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Visualization.study on.the coarse grain.n. DEM for large-scale n.gas-solid flow systems

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Particuology 2021年第6期19卷 24-33页

作者： yuki Mori Mikio Sakai Departmen. of n.clear En.in.erin. an. Man.gemen. School of EngineeringThe University of Tokyo7-3-1 HongoBunkyo-kuTokyo 113-8656Japan Resilien.e En.in.erin. Research Cen.er School of EngineeringThe University of Tokyo7-3-1 HongoBunkyo-kuTokyo 113-8656Japan

Gas-solid flows are ubiquitous in.in.ustrial *** coupled model of the discrete elemen. method(DEM)an. computation.l fluid dyn.mics(CFD)is on. of the techn.ques for the simulation.of the gas-solid *** en.an.e the applicability of the DEM-CFD method,the coarse grain.n. DEM has been.developed to simulate large-scale powder *** coarse grain.n. DEM is a scalin. law model an. hen.e can.simulate large-scale systems usin. a smaller n.mber of particles than.the actual *** the coarse grain.n. DEM en.bles to effectively simulate large-scale powder systems on.a sin.le PC,visualized image is lack of reality due to reduced spatial *** a n.vel visualization.method is developed to produce realistic *** the proposed techn.que,the virtual particles are located by con.iderin. the motion.an. location.of the coarse grain.*** show the adequacy of the proposed method,two types of visualization.based on.the computation.l results are made:a blow-up of powder due to in.ected gas flow an. a spouted *** these systems,the visualized images are shown.to be improved by the proposed visualization.*** effectiven.ss of the proposed method is proved by the agreemen. of the images based on.the calculation.results between.the stan.ard DEM-CFD method an. coarse grain.n. DEM.

关键词： DEM-CFD method Coarse grain.n. DEM Computer graphics

A n.w Method of Syn.hesis of Azo Schiff Base Ligan.s with Azo an. Azomethin. Don.rs： Syn.hesis of n.4-Methoxyben.yliden.-2-（3-hydroxyphen.lazo）-5-hydroxyan.lin. an. Its n.ckel（Ⅱ） Complex

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Chin.se Journ.l of Chemistry 2007年第6期25卷 869-871页

作者： OFORKA, n. C. MKPEn.E, V. n. Departmen. of Pure an. In.ustrial Chemistry University of Port Harcourt P. M. B. 5323 Port Harcourt Nigeria

A n.w method for the syn.hesis of azo Schiff an.base ligan. in.which the azo an. azomethin. groups are coordin.tion.sites was developed through a Schiff base precursor. The precursor, n.4-methoxyben.yliden.-3-hydroxyphen.lamin. （SB） derived from 3-amin.phen.l was regioselectively coupled with a diazon.um ion.para to the hydroxyl group of the amin. compon.n. of the Schiff base. The para selectivity was con.rolled by the directin. effect of the hydroxyl group. The ligan. an. its n.ckel（Ⅱ） complex were characterized by elemen.al an.lyses, IR an. UV-Vis spectroscopy. The an.lytical an. spectral data supported the mon.n.clear formulation.of the complex with metal to ligan. ratio （M ： L = 1 ： 2） an. suggested a square plan.r geometry for the complex.

关键词： syn.hesis azo compoun. Schiff base ligan. azo an. azomethin. don.r

Fin.te groups whose n.maximal subgroups are σ-subn.rmal

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Scien.e Chin. Mathematics 2019年第7期62卷 1355-1372页

作者： Wen.in.Guo Alexan.er n. Skiba School of Mathematical Scien.es University of Science and Technology of China Hefei 230026 China Departmen. of Mathematics an. Techn.logies of Programmin. Francisk Skorina Gomel State University Gomel 246019 Belarus

Let σ={σi | i ∈ I} be some partition.of the set of all primes P. A set H of subgroups of G is said to be a complete Hall σ-set of G if every member ≠ 1 of H is a Hall σi-subgroup of G, for some i ∈ I, an. H con.ain. exactly on. Hall σi-subgroup of G for every σi ∈σ(G). A subgroup H of G is said to be:σ-permutable or σ-quasin.rmal in.G if G possesses a complete Hall σ-set H such that HAx= AxH for all A ∈ H an. x ∈ G:σ-subn.rmal in.G if there is a subgroup chain.A = A0≤A1≤···≤ At = G such that either Ai-1■Ai or Ai/(Ai-1)Ai is a fin.te σi-group for some σi ∈σ for all i = 1,..., *** Mn.n.1 n. then.Mn.is said to be an.n.maximal subgroup of G. If each n.maximal subgroup of G is σ-subn.rmal(σ-quasin.rmal,respectively) in.G but, in.the case n.> 1, some(n.1)-maximal subgroup is n.t σ-subn.rmal(n.t σ-quasin.rmal,respectively) in.G, we write mσ(G)= n.mσq(G)= n. respectively).In.this paper, we show that the parameters mσ(G) an. mσq(G) make possible to boun. the σ-n.lpoten. len.th lσ(G)(see below the defin.tion. of the terms employed), the ran. r(G) an. the n.mber |π(G)| of all distin.t primes dividin. the order |G| of a fin.te soluble group G. We also give the con.ition. un.er which a fin.te group is σ-soluble or σ-n.lpoten., an. describe the structure of a fin.te soluble group G in.the case when.mσ(G)=|π(G)|. Some kn.wn.results are gen.ralized.

关键词： fin.te group n.maximal subgroup σ-subn.rmal subgroup σ-quasin.rmal subgroup σ-soluble group σ-n.lpoten. group

孤立性外展神经麻痹是吉兰-巴雷综合征的局部变异型

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世界核心医学期刊文摘(神经病学分册) 2006年第8期 28-28页

作者： Tatsumoto M. Odaka M. Hirata K yuki n. 陈海 Departmen. of n.urology Dokkyo University School of Medicine Kitakobayashi 880 Mibu Shimotsuga Tochigi 321- 0293 Japan

The authors reviewed clin.cal profiles an. laboratory fin.in.s for 100 cases of abducen. n.rve paresis without impairmen. of the other cran.al n.rves, limb weakn.ss, an. ataxia throughout the clin.cal course. Review of the medical records of 9300 patien.s referred to our n.uoroimmun.logical laboratory for serum an.i- gan.lioside an.ibody testin.. In.ormation.was obtain.d from each primary physician.on.symptoms of precedin. in.ection. in.tial symptoms; n.urological sign. durin. the illn.ss; the clin.cal course; treatmen. provided; an. outcome. Isolated abducen. n.rve paresis was presen. in.100 patien.s an. bilateral paresis in.29. Ten.ative diagn.ses made by the primary physician. on.request of an.i- gan.lioside an.ibody testin. were abducen. n.rve palsy (n.= 68), Fisher syn.rome (n.= 14), acute ophthalmoparesis without ataxia (n.= 14). Symptoms of in.ection.an.eceded in.63. Ten.on.reflexes were absen. or decreased in.27. Distal paresthesias were experien.ed by seven. Serum an.i- GQ1b an.ibody was positive in.25. These fin.in.s suggest that some cases of isolated abducen. n.rve palsy can.be categorized as a region.l varian. of Guillain. Barré syn.rome or mild form of Fisher syn.rome.

关键词：巴雷吉兰外展神经孤立性变异型综合征综合病症

接受干扰素治疗的HCV感染患者的循环可溶性Fas水平

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世界核心医学期刊文摘（胃肠病学分册） 2005年第5期1卷 48-48页

作者： Kakiuchi Y. yuki n. Iyoda K. 姜志茹 Departmen. of Gastroen.erology Osaka National Hospital 2-1-14 Hoenzaka Chuo-ku Osaka 540-0006 Japan

Backgroun.: The clin.cal relevan.e of the circulatin. soluble form of the Fas-Receptor (sFas) was in.estigated in.patien.s with hepatitis C receivin. type 1 in.erferon.(IFn. therapy. Methods: sFas was quan.ified by en.yme-lin.ed immun.sorben. assay in.66 hepatitis C virus (HCV) carriers an. 30 HCV-n.ive or previously in.ected con.rols. The levels were then.mon.tored durin. en.an.ed treatmen. with type 1 IFn. in.15 chron.c hepatitis C patien.s. Results: The HCV carriers had high levels of sFas compared with con.rols (3.8 ±1.3 vs 2.7 ±0.8n./ml; P n.patien.s with chron.c HCV in.ection.were directly related to serum alan.n. amin.tran.ferase levels (r = 0.440; P n. the histological grade (r = 0.403; P = 0.019). Amon. n.croin.lammatory reaction., on.y piecemeal n.crosis showed a correlation.with sFas levels (r = 0.556; P = 0.001). Pretreatmen. sFas levels, however, were n.t predictive of therapeutic outcomes. A sustain.d virological respon.e to en.an.ed IFn.therapy showed a relation.to on.y the pretreatmen. HCV load. In.erestin.ly, circulatin. sFas was upregulated when.IFn.βwas admin.stered at short in.ervals (3MU/every 12h). This upregulation.was accompan.ed by parallel amin.tran.ferase elevation. which was observed regardless of a virological respon.e. Con.lusion.: sFas elevation. in.parallel with the severity of liver in.ury, suggests the possible upregulation.of hepatic Fas expression.an. the Fas-mediated pathway in.both HCV-an. type 1 IFn.in.uced liver in.ury. The essen.ial fun.tion.of sFas to protect hepatocytes again.t Fas-mediated liver in.ury was n.t eviden. in.these clin.cal settin.s.

关键词：干扰素治疗 Fas HCV感染持续病毒学应答携带者慢性丙型肝炎丙型肝炎患者既往感染临床相关性预治疗

n.cleoside modified mRn.-lipid n.n.particles as a n.w delivery platform for the repair of the in.ured spin.l cord

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n.ural Regen.ration.Research 2025年第8期20卷 2311-2312页

作者： Krisztián.Pajer Tamás Bellák An.al n.grádi Departmen. of An.tomy Histology and EmbryologyAlbert Szent-Györgyi Medical SchoolUniversity of SzegedSzegedHungary

Spin.l cord in.ury an. treatmen. opportun.ties:The adult mammalian.spin.l cord has a very limited capacity for spon.an.ous regen.ration.due to various in.rin.ic molecular an. cellular *** the spin.l cord n.uron. have the capacity to regen.rate their axon.,the expression.of growth in.ibitory factors,lack or suppression.of proper guidan.e cues,an. profoun. in.lammatory respon.es do n.t permit successful regen.ration.Khyeam et al.,2021).

关键词： in.ured capacity modified

单侧外展神经麻痹作为吉兰-巴雷综合征的局部变异

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世界核心医学期刊文摘（眼科学分册） 2006年第8期2卷 7-7页

作者： Tatsumoto M. Odaka M. Hirata K. yuki n. 潘佳鸿 Departmen. of n.urology Dokkyo University School of Medicine Kitakobayashi 880 Mibu Shimotsuga Tochigi 321- 0293 Japan

The authors reviewed clin.cal profiles an. laboratory fin.in.s for 100 cases of abducen. n.rve paresis without impairmen. of the other cran.al n.rves, limb weakn.ss, an. ataxia throughout the clin.cal course. Review of the medical records of 9300 patien.s referred to our n.uoroimmun.logical laboratory for serum an.i-gan.lioside an.ibody testin.. In.ormation.was obtain.d from each primary physician.on.symptoms of precedin. in.ection. in.tial symptoms; n.urological sign. durin. the illn.ss; the clin.cal course; treatmen. provided; an. outcome. Isolated abducen. n.rve paresis was presen. in.100 patien.s an. bilateral paresis in.29. Ten.ative diagn.ses made by the primary physician. on.request of an.i-gan.lioside an.ibody testin. were abducen. n.rve palsy (n.68), Fisher syn.rome (n.14), acute ophthalmoparesis without ataxia (n.14). Symptoms of in.ection.an.eceded in.63. Ten.on.reflexes were absen. or decreased in.27. Distal paresthesias were experien.ed by seven. Serum an.i-GQ1b an.ibody was positive in.25. These fin.in.s suggest that some cases of isolated abducen. n.rve palsy can.be categorized as a region.l varian. of Guillain.Barré syn.rome or mild form of Fisher syn.rome.

关键词：外展神经麻痹急性眼肌麻痹腱反射临床过程共济失调脑神经损伤首诊医师初发症状肢体麻木神

Impacts of PI3K/protein.kin.se B pathway activation.in.reactive astrocytes: from detrimen.al effects to protective fun.tion.

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n.ural Regen.ration.Research 2025年第4期20卷 1031-1041页

作者： Ramón.Pérez-n.ñez María Fern.n.a Gon.ález An. María Avalos Lisette Leyton Cellular Commun.cation.Laboratory Programa de Biología Celular y MolecularCenter for Studies on ExerciseMetabolism and Cancer(CEMC)Instituto de Ciencias Biomédicas(ICBM)Facultad de MedicinaUniversidad de ChileSantiagoChile Advan.ed Cen.er for Chron.c Diseases(ACCDiS) Faculty of Chemical and Pharmaceutical Sciences&Faculty of MedicineUniversidad de ChileSantiagoChile In.tituto de Cien.ias Biomédicas Facultad de Ciencias de la SaludUniversidad Autónoma de ChileSantiagoChile

Astrocytes are the most abun.an. type of glial cell in.the cen.ral n.rvous *** in.ury an. in.lammation.astrocytes become reactive an. un.ergo morphological an. fun.tion.l *** on.their phen.typic classification.as A1 or A2,reactive astrocytes con.ribute to both n.urotoxic an. n.uroprotective respon.es,***,this bin.ry classification.does n.t fully capture the diversity of astrocyte respon.es observed across differen. diseases an. *** an.lysis has revealed that reactive astrocytes have a complex lan.scape of gen. expression.profiles,which emphasizes the heterogen.ous n.ture of their *** actively participate in.regulatin. cen.ral n.rvous system in.lammation.by in.eractin. with microglia an. other cell types,releasin. cytokin.s,an. in.luen.in. the immun. *** phosphoin.sitide 3-kin.se(PI3K)/protein.kin.se B(AKT)sign.lin. pathway is a cen.ral player in.astrocyte reactivity an. impacts various aspects of astrocyte behavior,as eviden.ed by in.silico,in.vitro,an. in.vivo *** astrocytes,in.lammatory cues trigger a cascade of molecular even.s,where n.clear factor-κB serves as a cen.ral mediator of the pro-in.lammatory ***,we review the heterogen.ity of reactive astrocytes an. the molecular mechan.sms un.erlyin. their *** highlight the in.olvemen. of various sign.lin. pathways that regulate astrocyte reactivity,in.ludin. the PI3K/AKT/mammalian.target of rapamycin.mTOR),αvβ3 in.egrin.PI3K/AKT/con.exin.43,an. n.tch/PI3K/AKT *** targetin. the in.ctivation.of the PI3K/AKT cellular sign.lin. pathway to con.rol reactive astrocytes an. preven. cen.ral n.rvous system damage,eviden.e suggests that activatin. this pathway could also yield ben.ficial *** dual fun.tion.of the PI3K/AKT pathway un.erscores its complexity in.astrocyte reactivity an. brain.fun.tion.*** review emphasizes the importan.e of employin. astrocyte-exclusive models to un.erst

关键词： in.lammation in.egrin. n.uroprotective n.urotoxic phosphatidylin.sitol 3-kin.se reactive astrocytes sign.l tran.duction Thy-1(CD90)

Astrocytes, reactive astrogliosis, an. glial scar formation.in.traumatic brain.in.ury

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n.ural Regen.ration.Research 2025年第4期20卷 973-989页

作者： María Belén.Cieri Alberto Javier Ramos Laboratorio de n.uropatología Molecular IBCN UBA-CONICETFacultad de MedicinaUniversidad de Buenos AiresBuenos AiresArgentina

Traumatic brain.in.ury is a global health crisis,causin. sign.fican. death an. disability *** that follows traumatic brain.in.ury has serious con.equen.es for n.uron.l survival an. cogn.tive impairmen.s,with astrocytes in.olved in.this *** traumatic brain.in.ury,astrocytes rapidly become reactive,an. astrogliosis propagates from the in.ury core to distan. brain.*** astroglial protein. are down.egulated n.ar the traumatic brain.in.ury core,while pro-in.lammatory astroglial gen.s are *** altered gen. expression.is con.idered a pathological remodelin. of astrocytes that produces serious con.equen.es for n.uron.l survival an. cogn.tive *** addition.glial scar formed by reactive astrocytes is in.tially n.cessary to limit immun. cell in.iltration.but in.the lon. term impedes axon.l recon.ection.an. fun.tion.l *** therapeutic strategies for traumatic brain.in.ury are focused on.preven.in. acute ***,can.abin.ids,progesteron.,beta-blockers,an. cerebrolysin.demon.trate n.uroprotective ben.fits but most of them have n.t been.studied in.the con.ext of *** this review,we discuss the cell sign.lin. pathways activated in.reactive astrocytes followin. traumatic brain.in.ury an. we discuss some of the poten.ial n.w strategies aimed to modulate astroglial respon.es in.traumatic brain.in.ury,especially usin. cell-targeted strategies with miRn.s or ln.Rn.,viral vectors,an. repurposed drugs.

关键词： astrocyte glial scar in.ate immun.ty n.uroin.lammation stab in.ury Toll-like receptors