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Methane and nitrous oxide emissions from a subtropical coastal embayment(Moreton Bay, Australia)

Journal of Environmental Sciences 2015年第3期27卷 82-96页

作者： Ronald S.Musenze ursula werner Alistair Grinham James Udy Zhiguo Yuan Advanced Water Management Centre (AWMC) The University of Queensland Department of Civil and Environmental Engineering College of Engineering Design Arts and Technology Makerere University School of Civil Engineering The University of Queensland Healthy Waterways Ltd.

Surface water methane （CH4） and nitrous oxide （N20） concentrations and fluxes were investigated in two subtropical coastal embayments （Bramble Bay and Deception Bay, which are part of the greater Moreton Bay, Australia）. Measurements were done at 23 stations in seven campaigns covering different seasons during 2010-2012. Water-air fluxes were estimated using the Thin Boundary Layer approach with a combination of wind and currents-based models for the estimation of the gas transfer velocities. The two bays were strong sources of both CH4 and N2O with no significant differences in the degree of saturation of both gases between them during all measurement campaigns. Both CH4 and N2O concentrations had strong temporal but minimal spatial variability in both bays. During the seven seasons, CH4 varied between 500% and 4000% saturation while N2O varied between 128 and 255% in the two bays. Average seasonal CH4 fluxes for the two bays varied between 0.5 ± 0.2 and 6.0 ± 1.5 mg CH4/（m^***） while N20 varied between 0.4 ± 0.1 and 1.6 ± 0.6 mg N2O/（m^2-day）. Weighted emissions （t CO2-e） were 63%-90% N2O dominated implying that a reduction in N2O inputs and/or nitrogen availability in the bays may significantly reduce the bays＇ greenhouse gas （GHG） budget. Emissions data for tropical and subtropical systems is still scarce. This work found subtropical bays to be significant aquatic sources of both CH4 and N2O and puts the estimated fluxes into the global context with measurements done from other climatic regions.

关键词： Greenhouse gas emissions Subtropical aquatic systems Bay Methane Nitrous oxide Uncertainty

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Mobilization of hematopoietic progenitor cells in patients with liver cirrhosis

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World Journal of Gastroenterology 2010年第2期16卷 217-224页

作者： ursula M Gehling Marc Willems Kathleen Schlagner Ralf A Benndorf Maura Dandri Jrg Petersen Martina Sterneck Joerg-Matthias Pollok Dieter K Hossfeld Xavier Rogiers Department of Hepatobiliary Surgery and Transplant Surgery University Hospital Hamburg-Eppendorf20246 HamburgGermany Department of Medicine University Hospital Hamburg-Eppendorf20246 HamburgGermany Institute of Experimental and Clinical Pharmacology and Toxicology University Hospital Hamburg-Eppendorf20246 HamburgGermany Gent University Hospital and Medical School 9000 GentBelgium

AIM:To test the hypothesis that liver cirrhosis is associated with mobilization of hematopoietic progenitor cells. METHODS:Peripheral blood samples from 72 patients with liver cirrhosis of varying etiology were analyzed by flow *** progenitor cell subsets were immunoselected and used for functional assays in vitro. Plasma levels of stromal cell-derived factor-1(SDF-1) were measured using an enzyme linked immunosorbent ***:Progenitor cells with a CD133 + /CD45 + CD14 + phenotype were observed in 61%of th *** 1%and 26%of the peripheral bloo mononuclear cells(MNCs)displayed this phenotype Furthermore,a distinct population of c-kit + progenito cells(between 1%and 38%of the MNCs)could b detected in 91%of the ***,18% of the patients showed a population of progenito cells(between 1%and 68%of the MNCs)that wa characterized by expression of breast cancer resistanc *** phenotypic analysis disclosed tha the circulating precursors expressed CXC chemokin receptor 4,the receptor for *** line with thi finding,elevated plasma levels of SDF-1 were presen in all patients and were found to correlate with th number of mobilized CD133 + progenitor cells.

关键词： CD133 antigen CD14 antigen c-kit protein Breast cancer resistance protein-1 protein Progenitor cells CXC chemokine receptor 4 Stromal cell-derived factor-1 Liver cirrhosis

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