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Exercise-induced oxidative stress:Friend or foe?

Journal of Sport and Health Science 2020年第5期9卷 415-425页

作者： Scott K.Powers Rafael Deminice Mustafa Ozdemir toshinori yoshihara Matthew P.Bomkamp Hayden Hyatt Department of Applied Physiology and Kinesiology University of FloridaGainesvilleFL32608USA Department of Physical Education State University of LondrinaLondrina10011Brazil Department of Exercise and Sport Sciences Hacettepe UniversityAnkara06800Turkey Department of Exercise Physiology Juntendo UniversityTokyo270-1695Japan

The first report demonstrating that prolonged endurance exercise promotes oxidative stress in humans was published more than 4 decades *** this discovery,many ensuing investigations have corroborated the fact that muscular exercise increases the production of reactive oxygen species(ROS)and results in oxidative stress in numerous tissues including blood and skeletal *** several tissues may contribute to exercise-induced ROS production,it is predicted that muscular contractions stimulate ROS production in active muscle fibers and that skeletal muscle is a primary source of ROS production during *** contraction-induced ROS generation is associated with(1)oxidant damage in several tissues(e.g.,increased protein oxidation and lipid peroxidation),(2)accelerated muscle fatigue,and(3)activation of biochemical signaling pathways that contribute to exercise-induced adaptation in the contracting muscle *** our understanding of exercise and oxidative stress has advanced rapidly during the last decades,questions remain about whether exercise-induced increases in ROS production are beneficial or harmful to *** review addresses this issue by discussing the site(s)of oxidant production during exercise and detailing the health consequences of exercise-induced ROS production.

关键词： Hormesis Oxidants Radicals Reactive oxygen species Skeletal muscle

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Alterations in renin-angiotensin receptors are not responsible for exercise preconditioning of skeletal muscle fibers

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Sports Medicine and Health Science 2021年第3期3卷 148-156页

作者： Branden L.Nguyen toshinori yoshihara Rafael Deminice Jensen Lawrence Mustafa Ozdemir Hayden Hyatt Scott K.Powers Department of Applied Physiology and Kinesiology University of FloridaGainesville32611FloridaUSA Department of Exercise Physiology Juntendo UniversityTokyo270-1695Japan Department of Physical Education University of Estadual of LondrinaLondrina10011Brazil

Endurance exercise training promotes a protective phenotype in skeletal muscle known as exercise *** preconditioning protects muscle fibers against a variety of threats including inactivity-induced muscle *** mechanism(s)responsible for exercise preconditioning remain unknown and are explored in these ***,we investigated the impact of endurance exercise training on key components of the renin-angiotensin system(RAS).The RAS was targeted because activation of the classical axis of the RAS pathway via angiotensinⅡtypeⅠreceptors(AT1Rs)promotes muscle atrophy whereas activation of the non-classical RAS axis via Mas receptors(MasRs)inhibits the atrophic signaling of the classical RAS *** by prior studies,we hypothesized that an exercise-induced decrease in AT1Rs and/or increases in MasRs in skeletal muscle fibers is a potential mechanism responsible for exercise *** endurance exercise training in rats,we examined the abundance of AT1Rs and MasRs in both locomotor and respiratory *** results indicate that endurance exercise training does not alter the protein abundance of AT1Rs or MasRs in muscle fibers from the diaphragm,plantaris,and soleus muscles compared to sedentary controls(p>0.05).Furthermore,fluorescent angiotensinⅡ(AngⅡ)binding analyses confirm our results that exercise pre-conditioning does not alter the protein abundance of AT1Rs in the diaphragm,plantaris,and soleus(p>0.05).This study confirms that exercise-induced changes in RAS receptors are not a key mechanism that contributes to the beneficial effects of exercise preconditioning in skeletal muscle fibers.

关键词： Exercise preconditioning Renin-angiotensin system Skeletal muscle disuse atrophy Skeletal muscle

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Effect of a combination of astaxanthin supplementation, heat stress, and intermittent reloading on satellite cells during disuse muscle atrophy

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Journal of Zhejiang University-Science B(Biomedicine & Biotechnology) 2018年第11期19卷 844-852页

作者： toshinori yoshihara Takao SUGIURA Nobuyuki MIYAJI Yuki YAMAMOTO Tsubasa SHIBAGUCHI Ryo KAKIGI Hisashi NAITO Katsumasa GOTO Daijiro OHMORI Toshitada YOSHIOKA Graduate School of Health and Sports Science Juntendo UniversityInzaiChiba 270-1695Japan Faculty of Education Yamaguchi UniversityYamaguchiYamaguchi 753-8513Japan Toyo Koso Kagaku Co.Ltd. UrayasuChiba 279-0041Japan Institute of Health and Sports Science Kurume UniversityKurumeFukuoka 839-8502Japan Institute of Liberal Arts and Science Kanazawa UniversityKanazawaIshikawa 920-1192Japan Faculty of Medicine Juntendo UniversityBunkyo-kuTokyo 113-0033Japan Graduate School of Health Sciences Toyohashi SOZO UniversityToyohashiAichi 440-8511Japan Department of Chemistry School of MedicineJuntendo UniversityInzaiChiba 270-1695Japan Hirosaki Gakuin University HirosakiAomori 036-8577Japan

We examined the effect of a combination of astaxanthin （AX） supplementation, repeated heat stress, and intermittent reloading （IR） on satellite cells in unloaded rat soleus muscles. Forty-nine male Wistar rats （8-week-old） were divided into control, hind-limb unweighting （HU）, IR during HU, IR with AX supplementation, IR with repeated heat stress （41.0-41.5 ℃ for 30 min）, and IR with AX supplementation and repeated heat stress groups. After the experimental period, the antigravitational soleus muscle was analyzed using an immunohistochemical technique. Our results revealed that the combination of dietary AX supplementation and heat stress resulted in protection against disuse muscle atrophy in the soteus muscle. This protective effect may be partially due to a higher satellite cell number in the atrophied soleus muscle in the IR/AX/heat stress group compared with the numbers found in the other groups. We concluded that the combination treatment with dietary AX supplementation and repeated heat stress attenuates soleus muscle atrophy, in part by increasing the number of satellite cells.

关键词： Antioxidant astaxanthin Disuse muscle atrophy Heat stress Satellite cell

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