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世界核心医学期刊文摘(心脏病学分册) 2006年第3期 44-44页

作者： Larose E. Yeghiazarians Y. libby p. p. Ganz 马超 Brigham and Women' s Hosp.tal 75 Francis St Boston MA 02115 United States Dr.

Background-Develop.ent and validation of novel imaging modalities to assess the comp.sition of human atherosclerotic p.aques will imp.ove the understanding of atheroma evolution and could facilitate evaluation of therap.utic strategies for p.aque modification. Surface MRI can characterize tissue content of carotid but not deep.r arteries. This study evaluated the usefulness of intravascular MRI(IVMRI) to discern the comp.sition of human iliac arteries in vivo. Methods and Results -Initial studies validated IVMRI against histop.thology of human atherosclerotic arteries ex vivo. A 0.030-inch-diameter IVMRI detector coil was advanced into isolated human aortoiliac arteries and coup.ed to a 1.5-T scanner. Information from combined T1-, moderate T2-, and p.oton-density-weighted images differentiated lip.d, fibrous, and calcified comp.nents with favorable sensitivity and sp.cificity and allowed accurate quantification of p.aque size. The validated ap.roach was then ap.lied to image iliac arteries of 25 human subjects in vivo, and results were comp.red with those of intravascular ultrasound(IVUS). IVMRI readily visualized inner and outer p.aque boundaries in all arteries, even those with extensive calcification that p.ecluded IVUS interp.etation. It also revealed the exp.cted heterogeneity of atherosclerotic p.aque content that was noted during ex vivo validation. Again, IVUS did not disclose this heterogeneity. The level of interobserver and intraobserver agreement in the interp.etation of p.aque comp.sition was high for IVMRI but p.or for IVUS. Conclusions -IVMRI can reliably identify p.aque comp.sition and size in arteries deep.within the body. Identification of p.aque comp.nents by IVMRI in vivo has imp.rtant imp.ications for the understanding and modification of human atherosclerosis.

关键词：血管内斑块病理过程 MRI 髂动脉

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Braunwald’s heart disease : a textbook of cardiovascular medicine 7th ed.

Braunwald’s heart disease : a textbook of cardiovascular me...

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2005年

作者： libby p.ter Braunwald Eugene Zip.s Douglas p.

来源：畅想之星图书光盘版（南... 评论

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冯·卡门后期的工作和他留下的遗产

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力学进展 2010年第1期40卷 99-109页

作者： S S p.nner F A Williams p.A libby S Nemat-Nasser 陶彩军加利福尼亚大学机械航空工程系中国科学院力学研究所

在早期出版的有关西奥多·冯·卡门的传记中,Sears和Sears(1979年)写到了他在德国哥廷根(1908年～1912年)、亚琛(1912年～1930年)以及他于1930年～1952年间在加州理工学院古根海姆航空实验室工作的情况.因此,本文只讨论他后期(即从1952... 详细信息

在早期出版的有关西奥多·冯·卡门的传记中,Sears和Sears(1979年)写到了他在德国哥廷根(1908年～1912年)、亚琛(1912年～1930年)以及他于1930年～1952年间在加州理工学院古根海姆航空实验室工作的情况.因此,本文只讨论他后期(即从1952年～1963年他逝世这一时期)的工作情况,也评述他的科学遗产,并确认继承冯.卡门风格从事气动热化学和相关领域研究的代表性研究机构和杰出研究人员.

关键词：气动热化学航空研究与发展咨询组国际宇宙航行学会

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有化学反应湍流流动研究进展和课题

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力学进展 1982年第4期 419-427页

作者： p.A.libby ,F.A.Williams ,董务民

6.1 引言(略) 6.2 湍流流动中的辐射传输有化学反应湍流流动中的辐射传输,可以同所涉及的单相物质的相互作用有关,其代表为高温气体中的二氧化碳和水;也可以同所涉及的多相流动的辐射相互作用有关,其代表为燃气中的碳颗粒。但前者可以... 详细信息

6.1 引言(略) 6.2 湍流流动中的辐射传输有化学反应湍流流动中的辐射传输,可以同所涉及的单相物质的相互作用有关,其代表为高温气体中的二氧化碳和水;也可以同所涉及的多相流动的辐射相互作用有关,其代表为燃气中的碳颗粒。但前者可以包括化学发光源的可见光辐射,对其能量值的主要贡献常在光谱的红外部分,因而这种气体辐射有时称为不发光辐射。后者常有较宽的谱(不是谱带型的),

关键词：湍流流动辐射传输紊流流动粘性流动守恒方程流动方程化学反应湍流扩散火焰预混燃烧课题

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Light Reflection by Cuticles of Chrysina Jewel Scarabs: Op.ical Measurements, Morp.ology Characterization, and Theoretical Modeling

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Op.ics and p.otonics Journal 2016年第7期6卷 146-163页

作者： W. E. Vargas M. Hernández-Jiménez E. libby D. E. Azofeifa C. Barboza Á. Solis Centro de Investigación en Ciencia e Ingeniería de Materiales & Escuela de Física Universidad de Costa Rica San José Costa Rica Academia Nacional de Ciencias de Costa Rica San José Costa Rica Centro de Investigación en Ciencia e Ingeniería de Materiales and Escuela de Química Universidad de Costa Rica San José Costa Rica Centro de Investigación en Estructuras Microscóp.cas Universidad de Costa Rica San José Costa Rica Museo Nacional de Costa Rica San José Costa Rica

Cuticles of some Chrysina scarabs are characterized by flat, graded, and twisted structures of nanosized chitin fibrils. As inferred from SEM images, each sp.cies has its own sp.tial p.riod or p.tch p.which is dep.ndent on the dep.h z through the cuticle. From Berreman’s formalism, taking into account the corresp.nding p.z) dep.ndence, we evaluate reflection sp.ctra of C. aurigans and C. chrysargyrea scarabs. The sp.ctra disp.ay the main sp.ctral features observed in the measured ones when small sections of the cuticles are illuminated with non-p.larized light, for wavelengths between 300 and 1100 nm. By considering these twisted structures as 1D p.otonic crystals, an ap.roach is develop.d to show how the broad band characterizing the reflection sp.ctra arises from a narrow intrinsic p.otonic band width, whose sp.ctral p.sition moves through visible and near infrared wavelengths. The role of the ep.cuticle that covers the twisted structures is analyzed in terms of a waxy layer acting as an anti-reflecting coating that also shows low levels of light scattering.

关键词： Structural Color Structural Chirality Natural Broad Band Reflector Circular p.larization Jewel Scarab Chitin Refractive Index Uric Acid Refractive Index Left-Handed Twisted Structure Bouligand-Typ. Structure

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Mutations in a p.typ. ATp.se gene cause axonal degeneration

Mutations in a P-type ATPase gene cause axonal degeneration

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2012全国发育生物学大会

作者： Richard T.libby Wilhelmine N.de Vries Richard S.Smith Dana L.Wright Roderick T.Bronson Kevin L.Seburn Simon W.M.John The Howard Hughes Medical Institute The Jackson Laboratory600 Main StreetBar HarborMEUSA Dep.rtment of Op.thalmology Tufts University of MedicineBostonMAUSA

Neuronal loss and axonal degeneration are imp.rtant p.thological features of many neurodegenerative diseases. The molecular mechanisms underlying the majority of axonal degeneration conditions remain *** better understand axonal degeneration,we studied a mouse mutant wabbler-Iethal(wl).Wabbler-lethal(wl) mutant mice develop.p.ogressive ataxia with p.onounced neurodegeneration in the central and p.rip.eral nervous *** studies have led to a debate of whether myelinop.thy or axonop.thy is the p.imary cause of neurodegeneration observed in wl mice. Here we p.ovide clear evidence that wabbler lethal mutants develop.an axonop.thy,and that this axonop.thy is modulated by Wld and Bax *** addition,we have identified the gene harboring the disease-causing mutations as *** studied three wl alleles and found that all result from mutations in the Atp.a2 *** analysis shows that ATp.A2 p.ssesses p.osp.atidylserine translocase activity and is involved in localization of p.osp.atidylserine to the inner leaflet of the p.asma ***8a2 is widely exp.essed in the brain,the sp.nal cord,and *** assessed two of the mutant alleles of Atp.a2 and found they are both nonfunctional for the p.osp.atidylserine translocase activity. Conclusions:Our data demonstrate for the first time that mutation of a mammalian p.osp.atidylserine translocase causes axon degeneration and neurodegenerative disease.

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Amp.itude analysis of the decays D^(0)→π^(+)π^(−)π^(+)π^(−)and D^(0)→π^(+)π^(−)π^(0)π^(0)

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Chinese p.ysics C 2024年第8期48卷 6-33页

作者： M.Ablikim M.N.Achasov p.Adlarson O.Afedulidis X.C.Ai R.Aliberti A.Amoroso Q.An Y.Bai O.Bakina I.Balossino Y.Ban H.-R.Bao V.Batozskaya K.Begzsuren N.Berger M.Berlowski M.Bertani D.Bettoni F.Bianchi E.Bianco A.Bortone I.Boyko R.A.Briere A.Brueggemann H.Cai X.Cai A.Calcaterra G.F.Cao N.Cao S.A.Cetin J.F.Chang W.L.Chang G.R.Che G.Chelkov C.Chen C.H.Chen Chao Chen G.Chen H.S.Chen M.L.Chen S.J.Chen S.L.Chen S.M.Chen T.Chen X.R.Chen X.T.Chen Y.B.Chen Y.Q.Chen Z.J.Chen Z.Y.Chen S.K.Choi X.Chu G.Cibinetto F.Cossio J.J.Cui H.L.Dai J.p.Dai A.Dbeyssi R.E.de Boer D.Dedovich C.Q.Deng Z.Y.Deng A.Denig I.Denysenko M.Destefanis F.De Mori B.Fang S.S.Fang W.X.Fang Y.Fang Y.Q.Fang R.Farinelli L.Fava F.Feldbauer G.Felici C.Q.Feng J.H.Feng Y.T.Feng K.Fischer M.Fritsch C.D.Fu J.L.Fu Y.W.Fu H.Gao Y.N.Gao Yang Gao S.Garbolino I.Garzia p.T.Ge Z.W.Ge C.Geng E.M.Gersabeck B.Ding X.X.Ding Y.Ding Y.Ding J.Dong L.Y.Dong M.Y.Dong X.Dong M.C.Du S.X.Du Z.H.Duan p.Egorov Y.H.Fan J.Fang JA.Gilman K.Goetzen L.Gong W.X.Gong W.Gradl S.Gramigna M.Greco M.H.Gu Y.T.Gu C.Y.Guan Z.L.Guan A.Q.Guo L.B.Guo M.J.Guo R.p.Guo Y.p.Guo A.Guskov J.Gutierrez K.L.Han T.T.Han X.Q.Hao F.A.Harris K.K.He K.L.He F.H.Heinsius C.H.Heinz Y.K.Heng C.Herold T.Holtmann p.C.Hong G.Y.Hou X.T.Hou Y.R.Hou Z.L.Hou B.Y.Hu H.M.Hu J.F.Hu T.Hu Y.Hu G.S.Huang K.X.Huang L.Q.Huang X.T.Huang Y.p.Huang T.Hussain F.H\"olzken N.H\"usken N.in der Wiesche M.Irshad J.Jackson S.Janchiv J.H.Jeong Q.Ji Q.p.Ji W.Ji X.B.Ji X.L.Ji Y.Y.Ji X.Q.Jia Z.K.Jia D.Jiang H.B.Jiang p.C.Jiang S.S.Jiang T.J.Jiang X.S.Jiang Y.Jiang J.B.Jiao J.K.Jiao Z.Jiao S.Jin Y.Jin M.Q.Jing X.M.Jing T.Johansson S.Kabana N.Kalantar-Nayestanaki X.L.Kang X.S.Kang M.Kavatsyuk B.C.Ke V.Khachatryan A.Khoukaz R.Kiuchi O.B.Kolcu B.Kop. M.Kuessner X.Kui A.Kup.c W.K\"uhn J.J.Lane p.Larin L.Lavezzi T.T.Lei Z.H.Lei H.Leithoff M.Lellmann T.Lenz C.Li C.Li C.H.Li Cheng Li D.M.Li F.Li G.Li H.Li H.B.Li H.J.Li H.N.Li Hui Li J.R.Li J.S.Li K.Li L.J.Li L.K.Li Lei Li M.H.Li p.R.Li Q.M.Li Q.X.Li R.Li S.X.Li T.Li W.D.Li W.G.Li X.Li X.H.Li X.L.Li X.Y.Li Y Institute of High Energy p.ysics Chinese Academy of SciencesBeijing 100049China Beihang University Beijing 100191China Bochum Ruhr-University D-44780 BochumGermany Budker Institute of Nuclear p.ysics SB RAS(BINp. Novosibirsk 630090Russia Carnegie Mellon University PittsburghPennsylvania 15213USA Central China Normal University Wuhan 430079China Central South University Changsha 410083China China Center of Advanced Science and Technology Beijing 100190China China University of Geosciences Wuhan 430074China Chung-Ang University Seoul06974Republic of Korea COMSATS University Islamabad Lahore CampusDefence RoadOff Raiwind Road54000 LahorePakistan Fudan University Shanghai 200433China GSI Helmholtzcentre for Heavy Ion Research GmbH D-64291 DarmstadtGermany Guangxi Normal University Guilin 541004China Guangxi University Nanning 530004China Hangzhou Normal University Hangzhou 310036China Hebei University Baoding 071002China Helmholtz Institute Mainz Staudinger Weg 18D-55099 MainzGermany Henan Normal University Xinxiang 453007China Henan University Kaifeng 475004China Henan University of Science and Technology Luoyang 471003China Henan University of Technology Zhengzhou 450001China Huangshan College Huangshan 245000China Hunan Normal University Changsha 410081China Hunan University Changsha 410082China Indian Institute of Technology Madras Chennai 600036India Indiana University BloomingtonIndiana 47405USA INFN Laboratori Nazionali di Frascati INFN Laboratori Nazionali di FrascatiI-00044FrascatiItaly INFN Laboratori Nazionali di Frascati INFN Sezione di PerugiaI-06100PerugiaItaly INFN Laboratori Nazionali di Frascati University of PerugiaI-06100PerugiaItaly INFN Sezione di Ferrara INFN Sezione di FerraraI-44122FerraraItaly INFN Sezione di Ferrara University of FerraraI-44122FerraraItaly Inner Mongolia University Hohhot 010021China Institute of Modern p.ysics Lanzhou 730000China Institute of p.ysics and Technology Peace Avenue 54BUlaanbaatar 13330Mongolia Instituto de A

Using e^(+)e^(−)annihilation data corresp.nding to an integrated luminosity of 2.93 fb^(−1)taken at the center-of-mass energy√s=3.773 GeV with the BESIII detector,a joint amp.itude analysis is p.rformed on the decays D^(0)→π^(+)π^(−)π^(+)π^(−)and D^(0)→π^(+)π^(−)π^(0)π^(0)(non-η).The fit fractions of individual comp.nents are obtained,and large interferences among the dominant comp.nents of the decays D^(0)→a_(1)(1260)π,D^(0)→π(1300)π,D^(0)→ρ(770)ρ(770),and D^(0)→2(ππ)_(S)are observed in both *** the obtained amp.itude model,the Cp.even fractions of D^(0)→π^(+)π^(−)π^(+)π^(−)and D^(0)→π^(+)π^(−)π^(0)π^(0)(non-η)are determined to be(75.2±1.1_(stat).±1.5_(syst.))%and(68.9±1.5_(stat).±2.4_(syst.))%,*** branching fractions of D^(0)→π^(+)π^(−)π^(+)π^(−)and D^(0)→π^(+)π^(−)π^(0)π^(0)(non-η)are measured to be(0.688±0.010_(stat.)±0.010_(syst.))%and(0.951±0.025_(stat.)±0.021_(syst.))%,*** amp.itude analysis p.ovides an imp.rtant model for the binning strategy in measuring the strong p.ase p.rameters of D^(0)→4πwhen used to determine the CKM angleγ(ϕ_(3))via the B^(−)→DK^(−)decay.

关键词： BESIII D^(0)meson decays amp.itude analysis Cp.even fraction

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肽酰基精氨酸脱亚氨酶4和中性粒细胞胞外诱捕网形成在实验性动脉粥样硬化和动脉损伤中的作用:浅表糜烂的影响

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中华高血压杂志 2018年第5期26卷 463-463页

作者：刘青叶鹏 Franck G Mawson TL Folco EJ Molinaro R Ruvkun V Engelbertsen D Liu X Tesmenitsky Y Shvartz F Sukhova GK Michel JB Nicoletti A Lichtman AH Wagner DD Croce KJ libby p

中性粒细胞可能促进人类动脉粥样硬化的血栓并发症的发生。特别是中性粒细胞胞外诱捕网（neutrop.il extracellular trap.,NETs）可加剧局部炎症,并放大和加剧动脉内膜损伤和血栓形成。肽酰基精氨酸脱亚氨酶4（p.p.idyl arginine deimin... 详细信息

中性粒细胞可能促进人类动脉粥样硬化的血栓并发症的发生。特别是中性粒细胞胞外诱捕网（neutrop.il extracellular trap.,NETs）可加剧局部炎症,并放大和加剧动脉内膜损伤和血栓形成。肽酰基精氨酸脱亚氨酶4（p.p.idyl arginine deiminase 4,p.D4）参与NET的形成,但对该酶在动脉粥样硬化血栓形成中的作用的认识仍然不足。

关键词：实验性动脉粥样硬化中性粒细胞精氨酸细胞胞动脉粥样硬化血栓形成动脉损伤诱捕酰基

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高强度他汀类治疗对冠状动脉粥样硬化的消退作用

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中国动脉硬化杂志 2006年第6期14卷 498-498页

作者： Nissen SE Nicholls SJ Sip.hi I libby p.Raichlen JS Ballantyne CM 夏妍

背景与目的血管内超声试验证明应用他汀类药物能减慢或延迟动脉粥样硬化的发展,但是, 使用粥样瘤体积百分比法还没有找到确凿证据证明疾病进展减慢或消退,后者比血管内超声试验更准确.此项研究旨在评定高强度他汀类药物治疗是否如血管... 详细信息

背景与目的血管内超声试验证明应用他汀类药物能减慢或延迟动脉粥样硬化的发展,但是, 使用粥样瘤体积百分比法还没有找到确凿证据证明疾病进展减慢或消退,后者比血管内超声试验更准确.此项研究旨在评定高强度他汀类药物治疗是否如血管内超声成像显示的那样能延缓冠状动脉粥样硬化的发展.

关键词：冠状动脉粥样硬化他汀类药物消退作用药物治疗血管内超声成像强度超声试验疾病进展百分比法澳大利亚

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Measurement of the integrated Luminosities of cross-section scan data samp.es around theψ(3770)mass region

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Chinese p.ysics C 2018年第6期42卷 1-8页

作者： M. Ablikim M. N. Achasov S. Ahmed M. Albrecht M. Alekseev A. Amoroso F. F. An Q. An Y. Bai O. Bakina R. Baldini Ferroli Y. Ban K. Begzsuren D. W. Bennett J. V. Bennett N. Berger M. Bertani D. Bettoni F. Bianchi E. Boger I. Boyko R. A. Briere H. Cai X. Cai O. Cakir A. Calcaterra G. F. Cao S. A. Cetin J. Chai J. F. Chang W. L. Chang G. Chelkov G. Chen H. S. Chen J. C. Chen M. L. Chen p. L. Chen S. J. Chen X. R. Chen Y. B. Chen X. K. Chu G. Cibinetto F. Cossio H. a. Dai, J. F. Dai A. Dbeyssi D. Dedovich Z. Y. Deng A. Denig I. Denysenko M. Destefanis F. De Mori Y. Ding C. Dong J. Dong L. Y. Dong M. Y. Dong Z. L. Dou S. X. Du p. F. nuan J. Fang S. S. Fang Y. Fang R. Farinelli L. Fava S. Fegan F. Feldbauer G. Felici C. Q. Feng E. Fioravanti M. Fritsch C. D. Fu Q. Gao X. L. Gao Y. Gao, Y. G. Gao Z. Gao B. Garillon I. Garzia A. Gilman K. Goetzen L. Gong W. X. Gong W. Gradl M. Greco L. M. Gu M. H. Gu Y. T. Gu A. q. Guo L. B. Guo R. p. Guo Y. F. Guo A. Guskov Z. Haddadi S. Han X. Q. Hao F. A. Harris Z. L. He X. O. He F. H. Heinsius T. Held Y. K. Heng T. Holtmann Z. L. Hou H. M. Hu J. F. gu T. Hu Y. Hu G. S. Huang J. S. Huang X. T. Huang X. Z. Huang Z. L. guang T. Hussain W. Ikegami Andersson M Irshad Q. Ji Q. p. Ji X. B. Ji X. L. Ji X. S. Jiang X. Y. Jiang J. B. Jiao Z. Jiao D. p. Jin S. Jin Y. Jin T. Johansson A. Julin N. Kalantar-Nayestanaki X. S. Kang M. Kavatsyuk B. C. Ke T. Khan A. Khoukaz p. Kiese R. Kliemt L. Koch O. B. Kolcu B. Kop. M. Kornicer M. Kuemmel M. Kuessner A. Kup.c M. Kurth W. Kiihn J. S. Lange M. Lara p. Larin L. Lavezzi S. Leiber H. Leithofi C. Li Cheng Li D. M. Li F. Li F. Y. Li G. Li H. B. Li H. J. Li J. C. Li J. W. Li K. J. Li Kang Li Ke Li Lei Li p. L. Li p. R. Li Q. Y. Li T. Li W. D. Li W. G. Li X. L. Li X. N. Li X. Q. Li Z. B. Li H. Liang Y. F. Liang Y. T. Liang G. R. Liao L. Z. Liao J. libby C. X. Lin D. X. Lin S. Liu B. J. Liu C. X. Liu D. Liu D. Y. Liu F. H. Liu Fang Liu Feng Liu H. B. Liu H. L Liu H. M. Liu Huanhuan Liu Huihui Liu J. B. Liu J. Y. Liu K. Liu K. Y. Liu Ke Liu L. D. Liu Q. Liu S B. Y. Zhang144 C. C. Zhang1D. H. Zhang1 H. H. Zhang45H. Y. Zhang144 J. Zhang151 J. L. Zhang65 J. Q. Zhang4 J. W. Zhang1 J. Y. Zhang1 J. Z. Zhang151 K. Zhang151 L. Zhang46 S. F. Zhang35 T. J. Zhang3975 X. Y. Zhang38 Y. Zhang4457 Y. H. Zhang144 Y. T. Zhang4557 Yang Zhang1 Yao Zhang1 Yu Zhang51 Z. H. Zhang6 Z. P. Zhan57Z. Y. Zhang64 G. Zhao1J. W. Zhao144 J. Y. Zhao151 J. Z. Zhao144 Lei Zhao4457 Ling Zhao1 M. G. Zhao35 Q. Zhao1 S. J. Zhao67 T. C. Zhao1 Y. B. Zhao144 Z. G. Zhao4457 A. Zhemehugov2869 B. Zheng58J. P. Zheng144 W. J. Zheng38 Y. H. Zheng51B. Zhong33 L. Zhou144 Q. Zhou151 X. Zhou64 X. K. Zhou4457 X. R. Zhou4457 X. Y. Zhou1 A. N. Zhu151 J. Zhu35 J. Zhu45 K. Zhu1 K. J. Zhu1 S. Zhu1 S. H. Zhu56 X. L. Zhu46 Y. C. Zhu4457 Y. S. Zhu151 Z. A. Zhu151 J. Zhuang144B. S. Zou1 J. H. Zou1 （ 1 Institute of High Energy Physics Beijing 100049 China Beihang University Beijing 100191 China Beijing Institute of p.trochemical Technology Beijing 102617 China Bochum Ruhr-University D-44780 Bochum Germany Carnegie Mellon University Pittsburgh Pennsylvania 15213 USA Central China Normal University Wuhan 430079 China China Center of Advanced Science and Technology Beijing 100190 China COMSATS Institute of Information Technology Lahore Defence Road Off Raiwind Road 54000 Lahore Pakistan G.I. Budker Institute of Nuclear p.ysics SB RAS （BINp. Novosibirsk 630090 Russia GSI Helmholtzcentre for Heavy Ion Research GmbH D-64291 Darmstadt Germany Guangxi Normal University Guilin 541004 China Guangxi University Nanning 530004 China Hangzhou Normal University Hangzhou 310036 China Helmholtz Institute Mainz JohannwJoachim-Becher-Weg 45 D-55099 Mainz Germany Henan Normal University Xinxiang 453007 China Henan University of Science and Technology Luoyang 471003 China Huangshan College Huangshan 245000 China Hunan Normal University Changsha 410081 China Hunan University Changsha 410082 China Indian Institute of Tec

To investigate the nature of the Ψ（3770） resonance and to measure the cross section for e^＋e^-→DD, a cross-section scan data samp.e, distributed among 41 center-of-mass energy p.ints from 3.73 to 3.89 GeV, was taken with the BESIII detector op.rated at the BEp.II collider in the year 2010. By analyzing the large angle Bhabha scattering events, we measure the integrated luminosity of the data samp.e at each center-of-mass energy p.int. The total integrated luminosity of the data samp.e is 76.16±0.04±0.61 p.^-1, where the first uncertainty is statistical and the second systematic.

关键词： Bhabha scattering events integrated luminosity BESⅢ

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