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Natural regeneration responses to thinning and burning treatments in ponderosa pine forests and implications for restoration

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Journal of Forestry Research 2022年第3期33卷 741-753页

作者： Tzeidle N.wasserman Amy E.M.Waltz John Paul Roccaforte Judith D.Springer Joseph E.Crouse Ecological Restoration Institute Northern Arizona UniversityP.O.Box 15017FlagstaffAZ86011-5017USA

Understanding naturally occurring pine regeneration dynamics in response to thinning and burning treatments is necessary not only to measure the longevity of the restoration or fuels treatment,but also to assess how well regeneration meets forest sustainability guidelines and whether natural regeneration is sufficient for maintaining a sustainable forest structure and composition.A synthesis review was carried out on the effects of mechanical thinning and prescribed burn treatments on natural pine regeneration response in frequent-fire ponderosa pine forests across the western United States.The focus was on site-specific variability in pine regeneration dynamics,temporal trends in regeneration presence and abundance,and response to treatment as described in the current literature using 29 studies that met our evidence-based review protocols.Data showed that the effects of thinning and burning treatments on regeneration depended on time since treatment.Mechanical thinning,prescribed burning,and thinning plus burn treatments all increased seedling density,but there was high variability among sites and studies.There were mixed results in the short-term(20 years)concluded that stands can return to pre-treatment densities in terms of total trees per hectare and forest floor duff levels when there are no maintenance treatments applied.Several studies showed the average ponderosa pine seedling presence,survival and growth found in today’s forests to be at a high density;this combined with missed fire cycles could contribute to future fire risk and reduce the efficacy of maintaining fuel reduction goals.

关键词： Regeneration Ponderosa pine Frequent-fire Treatments Thinning Burning

Extracellular matrix remodelling in obesity and metabolic disorders

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Life Metabolism 2023年第4期2卷 7-21页

作者： Vishal Musale David H.wasserman Li Kang Division of Systems Medicine School of MedicineUniversity of DundeeDundee DD19SYUnited Kingdom Department of Molecular Physiology and Biophysics Mouse Metabolic Phenotyping CenterVanderbilt UniversityNashvilleTN 37235United States

Obesity causes extracellular matrix (ECM) remodelling which can develop into serious pathology and fibrosis, having metabolic effects in insulin-sensitive tissues. The ECM components may be increased in response to overnutrition. This review will focus on specific obesity-associated molecular and pathophysiological mechanisms of ECM remodelling and the impact of specific interactions on tissue metabolism. In obesity, a complex network of signalling molecules such as cytokines and growth factors has been implicated in fibrosis. Increased ECM deposition contributes to the pathogenesis of insulin resistance at least in part through the activation of cell surface integrin receptors and CD44 signalling cascades. These cell surface receptors transmit signals to the cell adhesome which orchestrates an intracellular response that adapts to the extracellular environment. Matrix proteins, glycoproteins, and polysaccharides interact through ligand-specific cell surface receptors that interact with the cytosolic adhesion proteins to elicit specific actions. Cell adhesion proteins may have catalytic activity or serve as scaffolds. The vast number of cell surface receptors and the complexity of the cell adhesome have made study of their roles challenging in health and disease. Further complicating the role of ECM-cell receptor interactions is the variation between cell types. This review will focus on recent insights gained from studies of two highly conserved, ubiquitous axes and how they contribute to insulin resistance and metabolic dysfunction in obesity. These are the collagen-integrin receptor-IPP (ILK-PINCH-Parvin) axis and the hyaluronan-CD44 interaction. We speculate that targeting ECM components or their receptor-mediated cell signalling may provide novel insights into the treatment of obesity-associated cardiometabolic complications.

关键词： extracellular matrix fibrosis insulin resistance obesity metabolism

0 Heroi （The Hero） and the Post-Colonial Angolan State

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International Relations and Diplomacy 2016年第3期4卷 203-208页

作者： Bonnie wasserman University of Arizona Arizona USA

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美教授引爆新影响者模型

成功营销 2007年第4期 50-50页

作者： Todd wasserman 张淼（翻译）

纽约——影响者、联系者和内行。多年来．传统的观点都认为这三个因素是制造引爆流行的关键因素，你必须找准并影响这三类人物，让他们将你的观点传播出去。这个论断是Malcolm Gladwell于2000年在他的年度畅销书《引爆流行》中提出来的... 详细信息

纽约——影响者、联系者和内行。多年来．传统的观点都认为这三个因素是制造引爆流行的关键因素，你必须找准并影响这三类人物，让他们将你的观点传播出去。这个论断是Malcolm Gladwell于2000年在他的年度畅销书《引爆流行》中提出来的。书中指出，人群中约占15％的人．对于其他人有着极大的影响力。这些影响者拥有比大多数人都要多的朋友．而且也有着比较强烈的欲望先于他人占有更多的社会资源（比如独家信息和产品）。尽管Gladwell不辞辛苦地指出这些社交人士（联系者）需要和社会信息持有者（内行）取得联系，甚至很多人集两种角色于一身，但现在哥伦比亚大学社会学教授Duncan Watts却提出质疑．他认为整个影响者理论都只是胡言乱语．绝大多数时候流行是通过人际关系网络传播的，那些易于被影响的大众个体都是在看到他们某个邻居采用了某些新的装扮和品牌后才开始尝试的。令人惊讶的是，美国最大的口碑传播公司波士顿BzzAgent公司创始者兼首席执行官Dave Baiter先生也赞同Watts的观点。Baiter认为：”很多人依旧相信影响者模式．就像愚人金，此类概念容易卖出去。”但是事实上，现实情况往往会更加复杂，在不同的时候．每个人都可能是影响者。《品牌周刊》的编辑Todd wasserman就Watts教授的理论对他进行了采访。

关键词：《引爆流行》《品牌周刊》模型哥伦比亚大学社会信息首席执行官社会资源网络传播

RFID:A Beacon of Light in the Banking System

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射频世界 2009年第3期 73-76页

作者： Elizabeth wasserman RFID Journal

Although RFID won’t fix the mortgage problem,banks are investing in the technology to improve other business practices-a move they believe will pay off in dollars and sense. By RFID Journal,used with

关键词：技术与应用

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过滤杂音

成功营销 2007年第2期 37-37页

作者： Todd wasserman 蒋永军（翻译）

任何一位有着网上购物经验的人都知道．网络商家推荐给他们的商品经常是他们不需要的。

关键词：网络商家网上购物商品质量销售技巧

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自然写作

Increased oxygen consumption and OXPHOS potential in superhealer mesenchymal stem cells

作者： (美)米罗(Ezra wasserman Mitchell)

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Cell Regeneration 2012年第1期1卷 11-18页

作者： Curtis C Hughey Maria P Alfaro Darrell D Belke Jeffery N Rottman Pampee P Young David H wasserman Jane Shearer Department of Biochemistry and Molecular Biology Faculty of MedicineUniversity of CalgaryCalgaryABCanadaT2N 1N4 Department of Pathology School of MedicineVanderbilt UniversityNashvilleTNUSA37232 Faculty of Kinesiology University of CalgaryCalgaryABCanadaT2N 1N4 Department of Medicine Division of Cardiovascular MedicineSchool of MedicineVanderbilt UniversityNashvilleTNUSA37240 Department of Veterans Affairs Medical Center NashvilleTNUSA37232 Department of Molecular Physiology and Biophysics School of MedicineVanderbilt UniversityNashvilleTNUSA37240 University of Calgary KNB Rm 3318.2500 University Dr.NWCalgaryABCanadaT2N 1N4.

Background:Cell-based therapies show promise in repairing cardiac tissue and improving contractile performance following a myocardial infarction.Despite this,ischemia-induced death of transplanted cells remains a major hurdle to the efficacy of treatment.‘Superhealer’MRL/MpJ mesenchymal stem cells(MRL-MSCs)have been reported to exhibit increased engraftment resulting in reduced infarct size and enhanced contractile function.This study determines whether intrinsic differences in mitochondrial oxidative phosphorylation(OXPHOS)assist in explaining the enhanced cellular survival and engraftment of MRL-MSCs.Findings:Compared to wild type MSCs(WT-MSCs),mitochondria from intact MRL-MSCs exhibited an increase in routine respiration and maximal electron transport capacity by 2.0-and 3.5-fold,respectively.When routine oxygen utilization is expressed as a portion of maximal cellular oxygen flux,the MRL-MSCs have a greater spare respiratory capcity.Additionally,glutamate/malate-and succinate-supported oxygen consumption in permeabilized cells was elevated approximately 1.25-and 1.4-fold in the MRL-MSCs,respectively.Conclusion:The results from intact and permeabilized MSCs indicate MRL-MSCs exhibit a greater reliance on and capacity for aerobic metabolism.The greater capacity for oxidative metabolism may provide a protective effect by increasing ATP synthesis per unit substrate and prevent glycolysis-mediated acidosis and subsequent cell death upon transplantation into the glucose-and oxygen-deprived environment of the infarcted heart.

关键词： Energetics Mitochondria Oxidative phosphorylation Stem cells

Shifting towards a Mediterranean Diet in the US： How Far Do We Have to Go, What Are the Potential Health Benefits and Can We Get There？

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Journal of Agricultural Science and Technology(B) 2015年第8期5卷 575-587页

作者： Allison Cappelaere Righter Arlin wasserman Department of Culinary Science The Culinary Institute of America 1946 Campus Dr. #246 Hyde Park NY 12538 USA Menus of Change Sustainable Business Leadership Council The Culinary Institute of America 69 Church Street Suite 4 LenoxMA 01240 USA