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全关节区关节软骨体内扩散张量成像的可行性研究

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国际医学放射学杂志 2014年第5期 498-499页

作者： J.G.Raya E.Dettmann m.notohamiprodjo S.Krasnokutsky S.Abramson C.Glaser 谢媛 Department of Radiology New York University Langone Medical Center660 First Avenue4th Floor10016New YorkNYUSA

摘要目的研究全关节区关节软骨的扩散张量成像(DTI)对区分正常和骨关节炎 (OA)受试者的价值。方法对10 名健康受试者和 5 例骨关节炎受试者(Kellgren-Lawrence

关键词：扩散张量 DTI 骨关节炎受试者关节区关节软骨

前列腺扩散加权mR成像:放大并行传输加速2D选择性激发EPI成像的优点

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国际医学放射学杂志 2015年第1期 100-100页

作者： K.m.Thierfelder m.K.Scherr m.notohamiprodjo J.Weiβ O.Dietrich U.G.mueller-Lisse Institute for Clinical Radiology Ludwig-Maximilians University Hospital Munich

目的评价2D选择性并行传输激励mRI用于前列腺扩散加权平面回波成像技术(p TX-EPI),并与传统单次激励EPI(c-EPI)进行对比。方法此前瞻性研究评估35例病

关键词：扩散加权平面回波成像 EPI mR 前列腺男生殖器并行传输数据传输

CT灌注成像技术评估同种异体肾移植术后早期肾功能障碍:初步研究结果

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国际医学放射学杂志 2013年第6期 596-596页

作者： A.Helck m.Wessely m.notohamiprodjo U.Schnermarck E.Klotz m.Fischereder 朱珊张亚楠 Department of Clinical Radiology University Hospitals Grosshadern Ludwig-Maximilians-University of Munich Marchioninistr.1581377 Munich Germany

摘要目的定量评估CT灌注成像鉴别同种异体肾移植术后急性肾小管坏死和急性排斥反应的能力。方法本研究纳入了22例同种异体肾移植术后出现急性肾功能障碍的病

关键词： CT灌注成像肾移植肾血流急性排斥反应急性肾小管坏死

膝关节高度屈曲后导致的关节软骨体层影像的变形模式:基本体素分析法的3T活体3D mRI研究

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国际医学放射学杂志 2015年第4期 405-405页

作者： A.Horng J.G.Raya m.Stockinger m.notohamiprodjo m.Pietschmann U.H.Hueckstaedt 高利珍 Institute for Clinical Radiology Ludwig-Maximilians University Hospital Munich

摘要目的应用基本体素层厚技术来确定统计学上有意义的软骨变形区域,并在膝关节高度屈曲姿势下,应用体素层厚图分析活体内关节软骨变形模式。方法 10例正常志

关键词：关节软骨变形模式膝关节 mRI 活体

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3.0 T mR淋巴管成像的可行性研究

国际医学放射学杂志 2010年第1期 94-94页

作者： m.notohamiprodjo R.G.Baumeister T.F.Jakobs K.U.Bauner H.F.Boehm A.Horng 朱晖 Department of Clinical Radiology University-Hospitals MunichCampus GrosshadernMarchioninistrasse 1581377 MunichGermany

本项研究的目的是确定和评价3.0T mR对比剂增强淋巴管成像(mRL)在检测和显示外周淋巴系统异常方面的意义。16例病人皮内注射二乙烯三胺五乙酸钆剂后行高分辨力3.0T mR检查,采用T1W-3D-GRE-(FLASH)序列(TR3.76ms/TE1.45ms/FA30°/体素0.... 详细信息

本项研究的目的是确定和评价3.0T mR对比剂增强淋巴管成像(mRL)在检测和显示外周淋巴系统异常方面的意义。16例病人皮内注射二乙烯三胺五乙酸钆剂后行高分辨力3.0T mR检查,采用T1W-3D-GRE-(FLASH)序列(TR3.76ms/TE1.45ms/FA30°/体素0.8mm×0.8mm×0.8mm)。2名放射科医

关键词：淋巴管 mRL 对比剂淋巴系统 T mR 淋巴回流

Neurogenesis dynamics in the olfactory bulb:deciphering circuitry organization, function, and adaptive plasticity

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Neural Regeneration Research 2025年第6期20卷 1565-1581页

作者： moawiah m.Naffaa Department of Psychology and Neuroscience Duke UniversityDurhamNCUSA Department of Cell Biology Duke University School of MedicineDurhamNCUSA

Adult neurogenesis persists after birth in the subventricular zone, with new neurons migrating to the granule cell layer and glomerular layers of the olfactory bulb, where they integrate into existing circuitry as inhibitory interneurons. The generation of these new neurons in the olfactory bulb supports both structural and functional plasticity, aiding in circuit remodeling triggered by memory and learning processes. However, the presence of these neurons, coupled with the cellular diversity within the olfactory bulb, presents an ongoing challenge in understanding its network organization and function. moreover,the continuous integration of new neurons in the olfactory bulb plays a pivotal role in regulating olfactory information processing. This adaptive process responds to changes in epithelial composition and contributes to the formation of olfactory memories by modulating cellular connectivity within the olfactory bulb and interacting intricately with higher-order brain regions. The role of adult neurogenesis in olfactory bulb functions remains a topic of debate. Nevertheless, the functionality of the olfactory bulb is intricately linked to the organization of granule cells around mitral and tufted cells. This organizational pattern significantly impacts output, network behavior, and synaptic plasticity, which are crucial for olfactory perception and memory. Additionally, this organization is further shaped by axon terminals originating from cortical and subcortical regions. Despite the crucial role of olfactory bulb in brain functions and behaviors related to olfaction, these complex and highly interconnected processes have not been comprehensively studied as a whole. Therefore, this manuscript aims to discuss our current understanding and explore how neural plasticity and olfactory neurogenesis contribute to enhancing the adaptability of the olfactory system. These mechanisms are thought to support olfactory learning and memory, potentially through increased

关键词： network adaptability neurogenesis neuronal communication olfactory bulb olfactory learning olfactory memory synaptic plasticity

mitochondria–cGAS–STING axis is a potential therapeutic target for senescence-dependent inflammaging-associated neurodegeneration

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Neural Regeneration Research 2025年第3期20卷 805-807页

作者： Josém.Izquierdo Centro de Biología molecular Severo Ochoa(CBm) Consejo Superior de Investigaciones Científicas-Universidad Autónoma de Madrid(CSIC-UAM)MadridSpain

The word “senescence” comes from the Latin senescens, meaning “to begin to age”, and is characterized by a long-lasting but reversible block in proliferation, resulting from stress-induced cell cycle arrest of pre... 详细信息

关键词： Latin potential lasting

Brain-penetrating neurotrophic factor mimetics: HER-096 as a disease-modifying therapy for Parkinson’s disease

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Neural Regeneration Research 2025年第4期20卷 1094-1095页

作者： Natalia Kulesskaya Kira m.Holmström Henri J.Huttunen Herantis Pharma Plc EspooFinland

Neurotrophic factors as a therapeutic approach in neurodegenerative diseases:A major unmet need in the field of central nervous system diseases is disease-modifying *** for decades there have been various symptomatic treatments available to alleviate the symptoms of the disease,disease-modification,*** that stop,significantly delay,or reverse the progression of the disease,has been turned out to a difficult goal to achieve.

关键词： diseases treatment therapeutic

Search for microglia-specific peptides:advances in the discovery toolbox

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Neural Regeneration Research 2025年第2期20卷 471-472页

作者： Ivan Sarabia Kyle m.Koss Department of Surgery University of ArizonaTucsonAZUSA

Uncontrolled and chronic inflammatory states in the central nervous system(CNS)are the hallmark of neurodegenerative pathology and every injury or stroke-related *** key mediators of these neuroinflammatory states are glial cells known as microglia,the resident immune cell at the core of the inflammatory event,and astroglia,which encapsulate inflammatory insults in proteoglycan-rich scar *** gliotic scar blocks significant portions of healthy axonal networking,preventing *** most neuroinflammation is exclusively based on the responses of said microglia,their phenotypes are suggested to follow those of macrophages;m1 and m2 are opposites of pro-and ***,microglial phenotypes have been identified to be on an inflammatory spectrum encompassing developmental,homeostatic,and reparative behaviors as opposed to their ability to affect devastating cell death cascades and scar tissue formation.

关键词： inflammation pathology death