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limitations of absolute current densities derived from the Semel & Skumanich method

Science China(Physics,Mechanics & astronomy) 2009年第11期52卷 1678-1685页

作者： li jing1 & fan yuhong2.1 institute for astronomy, university of hawaii,{2., honolulu, hi 9682., USA 2.high Altitude Observatory, Earth and Sun Systems Laboratory, National Center for Atmospheric Research, P.O. Box 3000, Boulder, CO 80307, USA institute for astronomy University of Hawaii 2680 Woodlawn Drive Honolulu HI 96822 USA high Altitude Observatory Earth and Sun Systems Laboratory National Center for Atmospheric Research P.O. Box 3000 Boulder CO 80307 USA Present address: UCLA institute of Geophysics and Planetary Physics 2713 Geology Building MG 951567 Los Angeles CA 90095-1567 USA

Semel and Skumanich proposed a method to obtain the absolute electric current density, |Jz|, without disambiguation of 180° in the transverse field directions. The advantage of the method is that the uncertainty in the determination of the ambiguity in the magnetic azimuth is removed. Here, we investigate the limits of the calculation when applied to a numerical MHD model. We have found that the combination of changes in the magnetic azimuth with vanis{2.ng horizontal field component leads to errors, where electric current densities are often strong. Where errors occur, the calculation gives |Jz| too small by factors typically 1.2.- 2.0.

关键词： numerical MHD model magnetic field electric current density

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ENO1 expression and Erk phosphorylation in PDAC and their effects on tumor cell apoptosis in a hypoxic microenvironment

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Cancer Biology & Medicine 2022年第11期19卷 1598-1616页

作者： Huizhi Sun jing Mo Runfen Cheng {2. li Yue li yuhong Guo Yanlei li Yanhui Zhang Xiaoyu Bai Yalei Wang Xueyi Dong Dan{2.g Zhang Jihui Hao Tianjin Medical university Cancer institute&Hospital National Clinical Research Center for CancerKey Laboratory of Cancer Prevention and TherapyTianjinTianjin's Clinical Research Center for CancerTianjin 300060China Department of Pathology Tianjin Medical UniversityTianjin300070China

Objective: Hypoxia is an important feature of pancreatic ductal adenocarcinoma(PDAC). Previously, we found that hypoxia promotes ENO1 expression and PDAC invasion. However, the underlying molecular mechanism was remains ***: The relationship between ENO1 expression and clinicopathological characteristics was analyzed in 84 patients with PADC. The effects of CoCl2.induced hypoxia and ENO1 downregulation on the apoptosis, invasion, and proliferation of PDAC cells were evaluated in vitro and in vivo. Hypoxia-and ENO1-induced gene expression was analyzed by transcriptomic ***: The prognosis of PDAC with high ENO1 expression was poor(P high ENO1 expression was closely associated with histological differentiation and tumor invasion in 84 PDAC cases(P 2.-treated ENO1-sh group were significantly elevated(P 2.-induced PDAC cells initiated MAPK signaling. Under hypoxic conditions, PDAC cells upregulated ENO1 expression, thereby accelerating ERK phosphorylation and inhibiting apoptosis(P hindlimb ischemia ***: Hypoxia-induced ENO1 expression promotes ERK phosphorylation and inhibits apoptosis, thus leading to PDAC survival and invasion. These results suggest that ENO1 is a potential therapeutic target for PDAC.

关键词： Pancreatic cancer hypoxia ENOl apoptosis Erk phosphorylation

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