CDK9-42 Translocation Promotes Alloresponse Predominantly via STAT1/IFN-γ Pathway
作者单位：Biomedical isotope research center
会议名称：《第十一届全国免疫学学术大会》
会议日期：2016年
学科分类：1001[医学-基础医学(可授医学、理学学位)] 100102[医学-免疫学] 10[医学]
关 键 词：allorejection CDK9-42 translocation STAT1 STAT3
摘 要：Objective:Recently it has been reported that CDK9-42,not CDK9-55,promoted allorejection in allotransplantation mice model through inducing anti-donor Th1 response.Here,we aimed to investigate whether CDK942 translocation show STAT1/IFN-γsignaling-dependent manner in alloresponse.