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A novel mitochondrial protein is required for cell wall integrity,auxin accumulation and root elongation in Arabidopsis under salt stress

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Stress Biology 2022年第1期2卷 530-544页

作者： Zheping Yu Yuying Ren Jianwei Liu Jian-Kang Zhu chunzhao zhao Institute of Horticulture Zhejiang Academy of Agricultural SciencesHangzhou 310021China Shanghai Center for Plant Stress Biology CAS Center for Excellence in Molecular Plant SciencesChinese Academy of SciencesShanghai 200032China

Maintenance of root elongation is beneficial for the growth and survival of plants under salt stress,but currently the cellular components involved in the regulation of root growth under high salinity are not fully understood.In this study,we identified an Arabidopsis mutant,rres1,which exhibited reduced root elongation under treatment of a variety of salts,including NaCl,NaNO3,KCl,and KNO3.RRES1 encodes a novel mitochondrial protein and its molecular function is still unknown.Under salt stress,the root meristem length was shorter in the rres1 mutant compared to the wild type,which was correlated with a reduced auxin accumulation in the mutant.Reactive oxygen species(ROS),as important signals that regulate root elongation,were accumulated to higher levels in the rres1 mutant than the wild type after salt treatment.Measurement of monosaccharides in the cell wall showed that arabinose and xylose contents were decreased in the rres1 mutant under salt stress,and application of boric acid,which is required for the crosslinking of pectic polysaccharide rhamnogalacturonan-II(RG-II),largely rescued the root growth arrest of the rres1 mutant,suggesting that RRES1 participates in the maintenance of cell wall integrity under salt stress.GUS staining assay indicated that the RRES1 gene was expressed in leaves and weakly in root tip under normal conditions,but its expression was dramatically increased in leaves and roots after salt treatment.Together,our study reveals a novel mitochondrial protein that regulates root elongation under salt stress via the modulation of cell wall integrity,auxin accumulation,and ROS homeostasis.

关键词： Salt stress Auxin Reactive oxygen species Plant cell wall Arabidopsis

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The MEKK1-MKK1/2-MPK4 cascade phosphorylates and stabilizes STOP1 to confer aluminum resistance in Arabidopsis

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Molecular Plant 2023年第2期16卷 337-353页

作者： Fanglin Zhou Somesh Singh Jie Zhang Qiu Fang Chongyang Li Jiawen Wang chunzhao zhao Pengcheng Wang Chao-Feng Huang National Key Laboratory of Plant Molecular Genetics Shanghai Center for Plant Stress BiologyCAS Center for Excellence in Molecular Plant SciencesShanghai Institute of Plant Physiology and EcologyChinese Academy of SciencesShanghai 200032China University of the Chinese Academy of Sciences Beijing 100049Cnina

Aluminum(Al)toxicity can seriously restrict crop production on acidic soils,which comprise 40%of the world’s potentially arable land.The zinc finger transcription factor STOP1 has a conserved and essential function in mediating plant Al resistance.Al stress induces STOP1 accumulation via post-transcriptional regulatory mechanisms.However,the upstream signaling pathway involved in Al-triggered STOP1 accumulation remains unclear.Here,we report that the MEKK1-MKK1/2-MPK4 cascade positively regulates STOP1 phosphorylation and stability.Mutations of MEKK1,MKK1/2,or MPK4 lead to decreased STOP1 stability and Al resistance.Al stress induces the kinase activity of MPK4,which interacts with and phosphorylates STOP1.The phosphorylation of STOP1 reduces its interaction with the F-box protein RAE1 that mediates STOP1 degradation,thereby leading to enhanced STOP1 stability and Al resistance.Taken together,our results suggest that the MEKK1-MKK1/2-MPK4 cascade is important for Al signaling and confers Al resistance through phosphorylation-mediated enhancement of STOP1 accumulation in Arabidopsis.

关键词： aluminum resistance Arabidopsis thaliana MAPK cascade MPK4 STOP1 phosphorylation STOP1 stability

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A Novel Mechanism Underlying the Coordination of Plant Growth and Salt Stress Response

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中国科学院院刊:英文版 2023年第2期37卷 121-122页

作者： chunzhao zhao CAS Center for Excellence in Molecular Plant Sciences(CEMPS) Chinese Academy of Sciences

Salt stress is a major environmental stress that threats crop growth and yield.It is of great significance to study the molecular genetic network of plants in response to salt stress and to cultivate salt-tolerant crop varieties for national food security.A study published in Nature Plants,Dr.zhao chunzhao’s group from the CAS Center for Excellence in Molecular Plant Sciences(CEMPS),Chinese Academy of Science,uncovers a novel mechanism underlying the coordination of plant growth and salt tolerance in plants.

关键词： Plant stress network

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The transcription factor ICE1 functions in cold stress response by binding to the promoters of CBF and COR genes

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Journal of Integrative Plant Biology 2020年第3期62卷 258-263页

作者： Kai Tang Lun zhao Yuying Ren Shuhua Yang Jian-Kang Zhu chunzhao zhao Shanghai Center for Plant Stress Biology and CAS Center for Excellence in Molecular Plant Sciences Chinese Academy of SciencesShanghai 200032China Department of Horticulture and Landscape Architecture Purdue UniversityWest LafayetteIN 47907USA University of the Chinese Academy of Sciences Beijing 100049China State Key Laboratory of Plant Physiology and Biochemistry College of Biological SciencesChina Agricultural UniversityBeijing 100193China State Key Laboratory of Crop Stress Adaptation and Improvement School of Life SciencesHenan UniversityKaifeng 475004China

A recent paper by Kidokoro et al.(2020)in The Plant Cell reported a transgene-dependent transcriptional silencing phenomenon in the dominant ice1-1 Arabidopsis mutant containing the CBF3-LUC reporter,and questioned whether ICE1 may regulate CBF genes and may be involved in plant cold response.Here,we evaluate available evidence supporting the involvement of ICE1 in plant cold response,and provide ChIP-seq data showing ICE1 binding to the promoters of CBF genes and other regulatory genes known to be critical for cold response as well as to the promoters of some COR genes.

关键词： ICE1 CBF stress

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The LRXs-RALFs-FER module controls plant growth and salt stress responses by modulating multiple plant hormones

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National Science Review 2021年第1期8卷 40-55页

作者： chunzhao zhao Wei Jiang Omar Zayed Xin Liu Kai Tang Wenfeng Nie Yali Li Shaojun Xie Yuan Li Tiandan Long Linlin Liu Yingfang Zhu Yang zhao Jian-Kang Zhu Shanghai Center for Plant Stress Biology and Center of Excellence in Molecular Plant Sciences Chinese Academy of Sciences Department of Horticulture and Landscape Architecture Purdue University Key laboratory of Plant Stress Biology School of Life SciencesHenan University National Center for Soybean Improvement Key Laboratory of Biology and Genetics and Breeding for Soybean Ministry of Agriculture State Key Laboratory of Crop Genetics and Germplasm Enhancement Nanjing Agricultural University Genetics Department Faculty of AgricultureMenofia University University of the Chinese Academy of Sciences State Key Laboratory of Plant Physiology and Biochemistry College of Biological SciencesChina Agricultural University State Key Laboratory of Crop Gene Exploration and Utilization in Southwest China Sichuan Agricultural University

Salt stress is a major environmental factor limiting plant growth and productivity. We recently discovered an important new salt tolerance pathway, where the cell wall leucine-rich repeat extensins LRX3/4/5, the RAPID ALKALINIZATION FACTOR (RALF) peptides RALF22/23 and receptor-like kinase FERONIA(FER) function as a module to simultaneously regulate plant growth and salt stress tolerance. However, the intracellular signaling pathways that are regulated by the extracellular LRX3/4/5-RALF22/23-FER module to coordinate growth, cell wall integrity and salt stress responses are still unknown. Here, we report that the LRX3/4/5-RALF22/23-FER module negatively regulates the levels of jasmonic acid (JA), salicylic acid(SA) and abscisic acid (ABA). Blocking JA pathway rescues the dwarf phenotype of the lrx345 and fer-4mutants, while disruption of ABA biosynthesis suppresses the salt-hypersensitivity of these mutants. Many salt stress-responsive genes display abnormal expression patterns in the lrx345 and fer-4 mutants, as well as in the wild type plants treated with epigallocatechin gallate (EGCG), an inhibitor of pectin methylesterases,suggesting cell wall integrity as a critical factor that determines the expression pattern of stress-responsive genes. Production of reactive oxygen species (ROS) is constitutively increased in the lrx345 and fer-4mutants, and inhibition of ROS accumulation suppresses the salt-hypersensitivity of these mutants.Together, our work provides strong evidence that the LRX3/4/5-RALF22/23-FER module controls plant growth and salt stress responses by regulating hormonal homeostasis and ROS accumulation.

关键词： salt stress cell wall integrity growth LRX RALF FER hormone

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纳米递药系统负载奥希替尼抗非小细胞肺癌的研究进展

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肿瘤防治研究 2024年第2期51卷 134-139页

作者：刘汝贵赵瑞瑞刘春朝武晓青岛大学医学部青岛266075 青岛大学附属青岛市中心医院青岛市肿瘤医院呼吸与危重症医学科青岛266042 青岛大学材料科学与工程学院青岛266071

奥希替尼是不可逆的第三代表皮生长因子受体酪氨酸激酶抑制剂(EGFR-TKI),用于治疗经典EGFR突变和T790M耐药突变的非小细胞肺癌(NSCLC)。然而,与其他EGFR-TKIs一样,奥希替尼不可避免地存在获得性耐药、水溶性差、肿瘤累积率低等问题,限... 详细信息

奥希替尼是不可逆的第三代表皮生长因子受体酪氨酸激酶抑制剂(EGFR-TKI),用于治疗经典EGFR突变和T790M耐药突变的非小细胞肺癌(NSCLC)。然而,与其他EGFR-TKIs一样,奥希替尼不可避免地存在获得性耐药、水溶性差、肿瘤累积率低等问题,限制了其治疗效果。纳米递药系统可增加药物的溶解度和稳定性,延长药物血液循环时间,提高细胞摄取率,增加在肿瘤组织中的聚集改善药物耐药问题,已成为解决传统靶向药物弊端的有效手段。本文综述了第三代EGFR-TKI奥希替尼的作用机制,重点阐述了奥希替尼纳米递药系统抗NSCLC的研究进展,并对该领域面临的挑战和未来发展方向进行了展望。

关键词：非小细胞肺癌奥希替尼纳米递药系统

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SWO1 modulates cell wall integrity under salt stress by interacting with importinɑin Arabidopsis

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Stress Biology 2021年第1期1卷 98-119页

作者： Zhidan Wang Mugui Wang Changhong Yang Lun zhao Guochen Qin Li Peng Qijie Zheng Wenfeng Nie Chun-Peng Song Huazhong Shi Jian-Kang Zhu chunzhao zhao Shanghai Center for Plant Stress Biology CAS Center for Excellence in Molecular Plant SciencesChinese Academy of SciencesShanghai 200032China University of Chinese Academy of Sciences Beijing 100049China National Key Laboratory of Crop Genetic Improvement Huazhong Agricultural UniversityWuhan 430070China Department of Horticulture College of Horticulture and Plant ProtectionYangzhou UniversityYangzhou 225009China Key Laboratory of Plant Stress Biology School of Life SciencesHenan UniversityKaifeng 475001China Department of Chemistry and Biochemistry Texas Tech UniversityLubbockTX 79409USA

Maintenance of cell wall integrity is of great importance not only for plant growth and development,but also for the adaptation of plants to adverse environments.However,how the cell wall integrity is modulated under salt stress is still poorly understood.Here,we report that a nuclear-localized Agenet domain-containing protein SWO1(SWOLLEN 1)is required for the maintenance of cell wall integrity in Arabidopsis under salt stress.Mutation in SWO1 gene results in swollen root tips,disordered root cell morphology,and root elongation inhibition under salt stress.The swo1 mutant accumulates less cellulose and pectin but more lignin under high salinity.RNA-seq and ChIP-seq assays reveal that SWO1 binds to the promoter of several cell wall-related genes and regulates their expression under saline conditions.Further study indicates that SWO1 interacts with importinɑIMPA1 and IMPA2,which are required for the import of nuclear-localized proteins.The impa1 impa2 double mutant also exhibits root growth inhibition under salt stress and mutations of these two genes aggravate the salt-hypersensitive phenotype of the swo1 mutant.Taken together,our data suggest that SWO1 functions together with importinɑto regulate the expression of cell wall-related genes,which enables plants to maintain cell wall integrity under high salinity.

关键词： Agenet domain Salt stress Importins Plant cell wall Arabidopsis

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植物非生物逆境学科发展综述

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植物生理学报 2024年第2期60卷 248-270页

作者：赵杨杨永青丁杨林张蘅谢彦杰赵春钊刘林川王鹏程中国科学院分子植物科学卓越创新中心/上海植物逆境生物学研究中心上海200032 中国农业大学生物学院北京100193 上海交通大学生命科学技术学院上海200240 南京农业大学生命科学学院南京210095 华南农业大学林学与风景园林学院广州510642 南方科技大学前沿生物技术研究院广东深圳518055

干旱、盐碱和极端温度等非生物胁迫严重抑制植物的生长和发育,是影响农业生产的主要环境因素。植物应答环境胁迫的分子机制一直是植物生物学的热点研究领域。本文邀请在非生物胁迫应答分子机制领域非常活跃的青年科学家,简要综述相关研... 详细信息

干旱、盐碱和极端温度等非生物胁迫严重抑制植物的生长和发育,是影响农业生产的主要环境因素。植物应答环境胁迫的分子机制一直是植物生物学的热点研究领域。本文邀请在非生物胁迫应答分子机制领域非常活跃的青年科学家,简要综述相关研究领域近年来的主要研究进展,并展望未来这一领域值得深入研究的一些科学问题,希望能引起对这一领域的更多的关注和思考。

关键词：非生物胁迫感受信号转导分子机制

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Mechanisms of Plant Responses and Adaptation to Soil Salinity

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The Innovation 2020年第1期1卷 69-109页

作者： chunzhao zhao Heng Zhang Chunpeng Song Jian-Kang Zhu Sergey Shabala Shanghai Center for Plant Stress Biology and CAS Center for Excellence in Molecular Plant Sciences Chinese Academy of SciencesShanghai 200032China State Key Laboratory of Crop Stress Adaptation and Improvement School of Life SciencesHenan UniversityKaifeng 475004China State Key Laboratory of Plant Molecular Genetics Shanghai Center for Plant Stress BiologyCenter of Excellence in Molecular Plant SciencesChinese Academy of SciencesShanghai 200032China Department of Horticulture and Landscape Architecture Purdue UniversityWest LafayetteIN 47907USA International Research Centre for Environmental Membrane Biology Foshan UniversityFoshan 528000China Tasmanian Institute of Agriculture University of TasmaniaHobartTAS 7001Australia

Soil salinity is a major environmental stress that restricts the growth and yield of crops.Understanding the physiological,metabolic,and biochemical responses of plants to salt stress and mining the salt tolerance-associated genetic resource in nature will be extremely important for us to cultivate salt-tolerant crops.In this review,we provide a comprehensive summary of the mechanisms of salt stress responses in plants,including salt stress-triggered physiological responses,oxidative stress,salt stress sensing and signaling pathways,organellar stress,ion homeostasis,hormonal and gene expression regulation,metabolic changes,as well as salt tolerance mechanisms in halophytes.Important questions regarding salt tolerance that need to be addressed in the future are discussed.

关键词： stress Plant salinity

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Publisher Correction:A novel mitochondrial protein is required for cell wall integrity,auxin accumulation and root elongation in Arabidopsis under salt stress

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Stress Biology 2022年第1期2卷 46-46页

作者： Zheping Yu Yuying Ren Jianwei Liu Jian‑Kang Zhu chunzhao zhao Institute of Horticulture Zhejiang Academy of Agricultural SciencesHangzhou 310021China Shanghai Center for Plant Stress Biology CAS Center for Excellence in Molecular Plant SciencesChinese Academy of SciencesShanghai 200032China

Correction:Stress Biol 2,13(2022)https://***/10.1007/s44154-022-00036-3 Following publication of this article(Yu et al.2022),it is noticed that the section‘Materials and methods’was placed in a wrong place in the ma... 详细信息

关键词： Arabidopsis stress auxin

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