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Evaluation of ruminal methane and ammonia formation and microbiota composition as affected by supplements based on mixtures of tannins and essential oils using Rusitec

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Journal of Animal Science and Biotechnology 2024年第4期15卷 1607-1622页

作者： Giulia Foggi Melissa Terranova Matteo Daghio sergej l.amel.hanka Giuseppe Conte Simon Ineichen Monica Agnolucci Carlo Viti Alberto Mantino Arianna Buccioni Michael Kreuzer Marcello Mele Department of Agriculture Food and Environment(DAFE)University of Pisa56124 PisaItaly AgroVet‑Strickhof ETH ZurichLindauSwitzerland Department of Agriculture FoodEnvironment and ForestryUniversity of FlorenceFlorence 50144Italy Centro Di Ricerche Agro‑Ambientali“E.Avanzi” University of PisaPisa 56122Italy School of Agricultural Forest and Food Sciences HAFLBern University of Applied SciencesZollikofenSwitzerland Institute of Agricultural Sciences ETH ZurichLindauSwitzerland

Background Dietary supplements based on tannin extracts or essential oil compounds(EOC)have been repeatedly reported as a promising feeding strategy to reduce the environmental impact of ruminant husbandry.A previous batch culture screening of various supplements identified selected mixtures with an enhanced potential to mitigate ruminal methane and ammonia *** these,Q-2(named after quebracho extract and EOC blend 2,composed of carvacrol,thymol,and eugenol)and C-10(chestnut extract and EOC blend 10,consisting of oregano and thyme essential oils and limonene)have been investigated in detail in the present study with the semi-continuous rumen simulation technique(Rusitec)in three independent *** this purpose,Q-2 and C-10,dosed according to the previous study,were compared with a non-supplemented diet(negative control,NC)and with one supplemented with the commercial EOC-based Agolin^(R) Ruminant(positive control,PC).Results From d 5 to 10 of fermentation incubation liquid was collected and analysed for pH,ammonia,protozoa count,and gas *** residues were collected for the determination of ruminal *** d 10,samples of incubation liquid were also characterised for bacterial,archaeal and fungal communities by high-throughput sequencing of 16S rRNA and 26S ribosomal large subunit gene *** of the duration of the fermentation period,Q-2 and C-10 were similarly efficient as PC in mitigating either ammonia(-37%by Q-2,-34%by PC)or methane formation(-12%by C-10,-12%by PC).The PC was also responsible for lower feed degradability and bacterial and fungal richness,whereas Q-2 and C-10 effects,particularly on microbiome diversities,were limited compared to *** All additives showed the potential to mitigate methane or ammonia formation,or both,in vitro over a period of 10 ***,several differences occurred between PC and Q-2/C-10,indicating different mechanisms of *** pronounced defaunation caused by PC a

关键词： Additive Bacteria Continuous culture Degradability Fermentation Fungi Greenhouse gas In vitro Microbiome Rumen

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解决一维材料黏附行为的实验方法综述

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Engineering 2023年第5期24卷 39-72,I0001,I0002页

作者： James l.Mead Shiliang Wang Sören Zimmermann sergej Fatikow Han Huang Department of Computing Science University of OldenburgOldenburg D-26129Germany School of Physics and Electronics Central South UniversityChangsha 410083China School of Mechanical and Mining Engineering The University of QueenslandBrisbaneQLD 4072Australia

一维(1D)材料(如纳米管和纳米线)的黏附行为在集成一维组件的新型器件以及基于一维阵列的仿生黏合剂的有效制造、功能和可靠性中起着决定性作用。本文汇编并批判性地评估了最近的实验技术,旨在表征由一维材料形成的界面的粘附行为(包括... 详细信息

一维(1D)材料(如纳米管和纳米线)的黏附行为在集成一维组件的新型器件以及基于一维阵列的仿生黏合剂的有效制造、功能和可靠性中起着决定性作用。本文汇编并批判性地评估了最近的实验技术,旨在表征由一维材料形成的界面的粘附行为(包括当这些材料与衬底或邻近的一维材料接触时)。讨论了一维材料与表面的构象以及与之相关的多粗糙度接触的发生,并探讨了界面附着和剥离过程中黏附和摩擦的耦合。考虑了一维材料作为增强剂在纳米复合材料中的应用以及相关的界面表征技术。研究了样品制备和黏附测试过程中存在的环境条件对一维界面相互作用的潜在影响,并最终改变了一维材料的粘附行为。最后,简要介绍了当前的挑战和未来的方向,包括测试环境的系统调查和通过表面改性改变附着力。

关键词：一维材料纳米复合材料一维阵列界面相互作用样品制备表面改性新型器件增强剂

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Transcatheter Ventricular Septal Defect Closure with Nit-Occlud lêVSD Device—Five Years’Experience and literature Review

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Congenital Heart Disease 2023年第3期18卷 361-371页

作者： Ivana B.Cerović Vladislav A.Vukomanović Jovan lj.Košutić Mila S.Stajević Sanja S.Ninić Saša S.Popović Ivan D.Dizdarević Staša D.Krasić sergej M.Prijić Cardiology Department Mother and Child Health Institute of Serbia“Dr VukanČupić”Belgrade11070Serbia Faculty of Medicine University of BelgradeBelgrade11070Serbia

Introduction:Transcatheter closure is an alternative to ventricular septal defect(VSD)occlusion ***-Occlud lêVSD coil is a new device yet to be *** study aimed to evaluate immediate and midterm results after transcatheter closure with the Nit-Occlud lêVSD ***:The retrospective analysis included 30 patients with VSD referred for closure during the period from October 2015 to December ***:At the time of intervention,the patients’mean age and body weights were 7.5±5.6 years and 29.3±19.1 *** majority of the defects had perimembranous location(24/30),four defects had muscular and two outlet subaortic *** mean effective right-side diameter of the VSDs was 3.6±1.3 *** ventricular fibrillation,device embolization,and hemolysis developed in different patients and were successfully *** of the patients had a complete atrioventricular *** coil was successfully placed in 25/30(83.3%)*** majority of the devices were 10 mm×6 mm(11/25)and 12 mm×8 mm(8/25)in *** patients required the implantation of a second *** follow-up period was 2.1±1.4 *** VSD closure was achieved in 48%of cases immediately after the intervention,74%during 2.1±1.6 months after the procedure,and 81%over *** remaining patients had a trivial residual *** the follow-up,approximately one-third of patients developed trivial aortic and mitral valve regurgitation,and half of the patients acquired trace/mild tricuspid ***(z-score)left ventricular end-diastolic diameter(0.15±0.37 vs.0.92±0.82,p=0.005)and left atrium dimension(0.47±0.58 vs.1.89±1.11,p=0.005),as well as the left atrium to aortic root ratio(1.2±0.1 vs.1.4±0.2,p=0.005)showed a significant decrease over follow-up related to the period before ***:Intervention with Nit-OccludlêVSD coil showed appropriate results regarding VSD closure rate,complications,and chamber *** introduction of t

关键词： Ventricular septal defect transcatheter device children

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Activation of RAS/MAPK pathway confers MCl-1 mediated acquired resistance to BCl-2 inhibitor venetoclax in acute myeloid leukemia

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Signal Transduction and Targeted Therapy 2022年第3期7卷 856-868页

作者： Qi Zhang Bridget Riley-Gillis lina Han Yanan Jia Alessia lodi Haijiao Zhang Saravanan Ganesan Rongqing Pan sergej N.Konoplev Shannon R.Sweeney Jeremy A.Ryan Yulia Jitkova Kenneth Dunner Jr Shaun E.Grosskurth Priyanka Vijay Sujana Ghosh Charles lu Wencai Ma Stephen Kurtz Vivian R.Ruvolo Helen Ma Connie CWeng Cassandra lRamage Natalia Baran Ce Shi Tianyu Cai Richard Eric Davis Venkata l.Battula Yingchang Mi Jing Wang Courtney D.DiNardo Michael Andreeff Jeffery W.Tyner Aaron Schimmer Anthony letai Rose Ann Padua Carlos E.Bueso-Ramos Stefano Tiziani Joel leverson Relja Popovic Marina Konopleva Department of leukemia The University of Texas MD Anderson Cancer CenterHoustonTXUSA AbbVie Inc. North ChicagoILUSA Institute of Hematology Chinese Academy of Medical Sciences&Peking Union Medical CollegeTianjinChina Department of Nutritional Sciences Department of PediatricsDepartment of OncologyDell Medical SchoolThe University of Texas at AustinAustinTX 78712USA Department of Cell Developmental&Cancer BiologyDivision of Hematology&Medical OncologyKnight Cancer InstituteOregon Health&Science UniversityPortlandORUSA Universite de Paris Institut de la Recherche Saint-Louis(IRSL)Inserm Unit 1131ParisFrance Dana-Farber Cancer Institute BostonMAUSA Department of Hematopathology The University of Texas MD Anderson Cancer CenterHoustonTXUSA Princess Margaret Cancer Center TorontoONCanada High Resolution Electron Microscopy Facility The University of Texas MD Anderson Cancer CenterHoustonTXUSA Department of Bioinformatics&Computational Biology The University of Texas MD Anderson Cancer CenterHoustonTXUSA Department of Hematology The First Hospital Affiliated Harbin Medical UniversityHarbinChina Department of lymphoma&Myeloma Research The University of Texas MD Anderson Cancer CenterHoustonTXUSA

Despite high initial response rates,acute myeloid leukemia(AMl)treated with the BCl-2-selective inhibitor venetoclax(VEN)alone or in combinations commonly acquires *** performed gene/protein expression,metabolomic and methylation analyses of isogenic AMl cell lines sensitive or resistant to VEN,and identified the activation of RAS/MAPK pathway,leading to increased stability and higher levels of MCl-1 protein,as a major acquired mechanism of VEN resistance.

关键词： acute myeloid acquired

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Correction: Activation of RAS/MAPK pathway confers MCl-1 mediated acquired resistance to BCl-2 inhibitor venetoclax in acute myeloid leukemia

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Signal Transduction and Targeted Therapy 2022年第5期7卷 1860-1860页

作者： Qi Zhang Bridget Riley-Gillis lina Han Yannan Jia Alessia lodi Haijiao Zhang Saravanan Ganesan Rongqing Pan sergej N.Konoplev Shannon R.Sweeney Jeremy A.Ryan Yulia Jitkova Kenneth Dunner Jr Shaun E.Grosskurth Priyanka Vijay Sujana Ghosh Charles lu Wencai Ma Stephen Kurtz Vivian R.Ruvolo Helen Ma Connie C.Weng Cassandra l.Ramage Natalia Baran Ce Shi Tianyu Cai Richard Eric Davis Venkata l.Battula Yingchang Mi Jing Wang Courtney D.DiNardo Michael Andreeff Jeffery W.Tyner Aaron Schimmer Anthony letai Rose Ann Padua Carlos E.Bueso-Ramos Stefano Tiziani Joel leverson Relja Popovic Marina Konopleva Department of leukemia The University of Texas MD Anderson Cancer Center Houston TX USA Institute of Hematology Chinese Academy of Medical Sciences & Peking Union Medical College Tianjin China Department of Hematology The First Hospital Affiliated Harbin Medical University Harbin China AbbVie Inc. North Chicago IL USA Department of Nutritional Sciences Department of Pediatrics Department of Oncology Dell Medical School The University of Texas at Austin Austin TX 78712 USA Department of Cell Developmental & Cancer Biology Division of Hematology & Medical Oncology Knight Cancer Institute Oregon Health & Science University Portland OR USA Université de Paris Institut de la Recherche Saint-Louis (IRSL) Inserm Unit 1131 Paris France Dana-Farber Cancer Institute Boston MA USA Department of Hematopathology The University of Texas MD Anderson Cancer Center Houston TX USA Princess Margaret Cancer Center Toronto ON Canada High Resolution Electron Microscopy Facility The University of Texas MD Anderson Cancer Center Houston TX USA Department of Bioinformatics & Computational Biology The University of Texas MD Anderson Cancer Center Houston TX USA Department of lymphoma & Myeloma Research The University of Texas MD Anderson Cancer Center Houston TX USA

Correction to:Signal Transduction and Targeted Therapy(2022)7:1–13,https://***/10.1038/s41392-021-00870-3 In the original version of this article,given name of 4th author Yannan Jia was incorrectly published as Yanan *** original article has been *** Access This article is licensed under a Creative Commons Attribution 4.0 International license,which permits use,sharing,adaptation,distribution and reproduction in any medium or format,as long as you give appropriate credit to the original author(s)and the source.

关键词： myeloid Therapy resistance

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