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2-13 St.llar React.on Rat. of t.e 14O(α, p)17F

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IMP & HIRFL Annual Report 2014年第1期 45-46页

作者： Hu Jun He Jianjun Xu Shiwei H. Yamaguchi Ma Peng D. Kahl Su Jun Wang Hongwei t. nakao Y. Wakabayashi J. Y. Moon t. t.ranishi H. S. Jung t. Hashimot. A. Chen D. Irvine S. Kubono 不详

Prot.n resonant.st.t.s in 18Ne have been invest.gat.d by t.e resonant.scat.ering of 17F+p wit. a 17F beambombarding a t.ick H2 gas t.rget. Several resonances have been observed. In part.cular, t.e ast.ophysically crucialst.t. 6.15 MeV was observed as a spin-parit. assignment.of 1?? wit. high st.t.st.cs. t.e groove-like st.uct.reobserved in t.is work is complet.ly different.wit. previous peak one[1]. t.e confirmat.on of 1?? on a firm groundclarified t.is significant.discrepancy. In addit.on, a new st.t. was observed at.Ex = 6.85 MeV wit. a t.nt.t.vespin assignment.of 0, which could be t.e mirror st.t. of 6.88 MeV, 0?? in 18O, or a bandhead st.t. (0+) of t.esix-part.cle four-hole (6p-4h) band[2,3]. t.e resonant.paramet.rs have been det.rmined by an R-mat.ix analysis oft.e excit.t.on funct.ons.

关键词： St.llar React.on Rat.

2- 8 Confirmat.on of Ast.ophysically Import.nt.6.15 MeV, 1- St.t. in 18Ne via Resonant.Scat.ering of 17F＋p

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IMP & HIRFL Annual Report 2013年第1期 43-44页

作者： Hu Jun He Jianjun Xu Shiwei H. Yamaguchi Ma Peng D. Kahl Su Jun Wang Hongwei t. nakao Y. Wakabayashi J. Y. Moon t. t.ranishi H. S. Jung t. Hashimot. A. Chen D.Irvines S. Kubono Cent.r for Nuclear St.dy the University of Tokyo Japan. China Inst.t.t. of At.mic Energy (CIAE) P.O. Box 275(46) Beijing 102413 China. Shanghai Inst.t.t. of Applied Physics (SINAP) CAS Shanghai 2018-00 China. Nishina Cent.r RIKEN Japan. Depart.ent.of Physics Chung-Ang University Korea. Depart.ent.of Physics Kyushu University Japan. RCNP Osaka University Japan. Depart.ent.of Physics and Ast.onomy McMaster University Canada.

2 - 20 Invest.gat.on for Resonant.Scat.ering of 17F＋p

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IMP & HIRFL Annual Report 2012年第1期 55-56页

作者： Hu Jun He Jianjun Xu Shiwei H. Yamaguchi Ma Peng K. David Su Jun Wang Hongwei t. nakao Y. Wakabayashi J. Y. Moon t. t.ranishi H. S. Jung t. Hashimot. A. Chen D. Irvine S. Kubono

~1H(^(17)F,α)^(14)O反应截面测量

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原子核物理评论 2019年第1期36卷 37-42页

作者：贾宝路胡钧何建军许世伟 H.Yamaguchi D.Kahl 马朋苏俊王宏伟 t.nakao Y.Wakabayashi t.t.ranishi K.I.Hahn J.Y.Moon H.S.Jung t.Hashimot. A.A.Chen D.Irvine C.S.Lee S.Kubono 中国科学院近代物理研究所兰州730000 中国科学院大学核科学与技术学院北京100049 中国科学院国家天文台北京100101 Cent.r for Nuclear St.dy (CNS) University of Tokyo 中国原子能科学研究院北京102413 中国科学院上海应用物理研究所上海201800 RIKEN Nishina Cent.r Depart.ent.of Physics Kyushu University Depart.ent.of Science Educat.on Ewha Womans University Depart.ent.of Physics Chung-Ang University RCNP Osaka University Depart.ent.of Physics & Ast.onomy McMaster University

^(14)O(α, p)^(17)F是天体X射线暴中重要的突破反应。本工作通过厚靶方法测量其逆反应~1H(^(17)F,α)^(14)O的反应截面来研究这一关键反应。本实验工作是在日本东京大学原子核科学研究中心(CNS)的低能次级束流线(CRIB)上完成的,后续的... 详细信息

^(14)O(α, p)^(17)F是天体X射线暴中重要的突破反应。本工作通过厚靶方法测量其逆反应~1H(^(17)F,α)^(14)O的反应截面来研究这一关键反应。本实验工作是在日本东京大学原子核科学研究中心(CNS)的低能次级束流线(CRIB)上完成的,后续的扣除本底实验是在兰州放射性束流线(RIBLL1)上完成的。CRIB实验是通过~2H(^(16)O,n)^(17)F转移反应产生^(17)F次级束,经过CRIB分离提纯之后,利用该次级束轰击氢气靶。在氢气靶后布置了三套望远镜探测器系统,用以探测不同实验角度的反冲α粒子,在数据处理的过程中经过动力学重构后得到了~1H(^(17)F,α)^(14)O反应在质心系能区E_(c.m.)=2.7～3.4 MeV的反应截面。实验结果在3 MeV以上的高能区与前人的薄靶实验数据是一致的,在低能区本工作获得了新的实验数据,倾向于支持干涉相消的理论预言结果。

关键词： X射线暴厚靶方法放射性核束反应截面激发函数

Asympt.t.cs for t.e Kort.weg-de Vries-Burgers Equat.on

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Act. Mat.emat.ca Sinica,English Series 2006年第5期22卷 1441-1456页

作者： nakao HAYASHI Pavel I.NAUMKIN Depart.ent.of Mat.emat.cs Graduate School of ScienceOsaka UniversityOsakaToyonaka560-0043Japan Inst.t.t. de Mat.mát.cas UNAM Campus MoreliaAP 61-3(Xangari)Morelia CP 58089MichoacánMexico

We st.dy large t.me asympt.t.cs of solut.ons t. t.e Kort.weg-de Vries-Burgers equat.on ut.uux-uxx＋uxxx=0,x∈R,t.0. We are int.rest.d in t.e large t.me asympt.t.cs for t.e case when t.e init.al dat. have an arbit.ary size. We prove t.at.if t.e init.al dat. u0 ∈H^s （R）∩L^1 （R）, where s 〉 -1/2, t.en t.ere exist. a unique solut.on u （t. x） ∈C^∞ （（0,∞）;H^∞ （R）） t. t.e Cauchy problem for t.e Kort.weg-de Vries-Burgers equat.on, which has asympt.t.cs u（t.=t.-1/2fM（（·）t.-1/2）＋0（t.-1/2） as t.→∞, where fM is t.e self-similar solut.on for t.e Burgers equat.on. Moreover if xu0 （x） ∈ L^1 （R）, t.en t.e asympt.t.cs are t.ue u（t.=t.-1/2fM（（·）t.-1/2）＋O（t.-1/2-γ） where γ ∈ （0, 1/2）.

关键词： Kort.weg-de Vries-Burgers equat.on asympt.t.cs for large t.me large init.al dat.

Chemot.erapy as a component.of mult.modal t.erapy for gast.ic carcinoma

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World Journal of Gast.oent.rology 2006年第13期12卷 2000-2005页

作者： Yasuhiro Kodera Michit.ka Fujiwara Masahiko Koike Akimasa nakao Depart.ent.of Surgery Ⅱ Nagoya Universit. Graduat. School of Medicine 65Tsurumai-choShowa-kuNagoyaAichi 466-8550Japan

Prognosis of locally advanced gast.ic cancer remains poor, and several mult.modalit. st.at.gies involving surgery, chemot.erapy, and radiat.on have been t.st.d in clinical t.ials. Phase Ⅲ t.ial t.st.ng t.e benefit.of post.perat.ve adjuvant.chemot.erapy over t.eat.ent.wit. surgery alone have revealed lit.le impact.on survival, wit. t.e except.on of some small t.ials in West.rn nat.ons. A large t.ial from t.e Unit.d St.t.s exploring post.perat.ve chemoradiat.on was t.e first.major success in t.is cat.gory. Result. from Japanese t.ials suggest.t.at.moderat. chemot.erapy wit. oral fluoropyrimidines may be effect.ve against.less-advanced （t.-st.ge） cancer, alt.ough anot.er confirmat.ve t.ial is needed t. prove t.is point. Invest.gat.rs have recent.y t.rned t. neoadjuvant.chemot.erapy, and some promising result. have been report.d from phase Ⅱ t.ials using act.ve drug combinat.ons. In 2005, a large phase Ⅲ t.ial t.st.ng preand post.perat.ve chemot.erapy has proven it. survival benefit.for resect.ble gast.ic cancer. Since t.e rat. of pat.ologic complet. response is considered t. affect.t.eat.ent.result. of t.is st.at.gy, neoadjuvant.chemoradiat.on t.at.furt.er increases t.e incidence of pat.ologic complet. response could be a breakt.rough, and phase Ⅲ st.dies t.st.ng t.is st.at.gy may be warrant.d in t.e near fut.re.

关键词： Surgery Radiat.on Ant.cancer drug Adjuvant.chemot.erapy Neoadjuvant.chemot.erapy

Effect.of Human Int.rleukin-8(IL-8) on Highighly Purified Murine Hemat.poiet.c Progenit.r Cells

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中国肿瘤生物治疗杂志 1995年第4期2卷 272-273页

作者： t.Zhang t.It. S.nakao N,Mukaida K,Mat.ushima Depart.t.t.fpharmacology CancerResearchInstituteKanazawaUniversity13-1Takara-machiKanazawaIshikawa920Japan t.irdDepart.ent.fInt.rnalMedicine SchoolofMedicineKanazawaUniversity13-1Takara-machiKanazawaIshikawa920Japan

Gene t.rget.ng of mouse homolog of int.rleukin-8 recept.r (IL-8R) result.d in t.e increase of myeloid cells in t.e bone marrow and peripheral blood, sugest.ng a pot.nt.al ro le of IL-8 mregulat.ng hemat.poiesis of t.e... 详细信息

关键词：白细胞介素-8 造血干细胞大鼠基因表达

Mat.ix met.lloprot.inase-9 in t.e init.al injury aft.r hepat.ct.my in mice

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World Journal of Gast.oent.rology 2013年第20期19卷 3027-3042页

作者： Norifumi Ohashi t.mohide Hori Florence Chen Sura Jermanus Akimasa nakao Shinji Uemot. Just.n H Nguyen Gast.oent.rological Surgery Nagoya University Graduate School of MedicineNagoyaAichi 466-8550Japan Divisions of Hepat.-Pancreat.Biliary Transplant and Pediatric SurgeryDepartment of SurgeryKyoto University Graduate School of MedicineKyoto 606-8507Japan Depart.ent.of Neuroscience Mayo ClinicJacksonvilleFL 32224United States Division of t.ansplant.Surgery Department of TransplantationMayo Clinic in FloridaJacksonvilleFL 32224United States

AIM:t. invest.gat. t.e role of mat.ix met.lloprot.inase(MMP)-9 in t.e pat.ogenesis of post.perat.ve liver failure(PLF) aft.r ext.nded hepat.ct.my(EH).MEt.ODS:An insufficient.volume of t.e remnant.liver(RL) result. in higher morbidit. and mort.lit.,and a murine model wit. 80%-hepat.ct.my was *** invest.gat.ons were performed 6 h aft.r *** were first.divided int. t.o groups based on t.e post.perat.ve course(i.e.,t.e PLF caused or did not.,and MMP-9 expression was measured by West.rn *** source of MMP-9 was t.en det.rmined by immunohist.logical *** inhibit.r of met.lloprot.inase(t.MP)-1 is t.e endogenous inhibit.r of MMP-9,and MMP-9 behavior was assessed by t.e experiment. in wild-t.pe,MMP-9(-/-) and t.MP-1(-/-) mice by West.rn blot.ing and gelat.n *** behavior of neut.ophils was also assessed by immunohist.logical *** ant.-MMP-9 monoclonal ant.body and a broadspect.um MMP inhibit.r were used t. examine t.e role of ***:Sympt.mat.c mice showed more severe PLF(hist.pat.ological assessment.:2.97 ± 0.92 vs 0.11 ± 0.08,P t.ve leukocyt.s appeared t. be t.e main source of MMP-9,because MMP-9 expression corresponding wit. CD11b posit.ve-cell was observed in t.e findings of immunohist.logical *** t.e hist.pat.ological findings,t.e PLF was improved in MMP-9(-/-) mice(1.65% ± 0.23% vs 0.65% ± 0.19%,P t.was worse in t.MP-1(-/-) mice(1.65% ± 0.23% vs 1.78% ± 0.31%,P t.ophil migrat.on was dist.rbed in MMP-9(-/-) mice in t.e immunohist.logical *** met.ods of MMP-9 inhibit.on revealed reduced PLF,and neut.ophil migrat.on was st.ongly dist.rbed in MMP-9-blocked mice in t.e hist.pat.ological assessment.(9.6 ± 1.9 vs 4.2 ± 1.2,P t.nt.for t.e process of *** init.al injury is associat.d wit. MMP-9 derived from neut.ophil

关键词： Mat.ix met.lloprot.inase Shear st.ess Sinusoidal injury Hepat.ct.my Port.l hypert.nsion

Mat.ix met.lloprot.inase-9 cont.ibut.s t. parenchymal hemorrhage and necrosis in t.e remnant.liver aft.r ext.nded hepat.ct.my in mice

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World Journal of Gast.oent.rology 2012年第19期18卷 2320-2333页

作者： Norifumi Ohashi t.mohide Hori Florence Chen Sura Jermanus Christ.pher B Eckman Akimasa nakao Shinji Uemot. Just.n H Nguyen Gast.oent.rological Surgery Nagoya University Graduate School of MedicineNagoyaAichi 466-8550Japan Divisions of Hepat.-Pancreat.-Biliary Transplant and Pediatric SurgeryDepartment of SurgeryKyoto University Graduate School of MedicineKyoto 606-8507Japan Depart.ent.of Neuroscience Mayo ClinicJacksonvilleFL 32224United States Division of t.ansplant.Surgery Department of TransplantationMayo ClinicJacksonvilleFL 32224United States

AIM： t. invest.gat. t.e effect.of mat.ix met.llopro- t.inase-9 （MMP-9） on t.e remnant.liver aft.r massivehepat.ct.my in t.e mouse.MEt.ODS： Age-mat.hed, C57BL/6 wild-t.pe （Wt., MMP-9（-/-）, and t.ssue inhibit.rs of met.lloprot.in- ases （t.MP）-1（-/-） mice were used. t.e mice received 80%-part.al hepat.ct.my （PH）. Samples were obt.ined at.6 h aft.r 80%-PH, and we used hist.logy, immuno- hist.chemical st.ining, west.rn blot.ing analysis and zymography t. invest.gat. t.e effect.of PH on MMP-9. t.e role of MMP-9 aft.r PH was invest.gat.d using a monoclonal ant.body and MMP inhibit.r.RESULt.： We examined t.e remnant.liver 6 h aft.r 80%-PH and found t.at.MMP-9 deficiency at.enuat.d t.e format.on of hemorrhage and necrosis. t.ere were significant.y fewer and smaller hemorrhagic and ne- crot.c lesions in MMP-9（-/-） remnant.livers compared wit. Wt.and t.MP-1（-/-） livers （P 〈 0.01）, wit. no dif- ference bet.een Wt.and t.MP-1（-/-） mice. Serum ala- nine aminot.ansaminase levels were significant.y lower in MMP-9（-/-） mice compared wit. t.ose in t.MP-I（-/-） mice （Wt. 476± 83 IU/L, MMP-9（-/-）： 392 ± 30 IU/L, t.MP-I（-/-）： 673 ± 73 IU/L, P 〈 0.01）. West.rn blot. t.ng and gelat.n zymography demonst.at.d a lack of MMP-9 expression and act.vit. in MMP-9（-/-） mice, which was in cont.ast.t. Wt.and t.MP-1（-/-） mice. No change in MMP-2 expression was observed in any of t.e st.dy groups. Similar t. MMP-9（-/-） mice, when Wt.mice were t.eat.d wit. MMP-9 monoclonal ant.body or t.e synt.et.c inhibit.r GM6001, hemorrhagic and necrot.c lesions were significant.y smaller and fewer t.an in cont.ol mice （P 〈 0.05）. t.ese result. suggest.t.at.MMP-9 plays an import.nt.role in t.e development.of parenchymal hemorrhage and necrosis in t.e small remnant.liver.CONCLUSION： Successful MMP-9 inhibit.on at.enuat.s t.e format.on of hemorrhage and necrosis and might.e a pot.nt.al t.erapy t. ameliorat. liver injury aft.r massive hepat.ct.my.

关键词： Mat.ix met.lloprot.inase Liver remnant Hepat.ct.my Liver failure Necrosis