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An in-situ self-etching enabled high-power electrode for aqueous zinc-ion batteries

Journal of Energy Chemistry 2024年第1期88卷 399-408,I0009页

作者： Shuang Hou Dingtao Ma Yanyi Wang Kefeng ouyang Sicheng Shen hongwei Mi Lingzhi Zhao Peixin Zhang College of Chemistry and Environmental Engineering Shenzhen UniversityShenzhen 518060GuangdongChina Guangdong Provincial Engineering Technology Research Center for Low Carbon and Advanced Energy Materials School of Semiconductor Science and TechnologySouth China Normal UniversityFoshan 528225GuangdongChina SCNU Qingyuan Institute of Science and Technology Innovation Co. Ltd.Qingyuan 511517GuangdongChina

Sluggish storage kinetics is considered as the main bottleneck of cathode materials for fast-charging aqueous zinc-ion batteries(AZIBs).In this report,we propose a novel in-situ self-etching strategy to unlock the Palm tree-like vanadium oxide/carbon nanofiber membrane(P-VO/C)as a robust freestanding electrode.Comprehensive investigations including the finite element simulation,in-situ X-ray diffraction,and in-situ electrochemical impedance spectroscopy disclosed it an electrochemically induced phase transformation mechanism from VO to layered Zn_(x)V_(2)O_5·nH_(2)O,as well as superior storage kinetics with ultrahigh pseudocapacitive contribution.As demonstrated,such electrode can remain a specific capacity of 285 mA h g^(-1)after 100 cycles at 1 A g^(-1),144.4 mA h g^(-1)after 1500 cycles at 30 A g^(-1),and even 97 mA h g^(-1)after 3000 cycles at 60 A g^(-1),respectively.Unexpectedly,an impressive power density of 78.9 kW kg^(-1)at the super-high current density of 100 A g^(-1)also can be achieved.Such design concept of in-situ self-etching free-standing electrode can provide a brand-new insight into extending the pseudocapacitive storage limit,so as to promote the development of high-power energy storage devices including but not limited to AZIBs.

关键词： In-situ self-etching Free-standing electrode Pseudocapacitive storage High-power Zinc-ion batteries

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Seamless and early gap healing of osteochondral defects by autologous mosaicplasty combined with bioactive supramolecular nanofiber-enabled gelatin methacryloyl(BSN-GelMA)hydrogel

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Bioactive Materials 2023年第1期19卷 88-102页

作者： hongwei Wu Yuna Shang Wei Sun Xinyi ouyang Wenyan Zhou Jieji Lu Shuhui Yang Wei Wei Xudong Yao g Xiaozhao Wang Xianzhu Zhang Yishan Chen Qiulin He Zhimou Yang hongwei ouyang Dr.Li Dak Sum&Yip Yio Chin Center for Stem Cells and Regenerative Medicine and Department of Orthopedic Surgery of the Second Affiliated HospitalZhejiang University School of MedicineHangzhouChina Department of Sports Medicine Zhejiang University School of MedicineHangzhouChina Zhejiang University-University of Edinburgh Institute Zhejiang University School of Medicineand Key Laboratory of Tissue Engineering and Regenerative Medicine of Zhejiang ProvinceZhejiang University School of MedicineHangzhouChina China Orthopedic Regenerative Medicine Group(CORMed) HangzhouChina College of Chemistry Tianjin Normal UniversityTianjin300387PR China Faculty of Science The University of MelbourneParkvilleVIC3010Australia International Institutes of Medicine The 4th Affiliated Hospital of Zhejiang University School of MedicineYiwuZhejiangChina Key Laboratory of Bioactive Materials Ministry of EducationState Key Laboratory of Medicinal Chemical BiologyCollege of Life SciencesCollaborative Innovation Center of Chemical Science and Engineeringand National Institute of Functional MaterialsNankai UniversityTianjin300071China

Autologous mosaicplasty is a common approach used to treat osteochondral defects in clinical practice.Gap integration between host and transplanted plugs requires bone tissue reservation and hyaline cartilage regeneration without uneven surface,graft necrosis and sclerosis.However,poor gap integration is a serious concern,which eventually leads to deterioration of joint function.To deal with such complications,this study has developed a strategy to effectively enhance integration of the gap region following mosaicplasty by applying injectable bioactive supramolecular nanofiber-enabled gelatin methacryloyl(GelMA)hydrogel(BSN-GelMA).A rabbit osteochondral defect model demonstrated that BSN-GelMA achieved seamless osteochondral healing in the gap region between plugs of osteochondral defects following mosaicplasty,as early as six weeks.Moreover,the International Cartilage Repair Society score,histology score,glycosaminoglycan content,subchondral bone volume,and collagen II expression were observed to be the highest in the gap region of BSN-GelMA treated group.This improved outcome was due to bio-interactive materials,which acted as tissue fillers to bridge the gap,prevent cartilage degeneration,and promote graft survival and migration of bone marrow mesenchymal stem cells by releasing bioactive supramolecular nanofibers from the GelMA hydrogel.This study provides a powerful and applicable approach to improve gap integration after autologous mosaicplasty.It is also a promising off-the-shelf bioactive material for cell-free in situ tissue regeneration.

关键词： Mosaicplasty Osteochondral integration Bioactive supramolecular nanofiber GelMA hydrogel Tissue engineering

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High-resolution aging niche of human adipose tissues

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Signal Transduction and Targeted Therapy 2023年第4期8卷 1368-1371页

作者： Wenyan Zhou Junxin Lin Yan Xie Xueqing Hu Xudong Yao Yuemin Ou hongwei Wu Yiyang Yan Xiumao Li Jianan Lou Aaron Trent Irving James Q.Wang hongwei ouyang Dr.Li Dak Sum&Yip Yio Chin Center for Stem Cells and Regenerative Medicine and Department of Orthopedic Surgery of the Second Affiliated HospitalZhejiang University School of MedicineHangzhou 310058China Department of Sports Medicine Zhejiang University School of MedicineHangzhou 310058China Zhejiang University-University of Edinburgh Institute Zhejiang University School of Medicineand Key Laboratory of Tissue Engineering and Regenerative Medicine of Zhejiang ProvinceZhejiang University School of MedicineHangzhou 310058China Tissue Organ Bank&Tissue Engineering Centre General Hospital of Ningxia Medical UniversityNingxia 750003China Department of Plastic Surgery The Second Affiliated HospitalZhejiang University School of MedicineHangzhou 310052China The Fourth Affiliated Hospital Zhejiang University School of MedicineYiwu 322000China Department of Orthopedics The Second Affiliated HospitalZhejiang University School of MedicineHangzhou 310009China China Orthopedic Regenerative Medicine Group(CORMed) Hangzhou 310058China

Dear Editor,Age-dependent adipose tissue malfunction raises the risk of diseases like diabetes,cardiovascular disease,and even cancer by contributing to metabolic decline,heterotopic fat storage,and chronic systemic inflammation.1 Understanding adipose tissue aging requires in-depth knowledge of the cellular and molecular properties of various adipose tissue cell types.Although the heterogeneity of the cell population during mouse aging has been studied,2 little is known about the cellular and molecular basis of human adipose tissues aging.

关键词： aging cardiovascular diseases

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A high-resolution route map reveals distinct stages of chondrocyte dedifferentiation for cartilage regeneration

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Bone Research 2022年第3期10卷 578-593页

作者： Yishan Chen Yeke Yu Ya Wen Juan Chen Junxin Lin Zixuan Sheng Wenyan Zhou Heng Sun Chengrui An Jiansong Chen Weiliang Wu Chong Teng Wei Wei hongwei ouyang Dr.Li Dak Sum&Yip Yio Chin Center for Stem Cells and Regenerative Medicine and Department of Orthopedic Surgery of the Second Affiliated HospitalZhejiang University School of MedicineHangzhou 310058China Department of Sports Medicine Zhejiang University School of MedicineHangzhou 310058China Zhejiang University-University of Edinburgh Institute Zhejiang University School of Medicineand Key Laboratory of Tissue Engineering and Regenerative Medicine of Zhejiang ProvinceZhejiang University School of MedicineHangzhou 310058China Department of Oral Surgery Ninth People’s HospitalShanghai Jiao Tong University School of MedicineShanghai 200011China Department of General Intensive Care Unit The Second Affiliated Hospital of Zhejiang University School of MedicineHangzhou 310009China Department of Orthopedic Surgery The Children’s HospitalZhejiang University School of MedicineNational Clinical Research Center for Child HealthHangzhou 310052China Department of Orthopedic Surgery The Fourth Affiliated HospitalZhejiang University School of MedicineYiwu 322000China China Orthopedic Regenerative Medicine Group(CORMed) Hangzhou 310058China

Articular cartilage damage is a universal health problem.Despite recent progress,chondrocyte dedifferentiation has severely compromised the clinical outcomes of cell-based cartilage regeneration.Loss-of-function changes are frequently observed in chondrocyte expansion and other pathological conditions,but the characteristics and intermediate molecular mechanisms remain unclear.In this study,we demonstrate a time-lapse atlas of chondrocyte dedifferentiation to provide molecular details and informative biomarkers associated with clinical chondrocyte evaluation.We performed various assays,such as single-cell RNA sequencing(scRNA-seq),live-cell metabolic assays,and assays for transposase-accessible chromatin with high-throughput sequencing(ATAC-seq),to develop a biphasic dedifferentiation model consisting of early and late dedifferentiation stages.Early-stage chondrocytes exhibited a glycolytic phenotype with increased expression of genes involved in metabolism and antioxidation,whereas late-stage chondrocytes exhibited ultrastructural changes involving mitochondrial damage and stress-associated chromatin remodeling.Using the chemical inhibitor BTB06584,we revealed that early and late dedifferentiated chondrocytes possessed distinct recovery potentials from functional phenotype loss.Notably,this two-stage transition was also validated in human chondrocytes.An image-based approach was established for clinical use to efficiently predict chondrocyte plasticity using stage-specific biomarkers.Overall,this study lays a foundation to improve the quality of chondrocytes in clinical use and provides deep insights into chondrocyte dedifferentiation.

关键词： chondrocyte cartilage metabolism

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Current research on pharmacologic and regenerative therapies for osteoarthritis

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Bone Research 2015年第4期3卷 185-198页

作者： Wei Zhang hongwei ouyang Crispin R Dass Jiake Xu Center for Stem Cell and Tissue Engineering School of MedicineZhejiang University School of Pathology and Laboratory Medicine The University of Western Australia School of Pharmacy Building 306Curtin UniversityBentley

Osteoarthritis（OA）is a degenerative joint disorder commonly encountered in clinical practice,and is the leading cause of disability in elderly people.Due to the poor self-healing capacity of articular cartilage and lack of specific diagnostic biomarkers,OA is a challenging disease with limited treatment options.Traditional pharmacologic therapies such as acetaminophen,non-steroidal anti-inflammatory drugs,and opioids are effective in relieving pain but are incapable of reversing cartilage damage and are frequently associated with adverse events.Current research focuses on the development of new OA drugs（such as sprifermin/recombinant human fibroblast growth factor-18,tanezumab/monoclonal antibody againstβ-nerve growth factor）,which aims for more effectiveness and less incidence of adverse effects than the traditional ones.Furthermore,regenerative therapies（such as autologous chondrocyte implantation（ACI）,new generation of matrix-induced ACI,cell-free scaffolds,induced pluripotent stem cells（iPS cells or iPSCs）,and endogenous cell homing）are also emerging as promising alternatives as they have potential to enhance cartilage repair,and ultimately restore healthy tissue.However,despite currently available therapies and research advances,there remain unmet medical needs in the treatment of OA.This review highlights current research progress on pharmacologic and regenerative therapies for OA including key advances and potential limitations.

关键词： cartilage regenerative articular challenging pluripotent disability restore repair degenerative advances

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Free or fixed state of nHAP differentially regulates hBMSC morphology and osteogenesis through the valve role of ITGA7

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Bioactive Materials 2022年第12期7卷 539-551页

作者： Fangyuan Bao Junzhi Yi Yixiao Liu Yuliang Zhong Hui Zhang Zhonglin Wu Boon Chin Heng Ying Wang Ziyang Wang Lizi Xiao Hua Liu hongwei ouyang Jing Zhou Dr Li Dak Sum&Yip Yio Chin Center for Stem Cells and Regenerative Medicine and Department of Orthopedic Surgery of The Second Affiliated HospitalZhejiang University School of MedicineHangzhouPR China Department of Sports Medicine Zhejiang University School of MedicineHangzhouPR China Zhejiang University-University of Edinburgh Institute Zhejiang University School of Medicineand Key Laboratory of Tissue Engineering and Regenerative Medicine of Zhejiang ProvinceZhejiang University School of MedicineHangzhouPR China China Orthopedic Regenerative Medicine Group(CORMed) HangzhouPR China School of Stomatology Peking UniversityBeijingPR China Zhongshan School of Medicine Sun Yat-sen UniversityGuangzhouPR China

Nano-hydroxyapatite(nHAP)has been widely used in bone repair as an osteo-inductive and naturally-occurring material.However,the optimal applied form of nHAP and the underlying mechanisms involved remain unclear.Herein,to investigate into these,a range of corresponding models were designed,including three applied forms of nHAP(Free,Coating and 3D)that belong to two states(Free or fixed).The results indicate that when fixed nHAP was applied in the 3D form,optimal osteogenesis was induced in human bone marrow stem cells(hBMSCs)with increased bone volume via integrinα7(ITGA7)-mediated upregulation of the PI3K-AKT signaling pathway,while contrary results were observed with free nHAP.Ectopic osteogenesis experiments in mice subcutaneous transplantation model further confirmed the different tendencies of ITGA7 expression and osteogenesis of hBMSCs in free and fixed states of nHAP.Our results revealed that the two states of nHAP play a different regulatory role in cell morphology and osteogenesis through the valve role of ITGA7,providing cues for better application of nanoparticles and a potential new molecular target in bone tissue engineering.

关键词： Applied forms nHAP Osteogenesis ITGA7

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GelNB molecular coating as a biophysical barrier to isolate intestinal irritating metabolites and regulate intestinal microbial homeostasis in the treatment of inflammatory bowel disease

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Bioactive Materials 2023年第1期19卷 251-267页

作者： Qijiang Mao Haoqi Pan Yiyin Zhang Yi Zhang Qiuwen Zhu Yi Hong Zhengze Huang Yang Li Xu Feng Yifeng Fang WenChao Chen Pengfei Chen Bo Shen hongwei ouyang Yuelong Liang Department of General Surgery Sir Run Run Shaw HospitalZhejiang University School of MedicineHangzhou310016China Key Laboratory of Laparoscopic Technology of Zhejiang province Hangzhou310016China Zhejiang Province Medical Research Center of Minimally Invasive Diagnosis and Treatment of Abdominal Diseases Hangzhou310028China Dr.Li Dak Sum&Yip Yio Chin Center for Stem Cells and Regenerative Medicine Department of Orthopedic Surgery of the Second Affiliated HospitalZhejiang University School of MedicineHangzhouChina Zhejiang University-University of Edinburgh Institute Zhejiang University School of MedicineKey Laboratory of Tissue Engineering and Regenerative Medicine of Zhejiang ProvinceZhejiang University School of MedicineHangzhouChina The State Key Laboratory of Fluid Power and Mechatronic Systems School of Mechanical EngineeringZhejiang UniversityHangzhou310028China Department of Orthopaedic Surgery Sir Run Run Shaw HospitalMedical College of Zhejiang UniversityHangzhouZhejiang310016China

Inflammatory bowel disease(IBD)is a chronic,immune-mediated inflammatory disease characterized by the destruction of the structure and function of the intestinal epithelial barrier.Due to the poor remission effect and severe adverse events associated with current clinical medications,IBD remains an incurable disease.Here,we demonstrated a novel treatment strategy with high safety and effective inflammation remission via tissue-adhesive molecular coating.The molecular coating is composed of o-nitrobenzaldehyde(NB)-modified Gelatin(GelNB),which can strongly bond with-NH_(2)on the intestinal surface of tissue to form a thin biophysical barrier.We found that this molecular coating was able to stay on the surface of the intestine for long periods of time,effectively protecting the damaged intestinal epithelium from irritations of external intestinal metabolites and harmful flora.In addition,our results showed that this coating not only provided a beneficial environment for cell migration and proliferation to promote intestinal repair and regeneration,but also achieved a better outcome of IBD by reducing intestinal inflammation.Moreover,the in vivo experiments showed that the GelNB was better than the classic clinical medication-mesalazine.Therefore,our molecular coating showed potential as a promising strategy for the prevention and treatment of IBD.

关键词： Inflammatory bowel GelNB Molecular coating Biophysical barrier

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Dynamic photoelectrical regulation of ECM protein and cellular behaviors

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Bioactive Materials 2023年第4期22卷 168-179页

作者： Xiaozhao Wang Cai Yao Xudong Yao Junxin Lin Rui Li Kun Huang Weiming Lin Xiaojun Long Chao Dai Jiajun Dong Xuegong Yu a Wenwen Huang Wenjian Weng Qi Wang hongwei ouyang Kui Cheng School of Materials Science and Engineering State Key Laboratory of Silicon MaterialsCyrus Tang Center for Sensor Materials and ApplicationsZhejiang UniversityHangzhou310027China Dr.Li Dak Sum&Yip Yio Chin Center for Stem Cells and Regenerative Medicine Department of Orthopedic Surgery of the Second Affiliated HospitalZhejiang University School of MedicineHangzhou310058China Department of Sports Medicine Zhejiang University School of MedicineHangzhou310058China Zhejiang University-University of Edinburgh Institute Zhejiang University School of MedicineKey Laboratory of Tissue Engineering and Regenerative Medicine of Zhejiang ProvinceZhejiang University School of MedicineHangzhou314400China China Orthopedic Regenerative Medicine Group(CORMed) Hangzhou310058China School of Chemistry Zhejiang UniversityHangzhou310027China International Institutes of Medicine The Fourth Affiliated HospitalZhejiang University School of MedicineNo.N1Shangcheng AvenueYiwu322000China

Dynamic regulation of cell-extracellular matrix(ECM)-material interactions is crucial for various biomedical applications.In this study,a light-activated molecular switch for the modulation of cell attachment/detachment behaviors was established on monolayer graphene(Gr)/n-type Silicon substrates(Gr/Si).Initiated by light illumination at the Gr/Si interface,pre-adsorbed proteins(bovine serum albumin,ECM proteins collagen-1,and fibronectin)underwent protonation to achieve negative charge transfer to Gr films(n-doping)throughπ-πinteractions.This n-doping process stimulated the conformational switches of ECM proteins.The structural alterations in these ECM interactors significantly reduced the specificity of the cell surface receptor-ligand interaction(e.g.,integrin recognition),leading to dynamic regulation of cell adhesion and eventual cell detachment.RNA-sequencing results revealed that the detached bone marrow mesenchymal stromal cell sheets from the Gr/Si system manifested regulated immunoregulatory properties and enhanced osteogenic differentiation,implying their potential application in bone tissue regeneration.This work not only provides a fast and feasible method for controllable cells/cell sheets harvesting but also gives new insights into the understanding of cell-ECM-material communications.

关键词： Graphene Protein conformational change Charge transfer Cell-materials interaction n-doping

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H3K36 methyltransferase NSD1 regulates chondrocyte differentiation for skeletal development and fracture repair

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Bone Research 2021年第3期9卷 349-359页

作者： Rui Shao Zhong Zhang Zhan Xu Huiling ouyang Lijun Wang hongwei ouyang Matthew Greenblatt Xi Chen Weiguo Zou Shanghai Institute of Microsurgery on Extremities and Department of Orthopedic SurgeryShanghai Jiao Tong University Affiliated Sixth People’s HospitalShanghaiChina State Key Laboratory of Cell Biology Shanghai Institute of Biochemistry and Cell BiologyCAS Center for Excellence in Molecular Cell SciencesChinese Academy of SciencesUniversity of Chinese Academy of SciencesShanghaiChina Dr.Li Dak Sum&Yip Yio Chin Center for Stem Cell and Regenerative Medicine Zhejiang University-University of Edinburgh InstituteZhejiang UniversityHangzhouZhejiangChina China Orthopedic Regenerative Medicine Group HangzhouZhejiangChina Department of Pathology and Laboratory Medicine Weill Cornell MedicineNew YorkNYUSA Department of Molecular and Cellular Biology Lester and Sue Smith Breast CenterDan L.Duncan Cancer CenterBaylor College of MedicineHoustonTXUSA

Chondrocyte differentiation is a critical process for endochondral ossification,which is responsible for long bone development and fracture repair.Considerable progress has been made in understanding the transcriptional control of chondrocyte differentiation;however,epigenetic regulation of chondrocyte differentiation remains to be further studied.NSD1 is a H3K36(histone H3 at lysine 36)methyltransferase.Here,we showed that mice with Nsd1 deficiency in Prx1+mesenchymal progenitors but not in Col2+chondrocytes showed impaired skeletal growth and fracture healing accompanied by decreased chondrogenic differentiation.Via combined RNA sequencing(RNA-seq)and chromatin immunoprecipitation sequencing(ChIP-seq)analysis,we identified sex determining region Y box 9(Sox9),the key transcription factor of chondrogenic differentiation,as a functional target gene of NSD1.Mechanistically,NSD1 regulates Sox9 expression by modulating H3K36me1 and H3K36me2 levels in the Sox9 promoter region,constituting a novel epigenetic regulatory mechanism of chondrogenesis.Moreover,we found that NSD1 can directly activate the expression of hypoxia-inducible factor 1α(HIF1α),which plays a vital role in chondrogenic differentiation through its regulation of Sox9 expression.Collectively,the results of our study reveal crucial roles of NSD1 in regulating chondrogenic differentiation,skeletal growth,and fracture repair and expand our understanding of the function of epigenetic regulation in chondrogenesis and skeletal biology.

关键词： skeletal impaired healing

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大直径球罐内液体闪烁体和超纯水置换数值模拟

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核动力工程 2024年第2期45卷 225-230页

作者：赵宏伟董瑞李俊杰欧阳峥嵘衡月昆中国科学技术大学合肥230026 中国科学院合肥物质科学研究院合肥230031 中核第四研究设计工程有限公司石家庄050021 上海科技大学上海201210 中国科学院高能物理研究所北京100049

中微子实验装置一方面需要屏蔽大量的宇宙射线和来自岩石、空气、灰尘的天然放射性;另一方面需要尽可能降低实验设施自身的放射性本底。本文利用计算流体动力学(CFD)软件模拟研究了中心探测器(大直径球罐)内液体闪烁体(简称液闪)和超纯... 详细信息

中微子实验装置一方面需要屏蔽大量的宇宙射线和来自岩石、空气、灰尘的天然放射性;另一方面需要尽可能降低实验设施自身的放射性本底。本文利用计算流体动力学(CFD)软件模拟研究了中心探测器(大直径球罐)内液体闪烁体(简称液闪)和超纯水的置换过程,分析水中的放射性杂质进入液闪的情况,研究自然对流对置换过程中液闪本底的影响。通过模拟无自然对流和有自然对流2种工况,得到2种工况下液闪和水的相态与流态分布以及相含量随时间的变化情况,提取出相界面处竖直向上速度,并计算得出截面向上总流量,以判断自然对流对置换过程的影响。结果表明:在有自然对流工况下液闪向上的平均体积流量是小于无自然对流状态的,可知自然对流可以抑制水中放射性杂质进入液闪。

关键词：大直径球罐计算流体动力学(CFD) 置换自然对流

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