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有化学反应湍流流动研究进展和课题

力学进展 1982年第4期 419-427页

作者： p.A.libby ,F.A.Williams ,董务民

6.1 引言(略) 6.2 湍流流动中的辐射传输有化学反应湍流流动中的辐射传输,可以同所涉及的单相物质的相互作用有关,其代表为高温气体中的二氧化碳和水;也可以同所涉及的多相流动的辐射相互作用有关,其代表为燃气中的碳颗粒。但前者可以... 详细信息

6.1 引言(略) 6.2 湍流流动中的辐射传输有化学反应湍流流动中的辐射传输,可以同所涉及的单相物质的相互作用有关,其代表为高温气体中的二氧化碳和水;也可以同所涉及的多相流动的辐射相互作用有关,其代表为燃气中的碳颗粒。但前者可以包括化学发光源的可见光辐射,对其能量值的主要贡献常在光谱的红外部分,因而这种气体辐射有时称为不发光辐射。后者常有较宽的谱(不是谱带型的),

关键词：湍流流动辐射传输紊流流动粘性流动守恒方程流动方程化学反应湍流扩散火焰预混燃烧课题

肽酰基精氨酸脱亚氨酶4和中性粒细胞胞外诱捕网形成在实验性动脉粥样硬化和动脉损伤中的作用:浅表糜烂的影响

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中华高血压杂志 2018年第5期26卷 463-463页

作者：刘青叶鹏 Franck G Mawson TL Folco EJ Molinaro R Ruvkun V Engelbertsen D Liu X Tesmenitsky Y Shvartz F Sukhova GK Michel JB Nicoletti A Lichtman AH Wagner DD Croce KJ libby p

中性粒细胞可能促进人类动脉粥样硬化的血栓并发症的发生。特别是中性粒细胞胞外诱捕网（neutrophil extracellular traps,NETs）可加剧局部炎症,并放大和加剧动脉内膜损伤和血栓形成。肽酰基精氨酸脱亚氨酶4（peptidyl arginine deimin... 详细信息

中性粒细胞可能促进人类动脉粥样硬化的血栓并发症的发生。特别是中性粒细胞胞外诱捕网（neutrophil extracellular traps,NETs）可加剧局部炎症,并放大和加剧动脉内膜损伤和血栓形成。肽酰基精氨酸脱亚氨酶4（peptidyl arginine deiminase 4,pAD4）参与NET的形成,但对该酶在动脉粥样硬化血栓形成中的作用的认识仍然不足。

关键词：实验性动脉粥样硬化中性粒细胞精氨酸细胞胞动脉粥样硬化血栓形成动脉损伤诱捕酰基

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高强度他汀类治疗对冠状动脉粥样硬化的消退作用

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中国动脉硬化杂志 2006年第6期14卷 498-498页

作者： Nissen SE Nicholls SJ Sipahi I libby p Raichlen JS Ballantyne CM 夏妍

背景与目的血管内超声试验证明应用他汀类药物能减慢或延迟动脉粥样硬化的发展,但是, 使用粥样瘤体积百分比法还没有找到确凿证据证明疾病进展减慢或消退,后者比血管内超声试验更准确.此项研究旨在评定高强度他汀类药物治疗是否如血管... 详细信息

背景与目的血管内超声试验证明应用他汀类药物能减慢或延迟动脉粥样硬化的发展,但是, 使用粥样瘤体积百分比法还没有找到确凿证据证明疾病进展减慢或消退,后者比血管内超声试验更准确.此项研究旨在评定高强度他汀类药物治疗是否如血管内超声成像显示的那样能延缓冠状动脉粥样硬化的发展.

关键词：冠状动脉粥样硬化他汀类药物消退作用药物治疗血管内超声成像强度超声试验疾病进展百分比法澳大利亚

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Braunwald’s heart disease : a textbook of cardiovascular medicine 7th ed.

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Braunwald’s heart disease : a textbook of cardiovascular me...

2005年

作者： libby peter Braunwald Eugene Zipes Douglas p.

来源：畅想之星图书光盘版（南... 评论

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低剂量甲氨蝶呤不能预防动脉粥样硬化事件

中华高血压杂志(中英文) 2019年第8期 799-799页

作者： Ridker pM Everett BM pradhan A MacFadyen JG Solomon DH Zaharris E Mam V Hasan A Rosenberg Y Iturriaga E Gupta M Tsigoulis M Verma S Clearfield M libby p Goldhaber SZ Seagle R Ofori C Saklayen M Butman S Singh N Le May M Bertrand O Johnston J paynter Np Glynn RJ CIRT Investigators 林凯玲余惠珍

炎症反应与动脉粥样硬化的血栓形成密切相关。卡纳单抗(canakinumab)为一种能与白细胞介素(interleukin,IL)1β结合抑制炎症反应的单克隆抗体。以往一项随机对照研究发现,给予卡纳单抗干预后,患者的心血管事件发生率明显低于安慰剂组。... 详细信息

炎症反应与动脉粥样硬化的血栓形成密切相关。卡纳单抗(canakinumab)为一种能与白细胞介素(interleukin,IL)1β结合抑制炎症反应的单克隆抗体。以往一项随机对照研究发现,给予卡纳单抗干预后,患者的心血管事件发生率明显低于安慰剂组。该文采用抑制炎症反应替代药物——低剂量甲氨蝶呤,企图验证应用甲氨蝶呤能否同样获益。研究人员对既往有心肌梗死或多支冠状动脉病变史、伴有2型糖尿病或代谢综合征的患者4786例进行一项随机、双盲试验

关键词：动脉粥样硬化甲氨蝶呤低剂量

Light Reflection by Cuticles of Chrysina Jewel Scarabs: Optical Measurements, Morphology Characterization, and Theoretical Modeling

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Optics and photonics Journal 2016年第7期6卷 146-163页

作者： W. E. Vargas M. Hernández-Jiménez E. libby D. E. Azofeifa C. Barboza Á. Solis Centro de Investigación en Ciencia e Ingeniería de Materiales & Escuela de Física Universidad de Costa Rica San José Costa Rica Academia Nacional de Ciencias de Costa Rica San José Costa Rica Centro de Investigación en Ciencia e Ingeniería de Materiales and Escuela de Química Universidad de Costa Rica San José Costa Rica Centro de Investigación en Estructuras Microscópicas Universidad de Costa Rica San José Costa Rica Museo Nacional de Costa Rica San José Costa Rica

Cuticles of some Chrysina scarabs are characterized by flat, graded, and twisted structures of nanosized chitin fibrils. As inferred from SEM images, each species has its own spatial period or pitch p which is dependent on the depth z through the cuticle. From Berreman’s formalism, taking into account the corresponding p(z) dependence, we evaluate reflection spectra of C. aurigans and C. chrysargyrea scarabs. The spectra display the main spectral features observed in the measured ones when small sections of the cuticles are illuminated with non-polarized light, for wavelengths between 300 and 1100 nm. By considering these twisted structures as 1D photonic crystals, an approach is developed to show how the broad band characterizing the reflection spectra arises from a narrow intrinsic photonic band width, whose spectral position moves through visible and near infrared wavelengths. The role of the epicuticle that covers the twisted structures is analyzed in terms of a waxy layer acting as an anti-reflecting coating that also shows low levels of light scattering.

关键词： Structural Color Structural Chirality Natural Broad Band Reflector Circular polarization Jewel Scarab Chitin Refractive Index Uric Acid Refractive Index Left-Handed Twisted Structure Bouligand-Type Structure

Mutations in a p-type ATpase gene cause axonal degeneration

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Mutations in a P-type ATPase gene cause axonal degeneration

Amplitude analysis of the decays D^(0)→π^(+)π^(−)π^(+)π^(−)and D^(0)→π^(+)π^(−)π^(0)π^(0)

2012全国发育生物学大会

作者： Richard T.libby Wilhelmine N.de Vries Richard S.Smith Dana L.Wright Roderick T.Bronson Kevin L.Seburn Simon W.M.John The Howard Hughes Medical Institute The Jackson Laboratory600 Main StreetBar HarborMEUSA Department of Ophthalmology Tufts University of MedicineBostonMAUSA

Neuronal loss and axonal degeneration are important pathological features of many neurodegenerative diseases. The molecular mechanisms underlying the majority of axonal degeneration conditions remain *** better understand axonal degeneration,we studied a mouse mutant wabbler-Iethal(wl).Wabbler-lethal(wl) mutant mice develop progressive ataxia with pronounced neurodegeneration in the central and peripheral nervous *** studies have led to a debate of whether myelinopathy or axonopathy is the primary cause of neurodegeneration observed in wl mice. Here we provide clear evidence that wabbler lethal mutants develop an axonopathy,and that this axonopathy is modulated by Wld and Bax *** addition,we have identified the gene harboring the disease-causing mutations as *** studied three wl alleles and found that all result from mutations in the Atp8a2 *** analysis shows that ATp8A2 possesses phosphatidylserine translocase activity and is involved in localization of phosphatidylserine to the inner leaflet of the plasma ***8a2 is widely expressed in the brain,the spinal cord,and *** assessed two of the mutant alleles of Atp8a2 and found they are both nonfunctional for the phosphatidylserine translocase activity. Conclusions:Our data demonstrate for the first time that mutation of a mammalian phosphatidylserine translocase causes axon degeneration and neurodegenerative disease.

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Chinese physics C 2024年第8期48卷 6-33页

作者： M.Ablikim M.N.Achasov p.Adlarson O.Afedulidis X.C.Ai R.Aliberti A.Amoroso Q.An Y.Bai O.Bakina I.Balossino Y.Ban H.-R.Bao V.Batozskaya K.Begzsuren N.Berger M.Berlowski M.Bertani D.Bettoni F.Bianchi E.Bianco A.Bortone I.Boyko R.A.Briere A.Brueggemann H.Cai X.Cai A.Calcaterra G.F.Cao N.Cao S.A.Cetin J.F.Chang W.L.Chang G.R.Che G.Chelkov C.Chen C.H.Chen Chao Chen G.Chen H.S.Chen M.L.Chen S.J.Chen S.L.Chen S.M.Chen T.Chen X.R.Chen X.T.Chen Y.B.Chen Y.Q.Chen Z.J.Chen Z.Y.Chen S.K.Choi X.Chu G.Cibinetto F.Cossio J.J.Cui H.L.Dai J.p.Dai A.Dbeyssi R.E.de Boer D.Dedovich C.Q.Deng Z.Y.Deng A.Denig I.Denysenko M.Destefanis F.De Mori B.Fang S.S.Fang W.X.Fang Y.Fang Y.Q.Fang R.Farinelli L.Fava F.Feldbauer G.Felici C.Q.Feng J.H.Feng Y.T.Feng K.Fischer M.Fritsch C.D.Fu J.L.Fu Y.W.Fu H.Gao Y.N.Gao Yang Gao S.Garbolino I.Garzia p.T.Ge Z.W.Ge C.Geng E.M.Gersabeck B.Ding X.X.Ding Y.Ding Y.Ding J.Dong L.Y.Dong M.Y.Dong X.Dong M.C.Du S.X.Du Z.H.Duan p.Egorov Y.H.Fan J.Fang JA.Gilman K.Goetzen L.Gong W.X.Gong W.Gradl S.Gramigna M.Greco M.H.Gu Y.T.Gu C.Y.Guan Z.L.Guan A.Q.Guo L.B.Guo M.J.Guo R.p.Guo Y.p.Guo A.Guskov J.Gutierrez K.L.Han T.T.Han X.Q.Hao F.A.Harris K.K.He K.L.He F.H.Heinsius C.H.Heinz Y.K.Heng C.Herold T.Holtmann p.C.Hong G.Y.Hou X.T.Hou Y.R.Hou Z.L.Hou B.Y.Hu H.M.Hu J.F.Hu T.Hu Y.Hu G.S.Huang K.X.Huang L.Q.Huang X.T.Huang Y.p.Huang T.Hussain F.H\"olzken N.H\"usken N.in der Wiesche M.Irshad J.Jackson S.Janchiv J.H.Jeong Q.Ji Q.p.Ji W.Ji X.B.Ji X.L.Ji Y.Y.Ji X.Q.Jia Z.K.Jia D.Jiang H.B.Jiang p.C.Jiang S.S.Jiang T.J.Jiang X.S.Jiang Y.Jiang J.B.Jiao J.K.Jiao Z.Jiao S.Jin Y.Jin M.Q.Jing X.M.Jing T.Johansson S.Kabana N.Kalantar-Nayestanaki X.L.Kang X.S.Kang M.Kavatsyuk B.C.Ke V.Khachatryan A.Khoukaz R.Kiuchi O.B.Kolcu B.Kopf M.Kuessner X.Kui A.Kupsc W.K\"uhn J.J.Lane p.Larin L.Lavezzi T.T.Lei Z.H.Lei H.Leithoff M.Lellmann T.Lenz C.Li C.Li C.H.Li Cheng Li D.M.Li F.Li G.Li H.Li H.B.Li H.J.Li H.N.Li Hui Li J.R.Li J.S.Li K.Li L.J.Li L.K.Li Lei Li M.H.Li p.R.Li Q.M.Li Q.X.Li R.Li S.X.Li T.Li W.D.Li W.G.Li X.Li X.H.Li X.L.Li X.Y.Li Y Institute of High Energy physics Chinese Academy of SciencesBeijing 100049China Beihang University Beijing 100191China Bochum Ruhr-University D-44780 BochumGermany Budker Institute of Nuclear physics SB RAS(BINp) Novosibirsk 630090Russia Carnegie Mellon University PittsburghPennsylvania 15213USA Central China Normal University Wuhan 430079China Central South University Changsha 410083China China Center of Advanced Science and Technology Beijing 100190China China University of Geosciences Wuhan 430074China Chung-Ang University Seoul06974Republic of Korea COMSATS University Islamabad Lahore CampusDefence RoadOff Raiwind Road54000 LahorePakistan Fudan University Shanghai 200433China GSI Helmholtzcentre for Heavy Ion Research GmbH D-64291 DarmstadtGermany Guangxi Normal University Guilin 541004China Guangxi University Nanning 530004China Hangzhou Normal University Hangzhou 310036China Hebei University Baoding 071002China Helmholtz Institute Mainz Staudinger Weg 18D-55099 MainzGermany Henan Normal University Xinxiang 453007China Henan University Kaifeng 475004China Henan University of Science and Technology Luoyang 471003China Henan University of Technology Zhengzhou 450001China Huangshan College Huangshan 245000China Hunan Normal University Changsha 410081China Hunan University Changsha 410082China Indian Institute of Technology Madras Chennai 600036India Indiana University BloomingtonIndiana 47405USA INFN Laboratori Nazionali di Frascati INFN Laboratori Nazionali di FrascatiI-00044FrascatiItaly INFN Laboratori Nazionali di Frascati INFN Sezione di PerugiaI-06100PerugiaItaly INFN Laboratori Nazionali di Frascati University of PerugiaI-06100PerugiaItaly INFN Sezione di Ferrara INFN Sezione di FerraraI-44122FerraraItaly INFN Sezione di Ferrara University of FerraraI-44122FerraraItaly Inner Mongolia University Hohhot 010021China Institute of Modern physics Lanzhou 730000China Institute of physics and Technology Peace Avenue 54BUlaanbaatar 13330Mongolia Instituto de A

Using e^(+)e^(−)annihilation data corresponding to an integrated luminosity of 2.93 fb^(−1)taken at the center-of-mass energy√s=3.773 GeV with the BESIII detector,a joint amplitude analysis is performed on the decays D^(0)→π^(+)π^(−)π^(+)π^(−)and D^(0)→π^(+)π^(−)π^(0)π^(0)(non-η).The fit fractions of individual components are obtained,and large interferences among the dominant components of the decays D^(0)→a_(1)(1260)π,D^(0)→π(1300)π,D^(0)→ρ(770)ρ(770),and D^(0)→2(ππ)_(S)are observed in both *** the obtained amplitude model,the Cp-even fractions of D^(0)→π^(+)π^(−)π^(+)π^(−)and D^(0)→π^(+)π^(−)π^(0)π^(0)(non-η)are determined to be(75.2±1.1_(stat).±1.5_(syst.))%and(68.9±1.5_(stat).±2.4_(syst.))%,*** branching fractions of D^(0)→π^(+)π^(−)π^(+)π^(−)and D^(0)→π^(+)π^(−)π^(0)π^(0)(non-η)are measured to be(0.688±0.010_(stat.)±0.010_(syst.))%and(0.951±0.025_(stat.)±0.021_(syst.))%,*** amplitude analysis provides an important model for the binning strategy in measuring the strong phase parameters of D^(0)→4πwhen used to determine the CKM angleγ(ϕ_(3))via the B^(−)→DK^(−)decay.

关键词： BESIII D^(0)meson decays amplitude analysis Cp-even fraction

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人粥样硬化斑块的血管内MRI特征

世界核心医学期刊文摘(心脏病学分册) 2006年第3期 44-44页

作者： Larose E. Yeghiazarians Y. libby p. p. Ganz 马超 Brigham and Women' s Hospital 75 Francis St Boston MA 02115 United States Dr.

Background-Development and validation of novel imaging modalities to assess the composition of human atherosclerotic plaques will improve the understanding of atheroma evolution and could facilitate evaluation of therapeutic strategies for plaque modification. Surface MRI can characterize tissue content of carotid but not deeper arteries. This study evaluated the usefulness of intravascular MRI(IVMRI) to discern the composition of human iliac arteries in vivo. Methods and Results -Initial studies validated IVMRI against histopathology of human atherosclerotic arteries ex vivo. A 0.030-inch-diameter IVMRI detector coil was advanced into isolated human aortoiliac arteries and coupled to a 1.5-T scanner. Information from combined T1-, moderate T2-, and proton-density-weighted images differentiated lipid, fibrous, and calcified components with favorable sensitivity and specificity and allowed accurate quantification of plaque size. The validated approach was then applied to image iliac arteries of 25 human subjects in vivo, and results were compared with those of intravascular ultrasound(IVUS). IVMRI readily visualized inner and outer plaque boundaries in all arteries, even those with extensive calcification that precluded IVUS interpretation. It also revealed the expected heterogeneity of atherosclerotic plaque content that was noted during ex vivo validation. Again, IVUS did not disclose this heterogeneity. The level of interobserver and intraobserver agreement in the interpretation of plaque composition was high for IVMRI but poor for IVUS. Conclusions -IVMRI can reliably identify plaque composition and size in arteries deep within the body. Identification of plaque components by IVMRI in vivo has important implications for the understanding and modification of human atherosclerosis.

关键词：血管内斑块病理过程 MRI 髂动脉