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Estrogens, inflammation and obesity： an overview

Frontiers in Biology 2012年第1期7卷 40-47页

作者： Colette N. mILLER Lynda m. BROWN srujana rayalam.m.ry anne della-fer Clifton A. BAILE Department of Foods and Nutrition University of Georgia Athens GA 30602 USA Department of Nutrition University of North Carolina at Greensboro Greensboro NC 27401 USA Department of Animal andDairy Science University of Georgia Athens GA 30602 USA

Emerging research has suggested that inflammatory stress may play a role in the development of *** the leptin and insulin receptor are sensitive to intracellular inflammatory signaling that can be stimulated through toll-like receptor 4 activation by saturated fat. Pharmacological intervention within this cascade often protects animals from becoming obese, thus highlighting inflammatory pathways as a possible site of study in the prevention of pathologic weight gain. It has been well established in animal models that females display a marked reduction in the susceptibility to weight gain on high-fat diets compared to males. In addition, it has been widely accepted that females are partially protected from inflammatory-related diseases. At the molecular level, this reduction in disease susceptibility has been suggested to be due to the anti-inflammatory properties of 17 β-estradiol. Through direct free radical scavenging, transcriptional regulation, and protein interactions, chronic exposure to estradiol can reduce systemic inflammatory stress. As the knowledge base continues to grow on the etiology of obesity, further research is needed on the precise molecular pathways that can be inhibited by estradiol. Understanding of such pathways may provide a basis for the future use of estrogen and its related compounds （daidzein, genistein, resveratrol） to prevent weight gain in peri- and post-menopausal females.

关键词： inflammation obesity sex differences estrogen high fat diets phytoestrogens

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A Dialogue between the Sirene Pathway in Synergids and the fertilization Independent Seed Pathway in the Central Cell Controls male Gamete Release during Double fertilization in Arabidopsis

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molecular Plant 2008年第4期1卷 659-666页

作者： Nicolas Rotman mathieu Gourgues anne-Elisabeth Guitton Jean-Emmanuel Faure Frederic Berger Ecole normale superieure de Lyon Unite Mixte de Recherches 5667 Reproduction et d~veloppement des plantes 46 allee d'ltalie 69007 Lyon cedex 07France Centre for Integrative Genomics University of Lausanne Le Genopode 1015 Lausanne Switzerland Unite mixte de Recherches IPmSV CNRSIINRA/Unice 400 route des chappes Sophia Antipolis F-06903 France European Commission Research Directorate General Research Infrastructures iUnit B3) Office SDME 1/133 B-1049 Brussels Belgium Temasek Life Sciences Laboratory 1 Research link National University of Singapore Singapore 117604

Angiosperms sexual reproduction involves interactions between the two female gametes in the embryo sac and the two male gametes released by the pollen tube. The two synergids of the embryo sac express the ferONIA/SIRENE receptor-like kinase, which controls the discharge of the two sperm cells from the pollen tube. fer/SRN may respond to a ligand from the pollen tube. Alternatively, the interaction between fer/SRN and a ligand from the embryo sac may lead to a state of competence of the synergids allowing pollen tube discharge. Here, we report the new mutant scylla （syl） impaired in the control of pollen tube discharge. This mutant also produces autonomous endosperm development in ab- sence of fertilization--a trait associated with the ferTILIZATION INDEPENDENT SEED （FIS） mutant class. This led us to iden- tify autonomous endosperm in srn mutants and to demonstrate synergistic interactions between srn and the fis mutants. In addition, the fis mutants display defects in pollen tube discharge as in srn and syl mutants, confirming the interaction between the two pathways. Our findings suggest that pollen tube discharge is controlled by an interaction between the synergids expressing SRN/fer and the central cell expressing FIS genes.

关键词： ferONIA/SIRèNE fer/SRN

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软膜紫外光固化纳米压印中Amonil光刻胶的刻蚀参数优化(英文)

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纳米技术与精密工程 2012年第1期10卷 52-58页

作者：陈静石剑刘正堂 DECANINI Dominique HAGHIRI-GOSNET anne-marie 西北工业大学凝固国家重点实验室西安710072 法国国家科研中心光子与纳米结构实验室马库锡91460 法国巴黎高等师范学院巴黎75005

报道了反应离子刻蚀转移图形过程中对Amonil光刻胶的刻蚀参数优化的结果.利用软膜紫外光固化纳米压印技术,首先制备了线宽/间距均为200 nm的纳米光栅结构.然后采用反应离子刻蚀的方法去除残留的Amonil光刻胶.研究了不同的气体组成、射... 详细信息

报道了反应离子刻蚀转移图形过程中对Amonil光刻胶的刻蚀参数优化的结果.利用软膜紫外光固化纳米压印技术,首先制备了线宽/间距均为200 nm的纳米光栅结构.然后采用反应离子刻蚀的方法去除残留的Amonil光刻胶.研究了不同的气体组成、射频功率、压强和气体流量对刻蚀形貌、表面粗糙度以及刻蚀速度的影响.在优化的工艺条件下,获得了理想的具有垂直侧壁形貌和较小表面粗糙度的纳米光栅阵列.结果表明,选择优化的刻蚀工艺参数,既能有效地改善图形转移的性能,同时也能提高所制备结构的光学应用特性.

关键词：软膜紫外光固化纳米压印反应离子刻蚀刻蚀形貌表面粗糙度

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