3504-3510页

作者： Peter Laszlo Lakatos zsofia Czegledi Tamas Szamosi Janos Banai Gyula David Ferenc Zsigmond Tunde Pandur Zsuzsanna Erdelyi Orsolya Gemela Janos Papp Laszlo Lakatos 1st Department of medicine Semmelweis University H-1083 Budapest Hungary Department of Gastroenterology State Health Center H-1062 Budapest Hungary 1st Department of medicine Csolnoky F. County Hospital HH8201-Veszprem Hungary

AIm:To assess the combined effect of disease phenotype, smoking and medical therapy [steroid, azathioprine(AZA), AZA/biological therapy] on the probability of disease behavior change in a Caucasian cohort of patients with Crohn's disease(CD).mETHODS:Three hundred and forty well-characterized, unrelated, consecutive CD patients were analyzed(m/F:155/185, duration:9.4 ± 7.5 years) with a complete clinical *** records including disease phenotype according to the montreal classification, extraintestinal manifestations, use of medications and surgical events were analyzed *** were interviewed on their smoking habits at the time of diagnosis and during the regular follow-up ***:A change in disease behavior was observed in 30.8% of patients with an initially non-stricturing, non-penetrating disease behavior after a mean diseaseduration of 9.0 ± 7.2 *** a logistic regression analysis corrected for disease duration, perianal disease, smoking, steroid use, early AZA or AZA/ biological therapy use were independent predictors of disease behavior *** a subsequent Kaplan-meier survival analysis and a proportional Cox regression analysis, disease location(P = 0.001), presence of perianal disease(P moking(P = 0.032) were independent predictors of disease behavior ***:Our data suggest that perianal disease, small bowel disease, smoking, prior steroid use, early AZA or AZA/biological therapy are all predictors of disease behavior change in CD patients.

关键词：生物治疗硫唑嘌呤疾病类固醇小肠吸烟预测行为

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A Prospective, Randomized Trial to Evaluate the Efficacy of Virtual Reality Treatment Versus Traditional Balance Treatment in Patients with Stroke

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Journal of Sports Science 2016年第3期4卷 150-155页

作者： Zsanett Sipos zsofia Hogye Zsuzsanna Vekerdy-Nagy Department of medical Rehabilitation and Physical medicine Clinical Center University of Debrecen Debrecen 4032 NagyerdeiCircuit 98 Hungary Department of Pediatric Rehabilitation the Bethesda Children Hospital of the Hungarian Reformed Church Budapest 1146Bethesda Street 3 Hungary

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Identification of new genetic risk factors for prostate cancer

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Asian Journal of Andrology 2009年第1期11卷 49-55页

作者： michelle Guy zsofia Kote-Jarai Graham G. Giles Ali Amin Al Olama Sarah K. Jugurnauth Shani mulholland Daniel A. Leongamomlert Stephen m. Edwards Jonathan morrison Helen I. Field melissa C. Southey Gianluca Severi Jenny L. Donovan Freddie C. Hamdy David R Dearnaley Kenneth R. muir Charmaine Smith melisa Bagnato Audrey T. Ardern-Jones Amanda L. Hall Lynne T. O＇Brien Beatrice N. Gehr-Swain Rosemary A. Wilkinson Angela Cox Sarah Lewis Paul m. Brown Sameer G. Jhavar malgorzata Tymrakiewicz Artitaya Lophatananon Sarah L. Bryant The UK Genetic Prostate Cancer Study Collaborators British Association of Urological Surgeons＇ Section of Oncology and The UK ProtecT Study Collaborators Alan Horwich Robert A. Huddart Vincent S. Khoo Christopher C. Parker Christopher J. Woodhouse Alan Thompson Tim Christmas Chris Ogden Cyril Fisher Charles Jameson Colin S. Cooper Dallas R. English John L. Hopper David E. Neal Douglas E Easton Rosalind A. Eeles Section of Cancer Genetics The Institute of Cancer Research Sutton Surrey SM2 5NG UK Cancer Epidemiology Centre The Cancer Council Victoria Carlton VIC 3053 Australia Centre for molecular Environmental Genetic and Analytic Epidemiology The University of Melbourne Carlton VIC 3053 Australia Cancer Research UK Genetic Epidemiology Unit University of Cambridge Strangeways Laboratory Worts Causeway Cambridge CB1 8RN UK Department of Oncology University of Cambridge Strangeways Laboratory Worts Causeway Cambridge CB1 8RN UK Genetic Epidemiology Laboratory Department of Pathology The University of Melbourne Parkville VIC 3052 Australia Department of Social medicine University of Bristol Bristol BS8 2PR UK Academic Urology Unit University of Sheffield Sheffield SIO 2JF UK The Royal marsden NHS Foundation Trust Sutton Surrey SM2 5PT and London SW3 6JJ UK University of Nottingham medical School Queens Medical Centre Nottingham NG7 2UH UK Institute for Cancer Studies University of Sheffield Sheffield SIO 2RX UK Surgical Oncology （Uro-Oncology S4） Departments of Oncology and Surgery University of Cambridge Addenbrooke＇s Hospital Cambridge CB2 2QQ UK A full list of authors is provided in the Supplementary Note online Ka Shing Centre Cambridge CB2 ORE UK Cancer Research UK Cambridge Research Institute Li Ka Shing Centre Cambridge CB2 ORE UK.

There is evidence that a substantial part of genetic predisposition to prostate cancer （PCa） may be due to lower penetrance genes which are found by genome-wide association studies. We have recently conducted such a study and seven new regions of the genome linked to PCa risk have been identified. Three of these loci contain candidate susceptibility genes： mSmB, LmTK2 and KLK2/3. The mSmB and KLK2/3 genes may be useful for PCa screening, and the LmTK2 gene might provide a potential therapeutic target. Together with results from other groups, there are now 23 germline genetic variants which have been reported. These results have the potential to be developed into a genetic test. However, we consider that marketing of tests to the public is premature, as PCa risk can not be evaluated fully at this stage and the appropriate screening protocols need to be developed. Follow-up validation studies, as well as studies to explore the psychological implications of genetic profile testing, will be vital prior to roll out into healthcare.

关键词： prostate cancer genetics susceptibility loci SNPs relative risks

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