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世界核心医学期刊文摘（心脏病学分册） 2007年第7期3卷 23-24页

作者： Livanis E.g. Kostopoulou A. theodorakis g.n. 杜媛 2nd Department of Cardiology Onassis Cardiac Surgery Center Attikon Hospital Athens Greece Dr.

晕厥是严重心力衰竭患者预后不良的征象。扩张型心肌病（DC）患者出现晕厥的原因尚未完全阐明，即使标准化评估后仍不明确。本研究的目的为观察神经介导机制在DC患者晕厥发作的病理生理学中的作用。26例左室射血分数≤40％的DC患者（21... 详细信息

晕厥是严重心力衰竭患者预后不良的征象。扩张型心肌病（DC）患者出现晕厥的原因尚未完全阐明，即使标准化评估后仍不明确。本研究的目的为观察神经介导机制在DC患者晕厥发作的病理生理学中的作用。26例左室射血分数≤40％的DC患者（21例男性，5例女性，平均年龄59±2岁，极差38～79岁）人选本研究。13例发生不明原因晕厥或前晕厥的患者及13例无不明原因晕厥的患者进行氯米帕明激发直立倾斜试验。两组患者在年龄、性别和左室射血分数方面相匹配，所采用药物治疗也没有显著差别。倾斜试验过程中进行心率变异性分析和前臂血流体积描记，同时采集血样测定儿茶酚胺浓度。

关键词：扩张型心肌病患者不明原因晕厥神经心源性特发性左室射血分数直立倾斜试验心率变异性分析心力衰竭患者

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ATHLETES’ SELF-TALK,COACHIng BEHAVIOUR AnD SIgnIFICAnT OTHERS’ POSITIVE AnD nEgATIVE STATEMEnTS

ATHLETES’ SELF-TALK,COACHING BEHAVIOUR AND SIGNIFICANT OTHE...

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The 2004 Pre-Olympic Congress

作者： Zourbanos n theodorakis Y Department of Physical Education and Sport Sciences University of Thessaly

＜正＞Introduction The important role of thoughts in the form of self-talk on emotions and behaviour has received significant support by researchers[1,2].Based on the review of multiple studies on the beneficial effects of self-talk on performance[3,4,5,6],no research to our knowledge in sport settings,has investigated the possible effects of coaches’behaviour and

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Thalidomide ameliorates portal hypertension via nitric oxide synthase independent reduced systolic blood pressure

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World Journal of gastroenterology 2015年第14期21卷 4126-4135页

作者： nicholas g theodorakis Yining.n.Wang Vyacheslav A Korshunov Mary A Maluccio nicholas J Skill Division of Transplant Surgery Department of SurgerySchool of MedicineIndiana University School of Medicine University of Rochester

AIM: Portal hypertension is a common complication of liver cirrhosis and significantly increases mortality and *** reports have suggested that the compound thalidomide attenuates portal hypertension(PHT).However, the mechanism for this action is not fully *** hypothesis is that thalidomide destabilizes tumor necrosis factor α(TnFα) mR nA and therefore diminishes TnFα induction of nitric oxide synthase(nOS) and the production of nitric oxide(nO).To examine this hypothesis, we utilized the murine partial portal vein ligation(PVL) PHT model in combination with endothelial or inducible nOS isoform g.n. knockout ***: Wild type, inducible nitric oxide synthase(i nOS)-/- and endothelial nitric oxide synthase(e nOS)-/-mice received either PVL or sham surgery and were given either thalidomide or *** nitrate(total nitrate, nOx) was measured daily for 7 d as a surrogate of nO *** TnFα level was quantified by enzyme-linked immunosorbent ***α m RnA was quantified in liver and aorta tissue by reverse transcription-polymerase chain *** was determined by recording splenic pulp pressure(SPP) and abdominal aortic flow after 0-7 *** to thalidomide was determined by measurement of SPP and mean arterial pressure(MAP).RESULTS: SPP, abdominal aortic flow(Qao) and plasma nOx were increased in wild type and i nOS-/-PVL mice when compared to sham operated control *** contrast, SPP, Qao and plasma nOx were not increased in e nOS-/- PVL mice when compared to sham *** TnFα level in both sham and PVL mice was below the detection limit of the commercial ELISA ***, the effect of thalidomide on serum TnFα levels was undetermined in wild type, e nOS-/-or i nOS-/- *** acutely increased plasma nOx in wild type and e nOS-/- mice but not i nOS-/-***, thalidomide temporarily(0-90 min) decreased mean arterial pressure, SPP and Qao in wild type, e nOS-/- and i nOS-/- PVL mice, aft

关键词： Portal hypertension Thalidomide nitric oxide Knock

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novel approaches for the treatment of ventricular tachycardia

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World Journal of Cardiology 2018年第7期10卷 52-59页

作者： Michael Spartalis Eleftherios Spartalis Eleni Tzatzaki Diamantis I Tsilimigras Demetrios Moris Christos Kontogiannis Efthimios Livanis Dimitrios C Iliopoulos Vassilis Voudris george n theodorakis ESC Working group on Cardiac Cellular Electrophysiology Sophia Antipolis Cedex 06903France Division of Cardiology Onassis Cardiac Surgery CenterAthens 17674Greece Laboratory of Experimental Surgery and Surgical Research University of Athens Medical SchoolAthens 11527Greece Department of Surgery Duke UniversityDurhamNC 27710United States Department of Clinical Therapeutics “Alexandra”HospitalUniversity of AthensAthens 11528Greece

Ventricular tachycardia(VT) is a crucial cause of sudden cardiac death(SCD) and a primary cause of mortality and morbidity in patients with structural cardiac disease. VT includes clinical disorders varying from benign to lifethreatening. Most life-threatening episodes are correlated with coronary artery disease, but the risk of SCD varies in certain populations, with various underlying heart conditions, specific family history, and g.n.tic variants. The targets of VT management are symptom alleviation, improved quality of life, reduced implantable cardioverter defibrillator shocks, prevention of reduction of left ventricular function, reduced risk of SCD, and improved overall survival. Antiarrhythmic drug therapy and endocardial catheter ablation remains the cornerstone of guideline-endorsed VT treatment strategies in patients with structural cardiac abnormalities. novel strategies such as epicardial ablation, surgical cryoablation, transcoronary alcohol ablation, pre-procedural imag.n., and stereotactic ablative radiotherapy are an appealing area of res-earch. In this review, we gathered all recent advances in innovative therapies as well as experimental evidence focusing on different aspects of VT treatment that could be significant for future favorable clinical applications.

关键词： Ventricular tachycardia Catheter ablation Epicardial Sudden cardiac death novel techniques Substrate

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