Stress corrosion cracking behavior of PH13-8Mo stainless steel in Cl^- solutions

作     者：Qiang Yu Chao-fang Dong Jian-xiong Liang Zhen-bao Liu Kui Xiao Xiao-gang Li 

作者机构：Corrosion and Protection Center Institute of Advanced Materials and Technology University of Science and Technology Beijing Beijing 100083 China Institute for Special Steel Central Iron and Steel Research Institute Beijing 100081 China 

出 版 物：《Journal of Iron and Steel Research International》 (国际钢铁研究杂志)

年 卷 期：2017年第24卷第3期

页      面：282-289页

核心收录：

学科分类：080503[工学-材料加工工程] 08[工学] 080502[工学-材料学] 0805[工学-材料科学与工程（可授工学、理学学位）] 

基　　金：supported by the National Natural Science Foundation of China(No.51171023) the Fundamental Research Funds for the Central Universities(No.FRF-TP-14-011C1) National Basic Research Program of China(973 Program )(No.2014CB643300 ) the Beijing Municipal Commission of Education 

主　　题：Stress corrosion cracking PH13-8Mo precipitation hardening stainless steel Neutral NaCl solution Slow-strain-rate tensile test Applied potential 

摘      要：The stress corrosion cracking（ SCC） behavior of PH13-8Mo precipitation hardening stainless steel（ PHSS） in neutral NaCl solutions was investigated through slow-strain-rate tensile（ SSRT） test at various applied potentials. Fracture morphology,elongation ratio,and percentage reduction of area were measured to evaluate the SCC susceptibility. A critical concentration of 1. 0 mol / L neutral NaCl existed for SCC of PH13-8Mo steel. Significant SCC emerged when the applied potential was more negative than -0. 15 VSCE,and the SCC behavior was controlled by an anodic dissolution（ AD） *** the applied potential was lower than -0. 55 VSCE,an obvious hydrogen-fracture morphology was observed,which indicated that the SCC behavior was controlled by hydrogen-induced cracking（ HIC）.Between -0. 15 and -0. 35 VSCE,the applied potential exceeded the equilibrium hydrogen evolution potential in neutral NaCl solutions and the crack tips were of electrochemical origin in the anodic region; thus,the SCC process was dominated by the AD mechanism.