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Effects of Red Palm Oil on Serum Lipids and Plasma Carotenoids Level in chinese Male Adults

Biomedical and Environmental Sciences 2003年第4期16卷 348-354页

作者： jian zhang , chun-rong wang, an-na xue, and ke-you geinstitute of nutrition and food safety, chinese center for disease control and prevention, beijing 100050, china Institute of nutrition and food safety Chinese Center for Disease Control and Prevention Beijing 100050 Chin

Objective Effects of red palm oil on major plasma carotenoids, tocopherol, retinol and serum lipids were evaluated when used in chinese diet. Methods Red palm oil group (RPO) composed of 20 male subjects(aged 18-32) and soybean oil group (SBO) composed of 22 male subjects (aged 18-32). Dietary fat provided about 28% of total calories, and the test oil accounted for about 60% of total dietary fat. In the 3 weeks of pretest period, diets were prepared with soybean oil, and then in the next 6 weeks subjects in each group consumed the diet prepared by test oil. Results Plasma α-carotene, β-carotene and lycopene concentration of RPO group significantly increased at the time of interim (21 days) and of the end (42 days) (Pchinese plasma retinol level was relatively low when compared with that of Westerners, red palm oil diet showed no significant effect on adult chinese plasma retinol level. Serum concentration of total cholesterol, triglyceride, high density lipoprotein cholesterol, apolipoprotein AI and apolipoprotein B of all subjects showed no significant changes in RPO group during the study. Conclusions The data in our study suggest that red palm oil is a good source of carotenoids and vitamin E when used in chinese diet preparation, and it can significantly increase plasma concentration of a-carotene, α-carotene, lycopene and β-tocopherol.

关键词： Red palm oil Retinal α-carotene β-carotene α-tocopherol Serum cholesterol Serum triglyeride Apolipoprotein AI Apolipoprotein B

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Biochemical and Morphological Changes in the Lenses of Selenium and/or Vitamin E Deficient Rats

引用

Biomedical and Environmental Sciences 1994年第2期7卷 109-115页

作者： CAI QIU-YIN CHEN XIAO-SHU ZHU LIAN-ZHEN xue AN-na LI WEN-XIAN wang SHU-QIN PIAO jian-HUA LI JIE SUN CUN-PU WU ke AND CONG jian-BO(Institute of nutrition and food Hygiene, chinese Academy of Preventive Medicine, 29 nan Wet Road, beijing 100050, Chin Institute of nutrition and food Hygiene Chinese Academy of Preventive Medicine Beijing.

The activities of glutathione peroxidase (GSH-Px), glutathione reductase (GSSG-R),superoalde dismutase (SOD) and the contents of malondialdehyde (MDA) and free radicals were measured, and the morphological changes were observed in the lens of control rats, selenium-deficient (SeD) and/or vitamin E deficient (VED) rats. The activities of GSH-Px in the lens of SeD rats decreased significantly. The GSH-Px activities of lens were positively related to erythrocytes selenium level. There was a free radical at g = 2.0015 in the rat lens of all groupe, but the content of free radicals in the lens of SeD group was significantly higher than that of the control group. The free radical content of lens was negatively related to erythrocytes selenium level, as well as the GSH-Px activities in the lens. In vitro, ultraviolet radiation caused the generation of another kind of free radical (g = 2.0097) in the lens of all groups, but the amount of the free radical in the lens of the SeD group was also significantly higher than that of the control group. The activities of SoD and GSSG-R in VED rat lens were significantly decreased. The amount of MDA in the lens of SeD and/or VED rats were significantly increased. The results showed that the decrease of antioaldative capability in the lenses of SeD and/or VED rats accelerated the lipid peroxidation and generation of free radicals. Although only early morphological changes in SeD and/or VED rat lens were observed, it is considered that selenium and vitamin E deficiency may be involved in the occurrence of cataract

关键词： Res GSH Biochemical and Morphological Changes in the Lenses of Selenium and/or Vitamin E Deficient Rats

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