检索结果-南通市图书馆

用CMd-3谱仪研究VEPP-2000对撞机上的e^+e^-→K^+K^-η过程（英文）

在线全文

学校读者我要写书评

暂无评论

中国科学技术大学学报 2016年第6期46卷 502-506页

作者： IVAnOV V.L. AMIRKHAnOV A.n. AnISEnKOV A.V. AULCHEnKO V.M. BAnZAROV V.S. BASHTOVOY n.S. BERKAEV d.E. BOndAR A.E. BRAGIn A.V. EIdELMAn S.I. EPIFAnOV d.A. EPSHTEYn L.B. EROFEEV A.L. FEdOTOVICH G.V. GAYAZOV S.E. GREBEnUK A.A. GRIBAnOV S.S. grigoriev d.n. IGnATOV F.V. KARPOV S.V. KAZAnIn V.F. KOROBOV A.A. KOVALEnKO O.A. KOZYREV A.n. KOZYREV E.A. KROKOVnY P.P. KUZMEnKO A.E. KUZMIn A.S. LOGASHEnKO I.B. LUKIn P.A. MIKHAILOV K.YU. OKHAPKIn V.S. PESTOVY U.n. POPOV A.S. RAZUVAEV G.P. RUBAn A.A. RYSKULOV n.M. RYZHEnEnKOV A.E. SHEBALIn V.E. SHEMYAKIn d.n. SHWARTZ B.A. SIBId.n.V A.L. SHATUnOVY U.M. SOLOdOV E.P. TITOV V.M. TALYSHEV A.A. VOROBIOV A.I. YUd.n. U.V. 俄罗斯布德克尔核物理研究所国立新西伯利亚大学国立新西伯利亚理工大学悉尼大学物理学院高能物理系

报道了用CMd-3在2011-2012年收集的积分亮度为22 pb-1的研究结果.在CMd-3当前的统计量下,只能被确定Ф(1020)η中间共振态.在1.59-2.0 Ge V能区的30个能量点测量了e^+e^-→Ф(1020)η.分析中,将η介子当做反冲粒子,穷尽了η衰变的所有模... 详细信息

报道了用CMd-3在2011-2012年收集的积分亮度为22 pb-1的研究结果.在CMd-3当前的统计量下,只能被确定Ф(1020)η中间共振态.在1.59-2.0 Ge V能区的30个能量点测量了e^+e^-→Ф(1020)η.分析中,将η介子当做反冲粒子,穷尽了η衰变的所有模式,选出的事例总数为1454±48.测量的截面近似为Ф(1680)和非共振振幅按照矢量介子主导模型的叠加,并得到了Ф(1680)介子共振参数的初步结果.

关键词：强子信号/本地分离截面

CMd-3实验π介子形状因子的初步结果（英文）

在线全文

学校读者我要写书评

暂无评论

中国科学技术大学学报 2016年第4期46卷 323-330页

作者： AKHMETSHIn R.R. AMIRKHAnOV A.n. AnISEnKOV A.V. AULCHEnKO V.M. BAnZAROV V.Sh. BASHTOVOY n.S. BERKAEV d.E. BOndAR A.E. BRAGIn A.V. FEdOTOVICH G.V. EIdELMAn S.I. EPIFAnOV d.A. EPSHTEYn L.B. EROFEEV A.L. GAYAZOV S.E. GREBEnUKV A.A. GRIBAnOVV S.S. grigoriev d.n. IGnATOV F.V. IVAnOV V.L. KARPOV S.V. KAZAnIn V.F. KHAZIn B.I. KOOP I.A. KOROBOV A.A. KOVALEnKO O.A. KOZYREV A.n. KOZYREV E.A. KROKOVnY P.P. KUZMEnKO A.E. KUZMIn A.S. LOGASHEnKO I.B. LUKIn P.A. MIKHAILOV K.Yu. OKHAPKIn V.S. PESTOV Yu.n. POPOV A.S. RAZUVAEV G.P. RUBAn A.A. RYSKULOV n.M. RYZHEnEnKOV A.E. SHEBALIn V.E. SHWARTZ B.A. SHWARTZ d.B. SIBId.n.V A.L. SHATUnOV Yu.M. SOLOdOV E.P. SHEMYAKIn d.n. TITOV V.M. TALYSHEV A.A. VOROBIOV A.I. YUd.n.Yu.V. 布德克尔核物理研究所新西伯利亚州立大学新西伯利亚州立理工大学悉尼大学物理学院

自2010年12月起,坐落在正负电子对撞机VEPP-2000上的CMd-3探测器成功取数.2013年首次在对撞能量小于1GeV能区进行扫描,并获取积分亮度为18pb-1的数据.本文给出了e^+e^-→π^+π^-截面的初步结果.

关键词： π介子形状因子正负电子对撞机强子截面

VEPP-2000 e^+e^-对撞机上用CMd-3探测器测量e^+e^-→ηπ^+π^-和e^+e^-→ωπ^+π^-的截面（英文）

在线全文

学校读者我要写书评

暂无评论

中国科学技术大学学报 2016年第6期46卷 514-522页

作者： POPOV A.S. AKHMETSHIn R.R. AMIRKHAnOV A.n. AnISEnKOV A.V. AULCHEnKO V.M. BAnZAROV V.S. BASHTOVOY n.S. BERKAEV d.E. BOndAR A.E. BRAGIn A.V. EIdELMAn d.E. EPIFAnOV d.A. EPSHTEYn L.B. EROFEEV A.L. FEdOTOVICH G.V. GAYAZOV S.E. GREBEnUK A.A. GRIBAnOV S.S. grigoriev d.n. GROMOV E.M. IGnATIn F.V. IVAnOV V.L. KARPOV S.V. KASAEV A.S. KORPOTIn A.n. KOOP I.A. KOVALEnKO O.A. KAZAnIn V.F. KOZYREV A.n. KOZYREV E.A. KROKOVnY P.P. KUZMEnKO A.E. KUZMIn A.S. LOGASHEnKO I.B. LIKIn P.A. MIKHAILOV K.YU. OKHAPKIn V.S. OTBOEV A.V. PESTOVY U.n. PIVOVAROV S.G. RAZUVAEV G.P. ROMAnOV A.L. RUBAn A.A. RYSKULOV n.M. RYZHEnEnKOV A.E. SHEBALIn V.E. SHEMYAKIn d.n. SHWARTZ B.A. SHWARTZ d.B. SIBId.n.OV A.L. SHATUnOV P.YU. SHATUnOVY U.M. SOLOdOV E.P. TITOV V.M. TALYSHEV A.A. VOROBIOV A.I.. YUd.n. U.V. ZHARInOVY U.M. 俄罗斯布德克尔核物理研究所国立新西伯利亚大学东京大学物理系国立新西伯利亚理工大学悉尼大学物理学院高能物理系

在VEPP^-2000e^+e^-对撞机上,CMd^-3探测器研究了e^+e^-→ηπ^+π^-→γγπ^+π^-,e^+e^-→ηπ^+π^-→π0π^+π^-π^+π^-以及e^+e^-→ωπ^+π^-→π0π^+π^-π^+π^-等过程.分析使用的是2011和2012年在1.2^-2.0Ge V能区收集的相应积分亮度为3×104nb^-1的数据.用η→γγ道测量了e^+e^-→ηπ^+π^-的波恩截面,结果与相关的实验结论相吻合;还用末态η→π^+π^-π0和ω→π^+π^-π0分别得到了e^+e^-→ηπ^+π^-和e^+e^-→ωπ^+π^-波恩截面的初步结果;e^+e^-→ηπ^+π^-波恩截面的数值还被用于确定τ^-→ηπ^-π0ντ的衰变分支比.

关键词：截面 ηπ+π- ωπ+π- 强子 τ

VEPP-2000 e^+e^-对撞机上用CMd-3谱仪研究1004-1060 MeV能区的e^+e^-→K■过程（英文）

在线全文

学校读者我要写书评

暂无评论

中国科学技术大学学报 2016年第6期46卷 507-513页

作者： KOZYREV E.A. AMIRKHAnOV A.n. AnISEnKOV A.V. M.AULCHEnKO V. BAnZAROV V.S. BASHTOVOY n.S. BERKAEV d.E. BOndAR A.E. BRAGIn A.V. EIdELMAn S.I. A.EPIFAnOV d. EPSHTEYn L.B. EROFEEV A.L. FEdOTOVICH G.V. GAYAZOV S.E. GREBEnUK A.A. GRIBAnOV S.S. grigoriev d.n. IGnATOV F.V. IVAnOV V.L. KARPOV S.V. KASAEV A.S. KAZAnIn V.F. IRPOTIn A.n. KOROBOV A.A. KOVALEnKO O.A. KOZYREV A.n. KOOP I.A. KROKOVnY P.P. KUZMEnKO A.E. KUZMIn A.S. LOGASHEnKO I.B. LUKIn P.A. MIKHAILOV K.YU. OKHAPKIn V.S. OTBOEV A.V. PESTOVY U.n POPOV A.S. RAZUVAEV G.P. RUBAn A.A. RYSKULOV n.M. RYZHEnEnKOV A.E. SEnCHEnKO A.I. SHEBALIn V.E. SHEMYAKIn V.E. SHWARTZ A.S. SHWARTZ d.B. SIBId.n.V A.L. SHATUnOV P.YU. SHATUnOVY U.M. SOLOdOV E.P. TITOV V.M. TALYSHEV A.A. VOROBIOV A.I. YUd.n. U.V. 俄罗斯布德克尔核物理研究所国立新西伯利亚大学国立新西伯利亚理工大学悉尼大学物理学院高能物理系

在质心能量1004-1060Me V之间的25个能量点测量了e^+e^-→K_S^0+K_L^0和e^+e^-→KK截面,系统误差大约为2%-3%.分析是基于VEPP-2000e^+e^-对撞机上CMd-3收集的积分亮度为5.5 pb-1的实验数据.测量的截面可近似看作Ф,ω,ρ类似的振幅及其... 详细信息

在质心能量1004-1060Me V之间的25个能量点测量了e^+e^-→K_S^0+K_L^0和e^+e^-→KK截面,系统误差大约为2%-3%.分析是基于VEPP-2000e^+e^-对撞机上CMd-3收集的积分亮度为5.5 pb-1的实验数据.测量的截面可近似看作Ф,ω,ρ类似的振幅及其激发态按照矢量介子主导模型的叠加,得到了Ф(1020)介子的共振参数.

关键词：强子正负电子对撞机介子形状因子

Effects of Periodically Inhomogeneous Birefringence on dark-Bright Vector Soliton Propagation and Interaction

在线全文

学校读者我要写书评

暂无评论

Chinese Physics Letters 2007年第2期24卷 462-464页

作者：李宏 d.n. Wang Hubei Key Laboratory of Bioanalytical Technique Hubei Normal University Huangshi 435002 School of Mechanical and Electronic Engineering Huangshi Institute of Technology Huangshi 435002 department of Electrical Engineering The Hong Kong Polytechnic University Hong Kong

The effects of periodically inhomogeneous birefringence on dark-bright vector soliton propagation and interaction are investigated by the numerical method. The birefringence leads to the submergence of the dark soliton and the disintegration of the bright soliton, and enhances the interaction between the neighbouring solitons. The system performance is determined by the bright soliton because the dark soliton has robust features. Finally, the avoid.n.e and the effective control are introduced, and the controlling mechanism is demonstrated.

关键词： RAndOMLY VARYInG BIREFRInGEnCE OPTICAL SOLITOnS PERTURBATIOnS PARAMETERS FIBERS

nucleoside modified mRnA-lipid.n.noparticles as a new delivery platform for the repair of the injured spinal cord

维普期刊数据库评论

在线全文

维普期刊数据库

学校读者我要写书评

暂无评论

neural Regeneration Research 2025年第8期20卷 2311-2312页

作者： Krisztián Pajer Tamás Bellák Antal nógrádi department of Anatomy Histology and EmbryologyAlbert Szent-Györgyi Medical SchoolUniversity of SzegedSzegedHungary

Spinal cord injury and treatment opportunities:The adult mammalian spinal cord has a very limited capacity for spontaneous regeneration due to various intrinsic molecular and cellular *** the spinal cord.n.urons have the capacity to regenerate their axons,the expression of growth inhibitory factors,lack or suppression of proper guid.n.e cues,and profound inflammatory responses do not permit successful regeneration(Khyeam et al.,2021).

关键词： injured capacity modified

Impacts of PI3K/protein kinase B pathway activation in reactive astrocytes: from detrimental effects to protective functions

在线全文

学校读者我要写书评

暂无评论

neural Regeneration Research 2025年第4期20卷 1031-1041页

作者： Ramón Pérez-núñez María Fernanda González Ana María Avalos Lisette Leyton Cellular Communication Laboratory Programa de Biología Celular y MolecularCenter for Studies on ExerciseMetabolism and Cancer(CEMC)Instituto de Ciencias Biomédicas(ICBM)Facultad de MedicinaUniversidad de ChileSantiagoChile Advanced Center for Chronic diseases(ACCdiS) Faculty of Chemical and Pharmaceutical Sciences&Faculty of MedicineUniversidad de ChileSantiagoChile Instituto de Ciencias Biomédicas Facultad de Ciencias de la SaludUniversidad Autónoma de ChileSantiagoChile

Astrocytes are the most abund.n. type of glial cell in the central nervous *** injury and inflammation,astrocytes become reactive and undergo morphological and functional *** on their phenotypic classification as A1 or A2,reactive astrocytes contribute to both neurotoxic and.n.uroprotective responses,***,this binary classification does not fully capture the diversity of astrocyte responses observed across different diseases and *** analysis has revealed that reactive astrocytes have a complex landscape of gene expression profiles,which emphasizes the heterogeneous nature of their *** actively participate in regulating central nervous system inflammation by interacting with microglia and other cell types,releasing cytokines,and influencing the immune *** phosphoinositide 3-kinase(PI3K)/protein kinase B(AKT)signaling pathway is a central player in astrocyte reactivity and impacts various aspects of astrocyte behavior,as evid.n.ed by in silico,in vitro,and in vivo *** astrocytes,inflammatory cues trigger a cascade of molecular events,where nuclear factor-κB serves as a central mediator of the pro-inflammatory ***,we review the heterogeneity of reactive astrocytes and the molecular mechanisms underlying their *** highlight the involvement of various signaling pathways that regulate astrocyte reactivity,includ.n. the PI3K/AKT/mammalian target of rapamycin(mTOR),αvβ3 integrin/PI3K/AKT/connexin 43,and.n.tch/PI3K/AKT *** targeting the inactivation of the PI3K/AKT cellular signaling pathway to control reactive astrocytes and prevent central nervous system damage,evid.n.e suggests that activating this pathway could also yield beneficial *** dual function of the PI3K/AKT pathway underscores its complexity in astrocyte reactivity and brain function *** review emphasizes the importance of employing astrocyte-exclusive models to underst

关键词： inflammation integrins neuroprotective neurotoxic phosphatidylinositol 3-kinase reactive astrocytes signal transduction Thy-1(Cd90)

Sex differences in aging and injured brain

在线全文

学校读者我要写书评

暂无评论

neural Regeneration Research 2025年第10期20卷 2901-2902页

作者： Jord.n.n.Williamson Yuan Yang University of Illinois Urbana-Champaign Grainger College of EngineeringDepartment of BioengineeringUrbanaILUSA Carle Foundation Hospital Stephenson Family Clinical Research InstituteClinical Imaging Research CenterUrbanaILUSA University of Illinois Urbana-Champaign Beckman Institute for Advanced Science and TechnologyUrbanaILUSA northwestern University Department of Physical Therapy and Human Movement SciencesChicagoILUSA

Background:The prevalence,age of onset,and symptomatology of traumatic brain injury,stroke,and.n.urodegenerative diseases(such as Alzheimer’s disease(Ad),Parkinson’s disease,amyotrophic lateral sclerosis,and Huntington’s disease)differ substantially between males and *** higher prevalence of these brain disorders has been attributed to females having a greater longevity compared with *** one of the greatest risk factors of acquired brain injury(such as stroke,traumatic brain injury caused by fall)and.n.urodegenerative disease is age.

关键词： Huntington prevalence traumatic

Astrocytes, reactive astrogliosis, and glial scar formation in traumatic brain injury

在线全文

学校读者我要写书评

暂无评论

neural Regeneration Research 2025年第4期20卷 973-989页

作者： María Belén Cieri Alberto Javier Ramos Laboratorio de neuropatología Molecular IBCN UBA-CONICETFacultad de MedicinaUniversidad de Buenos AiresBuenos AiresArgentina

Traumatic brain injury is a global health crisis,causing significant death and disability *** that follows traumatic brain injury has serious consequences for neuronal survival and cognitive impairments,with astrocytes involved in this *** traumatic brain injury,astrocytes rapidly become reactive,and astrogliosis propagates from the injury core to distant brain *** astroglial proteins are downregulated.n.ar the traumatic brain injury core,while pro-inflammatory astroglial genes are *** altered gene expression is considered a pathological remodeling of astrocytes that produces serious consequences for neuronal survival and cognitive *** addition,glial scar formed by reactive astrocytes is initially necessary to limit immune cell infiltration,but in the long term impedes axonal reconnection and functional *** therapeutic strategies for traumatic brain injury are focused on preventing acute ***,cannabinoids,progesterone,beta-blockers,and cerebrolysin demonstrate neuroprotective benefits but most of them have not been studied in the context of *** this review,we discuss the cell signaling pathways activated in reactive astrocytes following traumatic brain injury and we discuss some of the potential new strategies aimed to modulate astroglial responses in traumatic brain injury,especially using cell-targeted strategies with miRnAs or lncRnA,viral vectors,and repurposed drugs.

关键词： astrocyte glial scar innate immunity neuroinflammation stab injury Toll-like receptors