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Connected phytohormone biosynthesis pathways at the core of growth-stress tradeoffs

Molecular Plant 2022年第7期15卷 1087-1089页

作者： Katelyn Sageman-Furnas lucia strader Duke University DurhamNC 27708USA

How plants experiencing environmental stress regulate their growth to survive is a fundamental question crucial for future food security.Drought and high salinity both represent osmotic stresses that severely decrease crop yields worldwide.Understanding how plants cope with these stresses becomes increasingly critical as the world faces a rapid deterioration of arable land.Plants respond to stress by activating stress-avoidance or stress-tolerance pathways,reprogramming gene regulatory networks,and increasing protective solutes(reviewed in Claeys and lnze,2013;Zhang et al.,2020).

关键词： programming stress critical

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Transport and Metabolism of the Endogenous Auxin Precursor Indole-3-Butyric Acid

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Molecular Plant 2011年第3期4卷 477-486页

作者： lucia C. strader Bonnie Bartel Department of Biochemistry and Cell Biology Rice University Houston TX 77005 USA

Plant growth and morphogenesis depend on the levels and distribution of the plant hormone auxin. Plants tightly regulate cellular levels of the active auxin indole-3-acetic acid （IAA） through synthesis, inactivation, and transport. Although the transporters that move IAA into and out of cells are well characterized and play important roles in devel- opment, little is known about the transport of IAA precursors. In this review, we discuss the accumulating evidence sug- gesting that the IAA precursor indole-3-butyric acid （IBA） is transported independently of the characterized IAA transport machinery along with the recent identification of specific IBA efflux carriers and enzymes suggested to metabolize IBA. These studies have revealed important roles for IBA in maintaining IAA levels and distribution within the plant to support normal development.

关键词： Auxin auxin transport indole-3-butyric acid IBA.

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Nitric oxide-mediated S-nitrosylation of IAA17 protein in intrinsically disordered region represses auxin signaling

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Journal of Genetics and Genomics 2023年第7期50卷 473-485页

作者： Hongwei Jing Xiaolu Yang Ryan J.Emenecker Jian Feng Jian Zhang Marcelo Rodrigues Alves de Figueiredo Patarasuda Chaisupa R.Clay Wright Alex S.Holehouse lucia C.strader Jianru Zuo State Key Laboratory of Plant Genomics Institute of Genetics and Developmental BiologyChinese Academy of SciencesBeijing 100101China University of Chinese Academy of Sciences Beijing 100049China Department of Biology Duke UniversityDurhamNC 27008USA Department of Neurobiology Duke University Medical CenterDurhamNC 27710USA Department of Biochemistry and Molecular Biophysics Washington University School of MedicineSt.LouisMO 63110USA Center for Biomolecular Condensates(CBC) Washington University in St.LouisSt.LouisMO 63130USA Department of Biological Systems Engineering Virginia TechBlacksburgVA 24061USA The Translational Plant Sciences Center(TPSC) Virginia TechBlacksburgVA 24061USA CAS Center for Excellence in Molecular Plant Sciences Chinese Academy of SciencesBeijing 100101China

The phytohormone auxin plays crucial roles in nearly every aspect of plant growth and development.Auxin signaling is activated through the phytohormone-induced proteasomal degradation of the Auxin/INDOLE-3-ACETIC ACID(Aux/IAA)family of transcriptional repressors.Notably,many auxin-modulated physiological processes are also regulated by nitric oxide(NO)that executes its biological effects predominantly through protein S-nitrosylation at specific cysteine residues.However,little is known about the molecular mechanisms in regulating the interactive NO and auxin networks.Here,we show that NO represses auxin signaling by inhibiting IAA17 protein degradation.NO induces the S-nitrosylation of Cys-70 located in the intrinsically disordered region of IAA17,which inhibits the TIR1-IAA17 interaction and consequently the proteasomal degradation of IAA17.The accumulation of a higher level of IAA17 attenuates auxin response.Moreover,an IAA17^(C70W)nitrosomimetic mutation renders the accumulation of a higher level of the mutated protein,thereby causing partial resistance to auxin and defective lateral root development.Taken together,these results suggest that S-nitrosylation of IAA17 at Cys-70 inhibits its interaction with TIR1,thereby negatively regulating auxin signaling.This study provides unique molecular insights into the redox-based auxin signaling in regulating plant growth and development.

关键词： Arabidopsis thaliana Auxin Aux/IAA Nitric oxide S-nitrosylation Intrinsically disordered region

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