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Aetiological factors of Budd-Chiari syndrome in Algeria

World Journal of hepatology 2015年第6期7卷 903-909页

作者： Nawel Afredj Nawal Guessab Abdelbasset Nani Sid Ahmed Faraoun Ibtissem Ouled Cheikh Rafik Kerbouche Djouhar hannoun Zine Charef Amir hayet Ait Kaci Kamel Bentabak Aurélie Plessier Dominique-Charles Valla Valerie Cazals-hatem marie-hélène denninger Tadjeddine Boucekkine Nabil Debzi hepatology Unit Department of Medicine Mustapha Hospital Radiology Unit Centre Pierre and Marie Curie National Institute of Public health (INSP) Pathology Unit CHU Mustapha Pathology Unit Centre Pierre and Marie Curie Surgery Unit Centre Pierre and Marie Curie hepatology Unit Beaujon Hospital Pathology Unit Beaujon Hospital Immunohematology laboratory Beaujon Hospital

AIM: To study the clinical presentation of Budd-Chiari syndrome(BCS) and identify the aetiologies of this disease in Algeria. METhODS: Patients with BCS, hospitalised in our unit from January 2004 until June 2010 were included and the aetiological factors were assessed. Patients presenting a BCS in the setting of advanced-stage cirrhosis or a liver transplantation were excluded from the study. The diagnosis was established when an obstruction of hepatic venous outflow(thrombosis, stenosis or compression) was demonstrated. We diagnosed myeloproliferative disease(MPD) by bone marrow biopsy and V617 F JAK2 mutation. Anti-phospholipid syndrome(APlS) was detected by the presence of anticardiolipin antibodies, anti-β2 glycoprotein antibodies and lupus anticoagulant. We also detected paroxysmal nocturnal haemoglobinuria(PNh) by flow cytometry. Celiac disease and Behcet disease were systematically investigated in our patients. hereditary anticoagulant protein deficiencies were also assessed. We tested our patients for the G20210 A mutation at Beaujon hospital. Imaging procedures were performed to determine a local cause of BCS, such as a hydatid cyst or a liver ***: One hundred and fifteen patients were included. Mean follow up: 32.12 mo. Mean age: 34.41 years, M/F = 0.64. Chronic presentation was frequent:63.5%. The revealing symptoms for the BCS were ascites(74.8%) and abdominal pain(42.6%). The most common site of thrombosis was the hepatic veins(72.2%). Involvement of the inferior vena cava alone was observed in 3 patients. According to the radiological investigations, BCS was primary in 94.7% of the cases(n = 109) and secondary in 5.2%(n = 6). An aetiology was identified in 77.4% of the patients(n = 89); it was multifactorial in 27%(n = 31). The predominant aetiology of BCS in our patients was a myeloproliferative disease, observed in 34.6% of cases. APlS was found in 21.7% and celiac disease in 11.4%. Other acquired conditions were: PNh(n = 4), systemic dis

关键词：阿尔及利亚 Etiologie 腹的疾病 Budd-Chiari 血栓

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hepatic veins as a site of clot formation following liver resection

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World Journal of Gastroenterology 2011年第3期17卷 403-406页

作者： Emmanuel Buc Safi Dokmak Magaly Zappa marie helene denninger Dominique Charles Valla Jacques Belghiti Olivier Farges Department of hepato-Biliary Surgery Hpital BeaujonAssistance-Publique Hpitaux de ParisUniversity Paris 7 Department of Radiology Hpital Beaujon Assistance-Publique Hpitaux de ParisUniversity Paris 7 Department of Biological hematology Hpital Beaujon Assistance-Publique Hpitaux de ParisUniversity Paris 7 Department of hepatology Hpital BeaujonAssistance-Publique Hpitaux de ParisUniversity Paris 7

Pulmonary embolism occurs more frequently after hepatectomy than previously thought but is infrequently associated with peripheral deep vein thrombosis. In this paper, we report 2 cases of postoperative hepatic vein thrombosis after liver resection. Both patients had undergone major hepatectomy of a non-cirrhotic liver largely exposing the middle hepatic vein. Clots were incidentally found in the middle hepatic vein 4 and 17 d after surgery despite routine systemic thrombo-prophylaxis with low molecular weight heparin. Coagulation of the transition plan in a context of mutation of the prothrombin gene and inflammation induced biloma were the likely predisposing conditions. Clots disappeared following curative anticoagulation. We conclude that thrombosis of hepatic veins may occur after liver resection and is a potential source of pulmonary embolism.

关键词：血栓形成肝切除肝静脉网站低分子量肝素凝血酶原诱发条件基因突变

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用于中国人群的压力管理、心身增弹方案(SMART-C)及其子方案健康睡眠(hS)的编制

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世界睡眠医学杂志 2017年第2期4卷 72-75页

作者：王芳 herbert Benson Gregory l.Fricchione John W.denninger 中国中医科学院广安门医院睡眠心理科北京100053 哈佛大学医学院麻省总医院本森亨利心身医学研究所波士顿02114

压力的定义有很多,与慢性压力相关的疾病谱系非常广泛。压力管理相关疗法与心身医学密切相关。压力反应和放松反应的发现,为心身疗法应用于压力管理提供了依据。哈佛大学医学院麻省总医院本森亨利心身医学研究所的赫伯特·本森教授团队... 详细信息

压力的定义有很多,与慢性压力相关的疾病谱系非常广泛。压力管理相关疗法与心身医学密切相关。压力反应和放松反应的发现,为心身疗法应用于压力管理提供了依据。哈佛大学医学院麻省总医院本森亨利心身医学研究所的赫伯特·本森教授团队一直致力于运用心身疗法来干预慢性压力的临床和科研工作。他们开发出压力管理、弹性训练方案(SMART),专门针对压力相关症状或疾病工作。笔者通过在哈佛大学医学院麻省总医院本森亨利心身医学研究所两年博士后的工作和学习,结合自身临床经验,在充分考虑中美两国文化背景差异的基础上,编制出适用于中国人群的压力管理、心身增弹方案(SMARTC)及以健康睡眠(hS)为代表的若干子方案。用于临床实践,取得了一定的疗效。

关键词：心身医学压力管理睡眠质量

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本土化压力管理与心身增弹训练(SMART-C)对提升教师心理弹性与睡眠质量的作用研究

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世界睡眠医学杂志 2018年第7期5卷 808-812页

作者：王芳冯帆 John W.Denniger herbert Benson Gregory l.Fricchione 中国中医科学院广安门医院睡眠心理科北京100053 哈佛大学医学院麻省总医院本森亨利心身医学研究所波士顿02114

目的:探索本土化压力管理心身增弹训练(SMART-C)对于教师群体的应对压力能力以及睡眠质量的改善作用,为进一步完善压力情绪管理应用于教育方案(RS-C)提供依据。方法:通过本土化压力管理心身增弹训练(SMART-C)对北京市东城区38位教师进... 详细信息

目的:探索本土化压力管理心身增弹训练(SMART-C)对于教师群体的应对压力能力以及睡眠质量的改善作用,为进一步完善压力情绪管理应用于教育方案(RS-C)提供依据。方法:通过本土化压力管理心身增弹训练(SMART-C)对北京市东城区38位教师进行干预,采用心理弹性量表、匹兹堡睡眠质量指数判定效果。结果:参与团体的教师在心理弹性的忍受消极情感和精神影响方面有所改善,睡眠方面在睡眠质量、入睡时间、睡眠时间、睡眠效率、日间功能等方面所有改善。结论:本土化压力管理心身增弹训练(SMART-C)对于教师群体的应对压力能力以及睡眠质量有改善作用。

关键词：本土化压力管理与心身增弹训练教师心身弹性睡眠质量

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特发性肝窦扩张与抗磷脂综合征免疫学特征的相关

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世界核心医学期刊文摘（胃肠病学分册） 2005年第2期1卷 38-38页

作者： Saadoun D. Cazals-hatem D. denninger M.-h. D. Valla 翟惠虹 Service d'hép atalogie Hpital Beaujon 92118 Clichy France

Background: Isolated sinusoidal dilatation is an uncommon hepatic lesion and t he cause is largely unknown. Objective: To investigate whether prothrombotic dis orders or perisinusoidal cell changes could be involved in pure idiopathic hepat ic sinusoidal dilatation (hSD). Methods: Evaluation for associated conditions, p rothrombotic disorders, and studies of hepatic perisinusoidal cell activation in consecutive patients, seen between 1993 and 2002, with isolated sinusoidal dila tation unrelated to outflow block, sinusoidal infiltration, or hepatic granuloma s. Results: Among 11 patients, associated conditions were prothrombotic disorder s (n = 5) and oral contraceptive use (n = 3). Prothrombotic disorders were polyc ythemia vera (n = 1) and anticardiolipin antibodies combined with lupus anticoag ulant (n = 4). No genetic thrombophilia factor was found. Of four patients with lupus anticoagulant, three had antinuclear factors and high serum levels of anti cardiolipin antibodies at repeated testing. There was no evidence of intrahepati c or extrahepatic thrombosis in any of the patients. Sinusoidal dilatation was m arked in six of 11 patients (54%), including two patients with antiphospholipid antibodies. Activated perisinusoidal cells were only found around markedly dila ted sinusoids. Conclusion: Idiopathic pure hSD is frequently associated with the immunological features of the antiphospholipid syndrome. Therefore, finding pur e hSD in a liver biopsy specimen should prompt the search for antiphospholipid a ntibodies.

关键词：抗磷脂综合征免疫学特征肝窦扩张凝血酶原肝肉芽肿心肌磷脂窦状隙肝活检标本流出道梗阻狼疮抗凝因子

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