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A widely used nonionic surfactant with desired functional groups as aqueous electrolyte additives for stabilizing Zn anode

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Rare Metals 2024年第8期43卷 3692-3701页

作者： Yue-Xian Song Xiao-Feng Wang Cai-Bin Liu Kai-xuan guo xu-huan guo Xiao-Bin Zhong Yao-Hui Zhang Kai Wang Hui-Juan guo Li-Xin Zhang Jun-Fei Liang School of Energy and Power Engineering North University of ChinaTaiyuan 030051China Key Laboratory of Green Chemical Process of Ministry of Education Hubei Key Laboratory of Novel Chemical Reactor and Green Chemical TechnologySchool of Chemical Engineering and PharmacyWuhan Institute of TechnologyWuhan 430073China Shanxi Provincial Key Laboratory for High Performance Battery Materials and Devices Taiyuan 030051China

Aqueous Zn-ion batteries(AZIBs) have emerged as potential candidates for Li-ion batteries due to their intrinsic safety and high ***,metallic Zn anodes encounter dendrite growth and water-induced corrosion,rendering poor stability and severe irreversibility at the electrode/electrolyte interface during *** stabilize the Zn anode,we report a low-cost and effective nonionic surfactant,Tween-20 polymer,as an electrolyte additive for *** Tween-20,sequential oxyethylene groups tended to be preferentially adsorbed on the Zn electrode to form a shielding layer for regulating uniform Zn ***,the hydrophobic hendecyl chains prevented H_(2)O-induced corrosion on the Zn anode *** from the desired functional groups,when only trace amounts of Tween-20(0.050 g·L^(-1)) were used,the Zn anode displayed good cycling stability over 2170 h at10 mA·cm^(-2) and a high average Coulombic efficiency of98.94% over 1000 *** Tween-20 polymer can also be effectively employed in MnO_(2)/Zn full *** their toxicity,price and amount of usage,these surfactant additives provide a promising strategy for realizing the stability and reversibility of high-performance Zn anodes.

关键词： Zn anode Dendrite growth Tween-20 polymer Surfactant additive Aqueous electrolyte

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C-X-C chemokine receptor type 5+CD8+T cells as immune regulators in hepatitis Be antigen-positive chronic hepatitis B under interferonalpha treatment

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World Journal of Gastroenterology 2025年第3期31卷 73-83页

作者： Zhen-Yu xu Zhong-Shang Dai guo-Zhong Gong Min Zhang Department of Infectious Diseases Second Xiangya HospitalCentral South UniversityChangsha 410011Hunan ProvinceChina Institute of Hepatology and Department of Infectious Diseases The Second Xiangya HospitalCentral South UniversityChangsha 410011Hunan ProvinceChina

BACKGROUND C-X-C chemokine receptor type 5(CXCR5)+CD8+T cells represent a unique immune subset with dual roles,functioning as cytotoxic cells in persistent viral infections while promoting B cell *** their importance,the specific role of CXCR5+CD8+T cells in chronic hepatitis B(CHB),particularly during interferon-alpha(IFN-α)treatment,is not fully *** study aims to elucidate the relationship between CXCR5+CD8+T cells and sustained serologic response(SR)in patients undergoing 48 weeks of pegylated IFN-α(peg-IFN-α)treatment for *** To elucidate the relationship between CXCR5+CD8+T cells and sustained SR in patients undergoing 48 weeks of peg-IFN-αtreatment for *** This study enrolled 60 patients with hepatitis Be antigen(HBeAg)-positive CHB undergoing 48 weeks of peg-IFN-α*** were assessed for eligibility based on criteria such as persistent HBsAg-positive status for at least six months,HBeAb-negative,hepatitis B virus DNA levels exceeding 2×104 copies/mL,and alanine aminotransferase(ALT)levels between 2 and 10 times the upper limit of *** samples were collected at baseline and at weeks 12,24,48,and a 24-week treatment-free follow-up(week 72)to measure serum interleukin(IL)-21 concentration via ELISA and to analyze CXCR5 and programmed death-ligand 1(PD-L1)expression on CD8+T cells by flow cytometry,CXCR5 is a chemokine receptor that directs immune cells to specific tissues,while PD-L1 is a protein that regulates immune responses by inhibiting T cell *** Patients with CHB exhibited significantly lower levels of circulating CXCR5+CD8+T cells compared to healthy controls(P<0.01).Notably,CXCR5+CD8+T cells were prominently expressed in patients who achieved sustained SR compared to non-SR(NSR).A significant correlation was observed between CXCR5 and PD-L1 expression(r=-0.189,P=0.002).However,there was no significant correlation between serum IL-21 levels and CXCR5+CD8+lymphocytes(r=-0.03,P=0.62

关键词： C-X-C chemokine receptor type 5 Programmed death-ligand 1 Interleukin-21 Pegylated interferon-alpha Chronic hepatitis B

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Induced neural stem cells regulate microglial activation through Akt-mediated upregulation of CXCR4 and Crry in a mouse model of closed head injury

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Neural Regeneration Research 2025年第5期20卷 1416-1430页

作者： Mou Gao Qin Dong Dan Zou Zhijun Yang Lili guo Ruxiang xu Department of Neurosurgery Sichuan Academy of Medical Sciences and Sichuan Provincial People’s HospitalSchool of MedicineUniversity of Electronic Science and Technology of ChinaChengduSichuan ProvinceChina Department of Neurosurgery Chinese PLA General HospitalBeijingChina Department of Neurology Fu Xing HospitalCapital Medical UniversityBeijingChina

Microglial activation that occurs rapidly after closed head injury may play important and complex roles in neuroinflammation-associated neuronal damage and *** previously reported that induced neural stem cells can modulate the behavior of activated microglia via CXCL12/CXCR4 signaling,influencing their activation such that they can promote neurological ***,the mechanism of CXCR4 upregulation in induced neural stem cells remains *** this study,we found that nuclear factor-κB activation induced by closed head injury mouse serum in microglia promoted CXCL12 and tumor necrosis factor-αexpression but suppressed insulin-like growth factor-1 ***,recombinant complement receptor 2-conjugated Crry(CR2-Crry)reduced the effects of closed head injury mouse serum-induced nuclear factor-κB activation in microglia and the levels of activated microglia,CXCL12,and tumor necrosis factor-α.Additionally,we observed that,in response to stimulation(including stimulation by CXCL12 secreted by activated microglia),CXCR4 and Crry levels can be upregulated in induced neural stem cells via the interplay among CXCL12/CXCR4,Crry,and Akt signaling to modulate microglial *** agreement with these in vitro experimental results,we found that Akt activation enhanced the immunoregulatory effects of induced neural stem cell grafts on microglial activation,leading to the promotion of neurological recovery via insulin-like growth factor-1 secretion and the neuroprotective effects of induced neural stem cell grafts through CXCR4 and Crry upregulation in the injured cortices of closed head injury ***,these beneficial effects of Akt activation in induced neural stem cells were positively correlated with the therapeutic effects of induced neural stem cells on neuronal injury,cerebral edema,and neurological disorders post–closed head *** conclusion,our findings reveal that Akt activation may enhance the immunoregulatory effects of induced neura

关键词： Akt signaling cerebral edema closed head injury Crry CXCR4 induced neural stem cell microglia neuroinflammation

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N-氨甲酰谷氨酸对罗非鱼幼鱼生长、血液氨基酸组成及脂肪沉积的影响

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水生生物学报 2015年第3期39卷 490-497页

作者：程炜轩张丽许国焕吴清洋熊达郭莹姿谭文俊许迪广东省微生物研究所广州510070 广东省菌种保藏与应用重点实验室广州510070 广东省微生物应用新技术公共实验室广州510070 省部共建华南应用微生物国家重点实验室广州510070 广东碧德生物科技有限公司广州510663

为研究精氨酸对尼罗罗非鱼(Oreochromis niloticus)的生物学效应,采用精氨酸内源合成途径的必需辅助因子N-氨甲酰谷氨酸(NCG)代替精氨酸,研究不同剂量包被NCG制剂(实际NCG含量为80%)对尼罗罗非鱼生长及脂肪沉积的影响。实验共设6个制剂... 详细信息

为研究精氨酸对尼罗罗非鱼(Oreochromis niloticus)的生物学效应,采用精氨酸内源合成途径的必需辅助因子N-氨甲酰谷氨酸(NCG)代替精氨酸,研究不同剂量包被NCG制剂(实际NCG含量为80%)对尼罗罗非鱼生长及脂肪沉积的影响。实验共设6个制剂添加组,分别为0.025%、0.05%、0.1%、0.2%、0.4%、0.5%及对照组。结果显示,罗非鱼血液中所有NCG添加组(0.025%—0.5%)和对照组相比,赖氨酸、苏氨酸、亮氨酸、异亮氨酸、缬氨酸等必需氨基酸含量无显著性差异(P>0.05),而当NCG添加量为0.4%时,精氨酸含量显著高于其他各组(P0.05);所有NCG添加组的末均重、特定生长率、饲料系数、内脏比和肥满度等指标与对照组相比均无显著性差异(P>0.05),当NCG添加量为0.2%时,增重率和蛋白质效率均显著高于其他组(P<0.05),除了0.1%NCG添加组,其他各组全鱼蛋白质含量均高于对照组;脂肪沉积方面,所有NCG添加组均显示肝体比显著低于对照组(P<0.05),全鱼干物质中脂肪含量显著高于对照组(P<0.05),脂肪有从肝转移至肌肉的倾向。饲料中添加0.4%NCG,对于血液中精氨酸含量的提高有较好的促进作用,而且不影响其他氨基酸的吸收;饲料中添加0.025%—0.5%NCG有利于减少肝脏脂肪沉积而增加了肌肉中的脂肪沉积,研究结果为进一步开展精氨酸影响鱼体脂肪沉积的机理研究提供了科学依据。

关键词：精氨酸 N-氨甲酰谷氨酸脂肪尼罗罗非鱼生长指标内脏指标氨基酸

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PPARs调控剂对加州鲈生长及糖脂代谢的影响

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淡水渔业 2012年第3期42卷 55-59页

作者：张丽许国焕郭慧青吴月嫦江永明广东省微生物研究所广州510070 广东省菌种保藏与应用重点实验室广州510070 广东省微生物应用新技术公共实验室广州510070 广东省华南应用微生物重点实验室-省部共建国家重点实验室培育基地广州510070 广东碧德生物科技有限公司广州510070

为研究PPARs(过氧化物酶体增殖物激活受体)调控剂对加州鲈(Micropterus salmoides)生长及糖脂代谢的影响,试验采用单因素梯度设计,以基础饲料为对照组,在基础饲料中分别添加调控剂罗格列酮0.3、1.5、7.5和37.5 mg/kg,饲喂初始体重为(3.9... 详细信息

为研究PPARs(过氧化物酶体增殖物激活受体)调控剂对加州鲈(Micropterus salmoides)生长及糖脂代谢的影响,试验采用单因素梯度设计,以基础饲料为对照组,在基础饲料中分别添加调控剂罗格列酮0.3、1.5、7.5和37.5 mg/kg,饲喂初始体重为(3.90±0.04)g的加州鲈56 d后,测定了PPARs调控剂罗格列酮对加州鲈生长和糖脂代谢相关血液生化指标的影响。结果显示:与对照组相比,饲料中添加罗格列酮对加州鲈增重率、特定生长率、饲料系数和蛋白质效率无显著性影响(P>0.05)。添加7.5 mg/kg罗格列酮试验组加州鲈鱼体脂肪和肝脏脂肪含量显著低于对照组及其它试验组(P0.05)。结果表明,添加罗格列酮可预防加州鲈糖脂代谢混乱,防止加州鲈脂肪肝的发生。

关键词： PPARs 罗格列酮加州鲈(Micropterus salmoides) 生长糖脂代谢

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摄食不同饵料对加州鲈生长性能及体成分的影响

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淡水渔业 2011年第6期41卷 60-63页

作者：张丽许国焕郭慧青江永明王珺董小林广东省微生物研究所广州510070 广东省菌种保藏与应用重点实验室广州510070 广东碧德生物科技有限公司广州510070 广东省微生物应用新技术公共实验室广州510070 广东省华南应用微生物重点实验室-省部共建国家重点实验室培育基地广州510070

对摄食不同饵料(冰鲜小杂鱼和配合饲料)加州鲈(Micropterus salmoides)(初始体重为(4.87±0.02)g)生长性能、形体指标和体成分进行了比较分析。结果显示:饲喂冰鲜小杂鱼组的加州鲈末均重、增重率、特定生长率、成活率、肥满度、肌肉粗... 详细信息

对摄食不同饵料(冰鲜小杂鱼和配合饲料)加州鲈(Micropterus salmoides)(初始体重为(4.87±0.02)g)生长性能、形体指标和体成分进行了比较分析。结果显示:饲喂冰鲜小杂鱼组的加州鲈末均重、增重率、特定生长率、成活率、肥满度、肌肉粗蛋白质和脂肪显著高于配合饲料组(P<0.01),饲料系数、肝体比和脏体比显著低于配合饲料组(P<0.01)。结果表明:饲喂冰鲜小杂鱼组可显著提高加州鲈生长性能和饲料利用,改善加州鲈体形和肌肉品质。

关键词：加州鲈(Micropterus salmoides) 冰鲜小杂鱼配合饲料生长体成分

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罗伊氏乳杆菌对组胺引起黄颡鱼肝肠损伤修复效果评价

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水生生物学报 2019年第1期43卷 94-101页

作者：成艳波李薇韩木兰许国焕谢黎炜印遇龙梁建庆广东省微生物研究所广东省部共建华南应用微生物国家重点实验室广东省微生物应用新技术公共实验室广州510070 广东碧德生物科技有限公司广州510663 中国科学院亚热带农业生态研究所长沙410125

以黄颡鱼(Pelteobagrus fulvidraco)(14.23±0.65) g为实验对象,分别饲喂3组等氮饲料(C组:组胺含量147 mg/kg; H组:479 mg/kg和H+B组:496 mg/kg+1×10~5 CFU/g罗伊氏乳杆菌Lactobacillus reuteri),进行56d的养殖实验,研究组胺及罗伊氏乳杆菌对黄颡鱼的影响。结果显示, H+B组血清谷丙转氨酶(ALT)及谷草转氨酶(AST)活力显著低于H组(P0.05); H+B组肝脏谷胱甘肽S转移酶(GSH-ST)活力及丙二醛(MDA)含量显著低于H组(P0.05);肝脏HE染色显示, H组肝脏中有较多炎症细胞浸润,而H+B组肝脏细胞结构完整,无明显损伤;肠道HE染色显示, H组肠道皱襞数量明显减少、长度显著变短,有水肿发生,而H+B组肠道皱襞数量、长度与对照组无差异,水肿程度不明显。结果表明:组胺会引起黄颡鱼肝脏、肠道损伤,而罗伊氏乳杆菌对组胺引发的肝肠损伤有保护功效。

关键词：黄颡鱼罗伊氏乳杆菌组胺 HE染色保护功效

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微生物制剂对奥尼罗非鱼生长及消化酶活性的影响

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水生生物学报 2011年第6期35卷 998-1004页

作者：江永明付天玺张丽许国焕吴月嫦龚全广东省微生物研究所广州510070 广东省菌种保藏与应用重点实验室广州510070 广东省微生物应用新技术公共实验室广州510070 广东省华南应用微生物重点实验室-省部共建国家重点实验室培育基地广州510070 广东碧德生物科技有限公司广州510663

选用192尾初始体重(34.50±0.25)g的健康奥尼罗非鱼(Oreochromis niloticus×***),研究在基础饲料中分别添加相同剂量(活菌含量为3.0×1011 cfu/kg饲料)的汉逊德巴利酵母、枯草芽孢杆菌和凝结芽孢杆菌对奥尼罗非鱼生长及消化酶活性的影... 详细信息

选用192尾初始体重(34.50±0.25)g的健康奥尼罗非鱼(Oreochromis niloticus×***),研究在基础饲料中分别添加相同剂量(活菌含量为3.0×1011 cfu/kg饲料)的汉逊德巴利酵母、枯草芽孢杆菌和凝结芽孢杆菌对奥尼罗非鱼生长及消化酶活性的影响,试验期56d。试验结果表明,与对照组相比,添加枯草芽孢杆菌和凝结芽孢杆菌,奥尼罗非鱼的增重率分别提高12.27%和8.56%(P0.05)。结果显示,三种微生物制剂相互比较,饲料中添加剂量为3.0×1011 cfu/kg饲料的枯草芽孢杆菌和凝结芽孢杆菌能显著提高肠道、肝胰脏和胃的蛋白酶活性,满足最佳生长。

关键词：汉逊德巴利酵母枯草芽孢杆菌凝结芽孢杆菌生长表观消化率:消化酶活性

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凝结芽孢杆菌对奥尼罗非鱼消化酶活性、消化率及生长性能的影响

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淡水渔业 2008年第4期38卷 30-35页

作者：付天玺许国焕吴月嫦龚全江永明广东省微生物研究所

在基础饲料中分别添加不同剂量的凝结芽孢杆菌(Ⅰ:1.0×1011cfu/kg饲料,Ⅱ:3.0×1011cfu/kg饲料,Ⅲ:6.0×1011cfu/kg),室外水族箱中饲养奥尼罗非鱼(Oreochromis niloticus×***)(34.50±0.25 g),用基础饲料投喂作为对照,每饲料组设三个重复,每水族箱随机放养16尾鱼,投喂率为3%。采用静水饲养以避免各箱之间水的交换。56 d后测定鱼体的生长和消化酶活性。结果显示:不同添加量的凝结芽孢杆菌均能显著提高奥尼罗非鱼胃、肝胰脏和肠道蛋白酶活性(P0.05)。Ⅰ组和Ⅱ组的干物质表观消化率、蛋白质消化率、相对增重率、饵料系数和蛋白质效率均显著高于对照组(P0.05)。结果提示,饲料中添加1.0×1011cfu/kg饲料的凝结芽孢杆菌就能显著促进奥尼罗非鱼的生长和饲料营养物质的利用,满足最佳生长。

关键词：凝结芽孢杆菌(Bacillus coagulans) 奥尼罗非鱼(Oreochromis niloticus×O.aureus) 消化酶活性消化率生长

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Tranylcypromine upregulates Sestrin 2 expression to ameliorate NLRP3-related noise-induced hearing loss

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Neural Regeneration Research 2025年第5期20卷 1483-1494页

作者： Xihang Chen Zhifeng Chen Menghua Li Weiwei guo Shuolong Yuan Liangwei xu Chang Lin Xi Shi Wei Chen Shiming Yang Senior Department of Otolaryngology Head and Neck Surgery Chinese PLA General HospitalChinese PLA Medical SchoolBeijingChina State Key Laboratory of Hearing and Balance Science BeijingChina National Clinical Research Center for Otolaryngologic Diseases BeijingChina Key Laboratory of Hearing Science Ministry of EducationBeijingChina Beijing Key Laboratory of Hearing Impairment Prevention and Treatment BeijingChina Department of Otorhinolaryngology Head and Neck Surgery The First Affiliated Hospital of Fujian Medical UniversityFuzhouFujian ProvinceChina Department of Otolaryngology Head and Neck Surgery The 940th Hospital of Joint Logistics Support Force of Chinese PLALanzhouGansu ProvinceChina Key Laboratory of Tropical Biological Resources of Ministry of Education School of Pharmaceutical SciencesHainan UniversityHaikouHainan ProvinceChina Academician Workstation of Hainan University School of Pharmaceutical SciencesYazhou BaySanyaHainan ProvinceChina

Noise-induced hearing loss is the primary non-genetic factor contributing to auditory ***,there are currently no effective pharmacological interventions for patients with noise-induced hearing ***,we present evidence suggesting that the lysine-specific demethylase 1 inhibitor–tranylcypromine is an otoprotective agent that could be used to treat noise-induced hearing loss,and elucidate its underlying regulatory *** established a mouse model of permanent threshold shift hearing loss by exposing the mice to white broadband noise at a sound pressure level of 120 d B for 4 *** found that tranylcypromine treatment led to the upregulation of Sestrin2(SESN2)and activation of the autophagy markers light chain 3B and lysosome-associated membrane glycoprotein 1 in the cochleae of mice treated with *** noise exposure group treated with tranylcypromine showed significantly lower average auditory brainstem response hearing thresholds at click,4,8,and 16 k Hz frequencies compared with the noise exposure group treated with *** findings indicate that tranylcypromine treatment resulted in increased SESN2,light chain 3B,and lysosome-associated membrane glycoprotein 1 expression after noise exposure,leading to a reduction in levels of 4-hydroxynonenal and cleaved caspase-3,thereby reducing noise-induced hair cell ***,immunoblot analysis demonstrated that treatment with tranylcypromine upregulated SESN2 expression via the autophagy *** treatment also reduced the production of NOD-like receptor family pyrin domaincontaining 3(NLRP3)*** conclusion,our results showed that tranylcypromine treatment ameliorated cochlear inflammation by promoting the expression of SESN2,which induced autophagy,thereby restricting NLRP3-related inflammasome signaling,alleviating cochlear hair cell loss,and protecting hearing *** findings suggest that inhibiting lysine-specific demethylase 1 is a

关键词： 4-hydroxynonenal apoptosis autophagy cleaved caspase-3 inflammation NOD-like receptor family pyrin domain-containing 3(NLRP3) noise-induced hearing loss oxidative stress Sestrin2 tranylcypromine

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