A meta-analysis of soluble suppression of tumorigenicity 2 （sST2） and clinical outcomes in pulmonary hypertension
A meta-analysis of soluble suppression of tumorigenicity 2 （sST2） and clinical outcomes in pulmonary hypertension作者机构：Department of Medicine and Therapeutics Faculty of Medicine Chinese University ofHong Kong Hong Kong China Li Ka Shing Institute of Health Sciences Faculty of Medicine Chinese University ofHong Kong Hong Kong China Tianjin Key Laboratory of Ionic-Molecular Function of Cardiovascular disease Department of Cardiology Tianjin Institute of Cardiology Second Hospital of Tianjin Medical University Tianjin China Department of Anaesthesia and Intensive Care State Key Laboratory of Digestive Disease LKS Institute of Health Sciences The Chinese University of Hong Kong Hong Kong China The State and Shandong Province Joint Key Laboratory of Translational Cardiovascular Medicine Department of Cardiology Qilu Hospital Shandong University Jinan China Research Center of Ion Channelopathy Institute of Cardiology Union Hospital Tongii Medical College Huazhong University of Science and Technology Wuhan China JC School of Public Health and Primary Care The Chinese University ofHong Kong Hong Kong China State Key Laboratory of Digestive Disease LKS Institute of Health Sciences Chinese University ofHong Kong Hong Kong China
出 版 物：《Journal of Geriatric Cardiology》 (老年心脏病学杂志（英文版）)
年 卷 期：2017年第14卷第12期
页 面：766-771页
核心收录：
学科分类：090603[农学-临床兽医学] 0832[工学-食品科学与工程（可授工学、农学学位）] 1002[医学-临床医学] 08[工学] 09[农学] 0906[农学-兽医学] 083202[工学-粮食、油脂及植物蛋白工程]
基 金：Croucher Foundation of Hong Kong
主 题：高血压 临床 膜受体 ST2 IL-1 肺 免疫力 家庭
摘 要：Suppression of Tumorigenicity 2 (ST2) is a member of the interleukin (IL)-1 receptor family. The ST2 receptor exists in two isoforms - ST2 ligand (ST2L) and soluble ST2 (sST2). ST2L is a membrane receptor and sST2 is a truncated receptor which is soluble in the blood, allowing it to be detected in serum. IL-33 is a member of the IL-1 family of ligand and is the functional ligand of ST2L receptor. It binds to the ST2L, thereby mediating its immune function.
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