Background:Severe acute respiratory syndrome coronavirus 2(SARS-CoV-2),which causes COVId-19,is characterized by an increased risk of thromboembolic ***,more than 80%of patients are asymptomatic or have only minor/mil...
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Background:Severe acute respiratory syndrome coronavirus 2(SARS-CoV-2),which causes COVId-19,is characterized by an increased risk of thromboembolic ***,more than 80%of patients are asymptomatic or have only minor/mild *** addition,diagnosing thromboembolism in athletes is challenging,as symptoms can be confused with musculoskeletal complaints or physical *** presentation:Here we report the case of a previously healthy 34-year-old professional soccer player with COVId-19 infection and genetic predisposition to *** baseline,he was fit,had no symptoms,did not require hospital admission due to a COVId-19 infection,and was started on a five-day course of azithromycin and dexamethasone *** 10 days of returning to professional activity,he developed pulmonary embolism following a COVId-19 infection during a physical exercise *** showed positive acute and subacute pulmonary thromboembolism,being treated with rivaroxaban 20 mg/day *** shared decision-making between the medical team and the athlete was not to return to professional soccer,given the quantifiable ***:This case illustrates the potential risk of COVId-19-induced pulmonary thromboembolism,which can be affected by genetic predisposition and dexamethasone therapy or the consequences of *** this clinical period,the athlete’s condition may be overlooked due to the masking effects of other clinical conditions and physical *** residual effects of COVId-19 disease can appear late,requiring caution and follow-up by the medical team before releasing the athlete into a training program.
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