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DOUBLE WAVE DESCRIPTION OF SINGLE PARTICLE IN AN INFINITELY DEEP SQUARE POTENTIAL WELL

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Chinese Physics Letters 1990年第4期7卷 152-155页

作者： HUANG Xiangyou qian shangwu LIU Quanhui HE Zuoxiu Center of Theoretical Physics CCAST(Wor1d hb.)Beijing 100080 Department of Physics Peking UniversityBeijing 100871 Department of Physics Zhengzhou UniversityZhengzhou 450052 Institute of Theoretical Physics Academia SinicaBeijing100080

Recently we have suggested that the state of a single particle should be jointly defined by two wave *** this letter we use this suggestion to discuss the motion of a single particle in an infinitely deep square potential well;the results obiained in the classical limit correctly describe the motion of the single particle with clarity.

关键词： functions infinitely particle

Rederivation of AB Scattering Cross Section by the Method of Path Integration

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Chinese Physics Letters 1993年第4期10卷 197-200页

作者： GU Zhiyu XIE Yuan HUANG Bowen qian shangwu Capital Normal University Beiing 100053 Department of Physics Peking UniversityBeiing 100871

Taking advantage of the calculation based on the original fundamental ideas of Aharonov and Bohm(AB)on the one hand,and making some necessary revisions on the other hand,this paper rederives the famous AB scattering c... 详细信息

关键词： method derivation Scattering

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环形振子势的超对称性和形不变性

北京大学学报（自然科学版） 1999年第3期35卷 346-350页

作者：钱尚武黄博文北京大学物理学系首都师范大学物理学系

证明了在球极坐标下环形振子势在ｒ维和θ维具有超对称性和形不变性，得到了该势的能量本征值和能量本征函数。

关键词：超对称性形不变性能量本征值环形振子势

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费曼特性函数的物理解释

云南民族学院学报（自然科学版） 2001年第1期10卷 259-260,265页

作者：钱尚武北京大学物理系北京100871

笔者重温了为大多数物理学家遗忘或忽视了的费曼特性函数及其物理解释 ,指出它给出了生动的物理图象和解决某些复杂问题的洞察力 .并以另法推导极坐标下的传播子公式为例。

关键词：费曼特性函数传播子物理解释

在点正则变换下赫耳顿势映射至潘歇-推劳Ⅰ势(英文)

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北京大学学报（自然科学版） 2000年第5期36卷 637-640页

作者：钱尚武顾之雨黄博文北京大学物理学系北京100871 首都师范大学物理学系北京100037

利用在点正则变换下形不变势的映射方法 ,我们从潘歇推劳Ⅰ势的已知结果求得了赫耳顿势的束缚态能谱与本征函数。

关键词：量子力学赫耳顿势潘歇-推劳Ⅰ势点正则变换

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哈特曼势的超对称性和形不变性(英文)

北京大学学报（自然科学版） 2000年第1期36卷 78-82页

作者：钱尚武黄博文王德云顾之雨北京大学物理学系北京100871 首都师范大学物理学系北京100037

证明了在球极坐标下，哈特曼势在维度ｒ和维度θ都具有超对称性和形不变性，从而求得此势的能量本征值和能量本征函数。

关键词：哈特曼势超对称性形不变性能量本征函数

Interannual variability of boreal summer intraseasonal oscillation over the northwestern Pacific influenced by the Pacific Meridional Mode

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Atmospheric and Oceanic Science Letters 2025年第1期18卷 6-11页

作者： Haoyu Zhou Pang-Chi Hsu Lin Chen Yitian qian Key Laboratory of Meteorological Disaster Ministry of Education/Joint International Research Laboratory of Climate and Environmental Change/Collaborative Innovation Center on Forecast and Evaluation of Meteorological DisastersNanjing University of Information Science and TechnologyNanjingChina

During the boreal summer,intraseasonal oscillations exhibit significant interannual variations in intensity over two key regions:the central-western equatorial Pacific(5°S-5°N,150°E-150°W)and the subtropical Northwestern Pacific(10°-20°N,130°E-175°W).The former is well-documented and considered to be influenced by the ENSO,while the latter has received comparatively less attention and is likely influenced by the Pacific Meridional Mode(PMM),as suggested by partial correlation analysis *** elucidate the physical processes responsible for the enhanced(weakened)intraseasonal convection over the subtropical northwestern Pacific during warm(cold)PMM years,the authors employed a moisture budget *** findings reveal that during warm PMM years,there is an increase in summer-mean moisture over the subtropical northwestern *** increase interacts with intensified vertical motion perturbations in the region,leading to greater vertical moisture advection in the lower troposphere and consequently resulting in convective *** a process is pivotal in amplifying intraseasonal convection *** observational findings were further verified by model experiments forced by PMM-like sea surface temperature patterns.

关键词： Boreal summer intraseasonal oscillation Interannual variability Pacific Meridional Mode Moisture budget analysis

The pivotal role of microglia in injury and the prognosis of subarachnoid hemorrhage

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Neural Regeneration Research 2025年第7期20卷 1829-1848页

作者： Wenjing Ning Shi Lv qian Wang Yuzhen Xu Department of Rehabilitation The Second Affiliated Hospital of Shandong First Medical UniversityTaianShandong ProvinceChina Department of Central Laboratory The Affiliated Taian City Central Hospital of Qingdao UniversityTaianShandong ProvinceChina

Subarachnoid hemorrhage leads to a series of pathological changes,including vascular spasm,cellular apoptosis,blood–brain barrier damage,cerebral edema,and white matter ***,which are the key immune cells in the central nervous system,maintain homeostasis in the neural environment,support neurons,mediate apoptosis,participate in immune regulation,and have neuroprotective *** evidence has shown that microglia play a pivotal role in the pathogenesis of subarachnoid hemorrhage and affect the process of injury and the prognosis of subarachnoid ***,microglia play certain neuroprotective roles in the recovery phase of subarachnoid *** approaches aimed at modulating microglia function are believed to attenuate subarachnoid hemorrhage *** provides new targets and ideas for the treatment of subarachnoid ***,an in-depth and comprehensive summary of the role of microglia after subarachnoid hemorrhage is still *** review describes the activation of microglia after subarachnoid hemorrhage and their roles in the pathological processes of vasospasm,neuroinflammation,neuronal apoptosis,blood–brain barrier disruption,cerebral edema,and cerebral white matter *** also discusses the neuroprotective roles of microglia during recovery from subarachnoid hemorrhage and therapeutic advances aimed at modulating microglial function after subarachnoid ***,microglia in subarachnoid hemorrhage are targeted with TLR inhibitors,nuclear factor-κB and STAT3 pathway inhibitors,glycine/tyrosine kinases,NLRP3 signaling pathway inhibitors,Gasdermin D inhibitors,vincristine receptorαreceptor agonists,ferroptosis inhibitors,genetic modification techniques,stem cell therapies,and traditional Chinese ***,most of these are still being evaluated at the laboratory *** clinical studies and data on subarachnoid hemorrhage are required to improve the treatment of subarachnoid hemor

关键词： apoptosis blood–brain barrier brain edema microglia neuroinflammation neuron neuroprotection subarachnoid hemorrhage vasoconstriction white matter injury

Epigenetic regulation of the inflammatory response in stroke

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Neural Regeneration Research 2025年第11期20卷 3045-3062页

作者： Jingyi Liang Fei Yang Zixiao Li qian Li School of Basic Medical Sciences Capital Medical UniversityBeijingChina Department of Neurobiology School of Basic Medical SciencesCapital Medical UniversityBeijingChina Laboratory for Clinical Medicine Beijing Key Laboratory of Neural Regeneration and RepairCapital Medical UniversityBeijingChina Department of Neurology Beijing Tiantan HospitalCapital Medical UniversityBeijingChina China National Clinical Research Center for Neurological Diseases Beijing Tiantan HospitalCapital Medical UniversityBeijingChina National Center for Healthcare Quality Management in Neurological Diseases Beijing Tiantan HospitalCapital Medical UniversityBeijingChina Chinese Institute for Brain Research BeijingChina Research Unit of Artificial Intelligence in Cerebrovascular Disease Chinese Academy of Medical SciencesBeijingChina Beijing Engineering Research Center of Digital Healthcare for Neurological Diseases BeijingChina Department of Biochemistry and Molecular Biology School of Basic Medical SciencesCapital Medical UniversityBeijingChina Beijing Key Laboratory of Cancer Invasion and Metastasis Research Capital Medical UniversityBeijingChina

Stroke is classified as ischemic or hemorrhagic,and there are few effective treatments for either *** mechanisms play a critical role in secondary brain injury following a stroke,which manifests as cytokine release,blood–brain barrier disruption,neuronal cell death,and ultimately behavioral *** the inflammatory response has been shown to mitigate this cascade of events in experimental stroke ***,in clinical trials of anti-inflammatory agents,longterm immunosuppression has not demonstrated significant clinical benefits for *** may be attributable to the dichotomous roles of inflammation in both tissue injury and repair,as well as the complex pathophysiologic inflammatory processes in *** acute harmful inflammatory responses or inducing a phenotypic shift from a pro-inflammatory to an anti-inflammatory state at specific time points after a stroke are alternative and promising therapeutic *** agents that can modulate inflammation requires a detailed understanding of the inflammatory processes of ***,epigenetic reprogramming plays a crucial role in modulating post-stroke inflammation and can potentially be exploited for stroke *** this review,we summarize current findings on the epigenetic regulation of the inflammatory response in stroke,focusing on key signaling pathways including nuclear factor-kappa B,Janus kinase/signal transducer and activator of transcription,and mitogen-activated protein kinase as well as inflammasome *** also discuss promising molecular targets for stroke *** evidence to date indicates that therapeutic targeting of the epigenetic regulation of inflammation can shift the balance from inflammation-induced tissue injury to repair following stroke,leading to improved post-stroke outcomes.

关键词： DNA methylation histone modification intracerebral hemorrhage ischemic stroke neuroinflammation neuroprotection non-coding RNA RNA methylation subarachnoid hemorrhage treatment