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Quenching and flow of charm and bottom quarks via semi-leptonic decay of D and B mesons in Pb+Pb collisions at the LHC

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Chinese Physics C 2024年第8期48卷 190-197页

作者： Shu-qing Li Wen-Jing Xing Shanshan Cao guang-you qin School of Physics and Electronic Engineering Jining UniversityQufu 273155China Institute of Frontier and Interdisciplinary Science Shandong UniversityQingdao 266237China Institute of Particle Physics and Key Laboratory of Quark and Lepton Physics(MOE) Central China Normal UniversityWuhan 430079China

Heavy flavor particles provide important probes of the microscopic structure and thermodynamic properties of the quark-gluon plasma(QGP)produced in high-energy nucleus-nucleus *** studied the energy loss and flow of charm and bottom quarks inside the QGP via the nuclear modification factor(R_(AA))and elliptic flow coefficient(v_(2))of their decayed leptons in heavy-ion collisions at the *** dynamical evolution of the QGP was performed using the CLVisc(3+1)-dimensional viscous hydrodynamics model;the evolution of heavy quarks inside the QGP was simulated with our improved Langevin model that considers both collisional and radiative energy loss of heavy quarks;the hadronization of heavy quarks was simulated via our hybrid coalescence-fragmentation model;and the semi-leptonic decay of D and B mesons was simulated via *** the same spatial diffusion coefficient for charm and bottom quarks,we obtained smaller R_(AA) and larger v_(2) of charm decayed leptons than bottom decayed leptons,indicating stronger energy loss of charm quarks than bottom quarks inside the QGP within our current model setup.

关键词： quark-gluon plasma heavy-ion collisions jet quenching anisotropic flow heavy quark heavy flavor muon

Heavy flavor quenching and flow:the roles of initial condition,pre-equilibrium evolution,and in-medium interaction

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Chinese Physics C 2020年第11期44卷 127-137页

作者： Shu-qing Li Wen-Jing Xing Feng-Lei Liu Shanshan Cao guang-you qin Department of Physics and Information Engineering Jining UniversityQufu 273155China Institute of Particle Physics and Key Laboratory of Quark and Lepton Physics(MOE) Central China Normal UniversityWuhan 430079China Cyclotron Institute Texas A&M UniversityCollege StationTexas77843USA Department of Physics and Astronomy Wayne State UniversityDetroitMichigan 48201USA Nuclear Science Division Lawrence Berkeley National LaboratoryBerkeleyCA 94720USA

Within an advanced Langevin-hydrodynamics framework coupled to a hybrid fragmentation-coalescence hadronization model,we study heavy flavor quenching and flow in relativistic heavy-ion *** investigate how the initial heavy quark spectrum,the in-medium energy loss and hadronization mechanisms of heavy quarks,the evolution profile of the pre-equilibrium stage,the medium flow,and the temperature dependence of heavy quark diffusion coefficients influence the suppression and elliptic flow of heavy mesons at the RHIC and the *** results show that the different modeling of initial conditions,pre-equilibrium evolution,and in-medium interactions can individually yield uncertainties of approximately 10-40% in D meson suppression and flow at a low transverse *** also find that proper combinations of collisional versus radiative energy loss,coalescence versus fragmentation in hadronization,and the inclusion of medium flow are the most important factors for describing the suppression and elliptic flow of heavy mesons.

关键词： quark-gluon plasma heavy-ion collisions jet quenching heavy quark

A discussion on the anomalous threshold enhancement of J/ψ–ψ(3770)couplings and X(6900)peak

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Chinese Physics C 2024年第4期48卷 1-5页

作者：陆业陈畅秦广友郑汉青 Institute of Particle Physics and Key Laboratory of Quark and Lepton Physics(MOE) Central China Normal UniversityWuhan 430079China Department of Physics Peking UniversityBeijing 100871China College of Physics Sichuan UniversityChengdu 610065China

The attractive interaction between J/ψandψ(3770)has to be strong enough if X(6900)is of the molecule *** argue that sinceψ(3770)decays predominantly into a DD^(¯)pair,the interactions between J/ψandψ(3770)may be significantly enhanced owing to the three point DD^(¯)loop *** enhancement originates from the anomalous threshold located at t=−1.288 GeV^(2),whose effect propagates into the s-channel partial wave amplitude in the vicinity of √s≃6.94 *** effect may be helpful in the formation of the X(6900)peak.

关键词： exotic hadrons hadron molecule X(6900)

Physics perspectives of heavy-ion collisions at very high energy

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Science China(Physics,Mechanics & Astronomy) 2016年第2期59卷 1-27页

作者： Ning-bo Chang ShanShan Cao Bao-yi Chen Shi-yong Chen Zhen-yu Chen Heng-Tong Ding Min He Zhi-quan Liu Long-gang Pang guang-you qin Ralf Rapp Bjorn Schenke Chun Shen HuiChao Song Hao-jie Xu Qun Wang Xin-Nian Wang Ben-wei Zhang Han-zhong Zhang XiangRong Zhu Peng-fei Zhuang Key Laboratory of Quark and Lepton Physics (MOE) and Institute of Particle Physics Central China Normal University Wuhan 430079 China Nuclear Science Division MS70R0319 Lawrence Berkeley National Laboratory Berkeley 94720 USA Physics Department Tsinghua University Beijing 100084 China Department of Applied Physics Nanjing University of Science and Technology Nanjing 210094 China Cyclotron Institute and Department of Physics and Astronomy Texas A&M University College Station TX 77843 USA Physics Department Brookhaven National Laboratory Upton New York 11973 USA Department of Physics McGill University Montreal Quebec H3A 2T8 Canada Department of Physics and State Key Laboratory of Nuclear Physics and Technology Peking University Beijing 100871 China Department of Modern Physics University of Science and Technology of China Hefei 230026 China

Heavy-ion collisions at very high colliding energies are expected to produce a quark-gluon plasma(QGP) at the highest temperature obtainable in a laboratory setting. Experimental studies of these reactions can provide an unprecedented range of information on properties of the QGP at high temperatures. We report theoretical investigations of the physics perspectives of heavy-ion collisions at a future high-energy collider. These include initial parton production, collective expansion of the dense medium, jet quenching,heavy-quark transport, dissociation and regeneration of quarkonia, photon and dilepton production. We illustrate the potential of future experimental studies of the initial particle production and formation of QGP at the highest temperature to provide constraints on properties of strongly interaction matter.

关键词： quark-gluon plasma heavy-ion collisions QCD phase transition properties of QGP

通过相对论重粒子碰撞中的J/ψ产生揭秘胶子喷注淬火机制

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Science Bulletin 2023年第18期68卷 2003-2009,M0003页

作者：张善良廖劲峰秦广友王恩科邢宏喜 Key Laboratory of Atomic and Subatomic Structure and Quantum Control(MOE) Institute of Quantum MatterSouth China Normal UniversityGuangzhou 510006China guangdong Provincial Key Laboratory of Nuclear Science Institute of Quantum MatterSouth China Normal UniversityGuangzhou 510006China guangdong-Hong Kong Joint Laboratory of Quantum Matter Southern Nuclear Science Computing CenterSouth China Normal UniversityGuangzhou 510006China Physics Department and Center for Exploration of Energy and Matter Indiana UniversityBloomington 47408USA Key Laboratory of Quark&Lepton Physics(MOE)and Institute of Particle Physics Central China Normal UniversityWuhan 430079China Southern Center for Nuclear-Science Theory(SCNT) Institute of Modern PhysicsChinese Academy of SciencesHuizhou 516000China

长久以来,喷注淬火被认为是探测重离子碰撞中形成的夺克胶子等离子体的关键信号之一,尽管过去在喷注火研究方面已经付出了巨大的努力、但是区分夸克和胶子的喷注淬火仍然是一个非常大的挑战本文发现大横动量J/主要由胶子碎裂产生,因此... 详细信息

长久以来,喷注淬火被认为是探测重离子碰撞中形成的夺克胶子等离子体的关键信号之一,尽管过去在喷注火研究方面已经付出了巨大的努力、但是区分夸克和胶子的喷注淬火仍然是一个非常大的挑战本文发现大横动量J/主要由胶子碎裂产生,因此为探测胶子在热密介质中的能量损失提供了一个独特的敏感探针,作者首先基于领头幂次的NRQCD因子化理论计算了质子-质子碰撞中的大横动量J/产生,发现胶子碎裂是大横动量J/产生的主要机制,然后通过线性玻尔兹曼输运模型和相对论流体力学模拟了核-核碰撞中喷注与热密介质的相互作用,所得数值结果很好地描述了大横动量J/的核修正因子和椭圆流,揭示出胶子能量损失是大横动量J/产额压低的主要机制。作者进一步通过数据驱动的贝叶斯分析方法证实了这个新颖的结论,并定量提取出夸克胶子等离子体中的胶子能量损失分布。

关键词： Relativistic heavy-ion collisions Heavy quarkonium Jet quenching Elliptic flow

Electrocatalytic cleavage of a lignin β-O-4 model compound and coupling with nitrogen-containing aromatics using Prussian blue analogue-derived nickel–cobalt spinel

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Journal of Energy Chemistry 2025年第1期100卷 628-636页

作者： Yi Qi Xueying Chen Bowen Liu Xuliang Lin Xueqing Qiu Yanlin qin guangdong Provincial Key Laboratory of Plant Resources Biorefinery School of Chemical Engineering and Light Industry Guangdong University of Technology guangdong Provincial Laboratory of Chemistry and Fine Chemical Engineering Jieyang Center guangdong Basic Research Center of Excellence for Ecological Security and Green Development Guangdong University of Technology

Electrochemical conversion of lignin for the production of high-value heterocyclic aromatic compounds has great *** demonstrate the targeted synthesis and cation modulation of NiCo2O4spinel nanoboxes,synthesized via cation exchange and calcination *** catalysts exhibit excellent efficacy in the electrocatalytic conversion of lignin model compounds,specifically 2-phenoxy-1-phenylethanol,into nitrogen-containing aromatics,achieving high conversion rates and *** catalysts were synthesized via a cation exchange and calcination oxidation process,using Prussian blue nanocubes as *** porous architecture and polymetallic composition of the NiCo2O4spinel demonstrated superior performance in electrocatalytic oxidative coupling,achieving a 99.2 wt%conversion rate of the 2-phenoxy-1-phenylethanol with selectivities of 37.5 wt%for quinoline derivatives and 31.5 wt%for *** innovations include the development of a sustainable one-pot synthesis method for quinoline derivatives,the elucidation of a multistage reaction pathway involving CAO bond cleavage,hydroxyaldol condensation,and CAN bond formation,and a deeper mechanistic understanding derived from DFT *** work establishes a new strategy for lignin valorization,offering a sustainable route to produce high-value nitrogen-containing aromatics from renewable biomass under mild conditions,without the need for additional reagents.

关键词： Spinel Biomass valorization Electrocatalysis Lignin oxidation Coupling

The emerging role of nitric oxide in the synaptic dysfunction of vascular dementia

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Neural Regeneration Research 2025年第2期20卷 402-415页

作者： Xiaorong Zhang Zhiying Chen Yinyi Xiong qin Zhou Ling-Qiang Zhu Dan Liu Department of Pathology Affiliated Hospital of Jiujiang UniversityJiujiangJiangxi ProvinceChina Jiujiang Clinical Precision Medicine Research Center JiujiangJiangxi ProvinceChina Center for Cognitive Science and Transdisciplinary Studies Jiujiang UniversityJiangxi ProvinceChina Department of Neurology Affiliated Hospital of Jiujiang UniversityJiujiangJiangxi ProvinceChina Department of Rehabilitation Affiliated Hospital of Jiujiang UniversityJiujiangJiangxi ProvinceChina Department of Pathophysiology School of Basic MedicineTongji Medical CollegeHuazhong University of Science and TechnologyWuhanHubei ProvinceChina

With an increase in global aging,the number of people affected by cerebrovascular diseases is also increasing,and the incidence of vascular dementia-closely related to cerebrovascular risk-is increasing at an epidemic ***,few therapeutic options exist that can markedly improve the cognitive impairment and prognosis of vascular dementia *** in Alzheimer’s disease and other neurological disorders,synaptic dysfunction is recognized as the main reason for cognitive *** oxide is one of the ubiquitous gaseous cellular messengers involved in multiple physiological and pathological processes of the central nervous ***,nitric oxide has been implicated in regulating synaptic plasticity and plays an important role in the pathogenesis of vascular *** review introduces in detail the emerging role of nitric oxide in physiological and pathological states of vascular dementia and summarizes the diverse effects of nitric oxide on different aspects of synaptic dysfunction,neuroinflammation,oxidative stress,and blood-brain barrier dysfunction that underlie the progress of vascular ***,we propose that targeting the nitric oxide-sGC-cGMP pathway using certain specific approaches may provide a novel therapeutic strategy for vascular dementia.

关键词： endoplasmic reticulum stress endothelial nitric oxide synthase gene therapy nitric oxide NO-sGC-cGMP pathway synaptic dysfunction vascular dementia

Enhancing m^(6)A modification in the motor cortex facilitates corticospinal tract remodeling after spinal cord injury

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Neural Regeneration Research 2025年第6期20卷 1749-1763页

作者： Tian qin Yuxin Jin Yiming qin Feifei Yuan Hongbin Lu Jianzhong Hu Yong Cao Chengjun Li Department of Spine Surgery and Orthopedics Xiangya HospitalCentral South UniversityChangshaHunan ProvinceChina Key Laboratory of Organ Injury Aging and Regenerative Medicine of Hunan ProvinceChangshaHunan ProvinceChina National Clinical Research Center for Geriatric Disorders Xiangya HospitalCentral South UniversityChangshaHunan ProvinceChina Department of Sports Medicine Xiangya HospitalCentral South UniversityChangshaHunan ProvinceChina

Spinal cord injury typically causes corticospinal tract disruption. Although the disrupted corticospinal tract can self-regenerate to a certain degree, the underlying mechanism of this process is still unclear. N6-methyladenosine(m^(6)A) modifications are the most common form of epigenetic regulation at the RNA level and play an essential role in biological processes. However, whether m^(6)A modifications participate in corticospinal tract regeneration after spinal cord injury remains unknown. We found that expression of methyltransferase 14 protein(METTL14) in the locomotor cortex was high after spinal cord injury and accompanied by elevated m^(6)A levels. Knockdown of Mettl14 in the locomotor cortex was not favorable for corticospinal tract regeneration and neurological recovery after spinal cord injury. Through bioinformatics analysis and methylated RNA immunoprecipitation-quantitative polymerase chain reaction, we found that METTL14 regulated Trib2 expression in an m^(6)A-regulated manner, thereby activating the mitogen-activated protein kinase pathway and promoting corticospinal tract regeneration. Finally, we administered syringin, a stabilizer of METTL14, using molecular docking. Results confirmed that syringin can promote corticospinal tract regeneration and facilitate neurological recovery by stabilizing METTL14. Findings from this study reveal that m^(6)A modification is involved in the regulation of corticospinal tract regeneration after spinal cord injury.

关键词： corticospinal tract remodeling epigenetic regulations locomotor cortex m^(6)A modification methyltransferase 14 protein(METTL14) mitogen-activated protein kinase neural regeneration spinal cord injury syringin TRIB2

Potential role of tanycyte-derived neurogenesis in Alzheimer's disease

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Neural Regeneration Research 2025年第6期20卷 1599-1612页

作者： Guibo Qi Han Tang Jianian Hu Siying Kang Song qin Department of Anatomy Histologyand EmbryologySchool of Basic Medical SciencesFudan UniversityShanghaiChina State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science Fudan UniversityShanghaiChina

Tanycytes, specialized ependymal cells located in the hypothalamus, play a crucial role in the generation of new neurons that contribute to the neural circuits responsible for regulating the systemic energy balance. The precise coordination of the gene networks controlling neurogenesis in naive and mature tanycytes is essential for maintaining homeostasis in adulthood. However, our understanding of the molecular mechanisms and signaling pathways that govern the proliferation and differentiation of tanycytes into neurons remains limited. This article aims to review the recent advancements in research into the mechanisms and functions of tanycyte-derived neurogenesis. Studies employing lineage-tracing techniques have revealed that the neurogenesis specifically originating from tanycytes in the hypothalamus has a compensatory role in neuronal loss and helps maintain energy homeostasis during metabolic diseases. Intriguingly,metabolic disorders are considered early biomarkers of Alzheimer's disease. Furthermore,the neurogenic potential of tanycytes and the state of newborn neurons derived from tanycytes heavily depend on the maintenance of mild microenvironments, which may be disrupted in Alzheimer's disease due to the impaired blood–brain barrier ***, the specific alterations and regulatory mechanisms governing tanycyte-derived neurogenesis in Alzheimer's disease remain unclear. Accumulating evidence suggests that tanycyte-derived neurogenesis might be impaired in Alzheimer's disease, exacerbating neurodegeneration. Confirming this hypothesis, however, poses a challenge because of the lack of long-term tracing and nucleus-specific analyses of newborn neurons in the hypothalamus of patients with Alzheimer's disease. Further research into the molecular mechanisms underlying tanycyte-derived neurogenesis holds promise for identifying small molecules capable of restoring tanycyte proliferation in neurodegenerative diseases. This line of investigation could pro

关键词： Alzheimer's disease blood–brain barrier ependymoglial cells hypothalamus metabolic diseases neural stem cells neurogenesis neuroinflammatory diseases neurons tanycyte