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Expression Pattern of the Hippo Pathway Effector TAZ in Cellular and Fibrotic Nonspecific Interstitial Pneumonia

Chinese Medical Journal 2018年第5期131卷 626-628页

作者： Min-Kyung Yeo Hee Sun Park Yeon Hee Park Choong-Sik Lee Geon Yoo Dong II Park Jeong Eun Lee Jae Young Moon Sung Soo Jung Ju Ock Kim Dahyun Kang Hyun Jin Cho Min-Woong Kang Jin-Whan Kim Song-Soo Kim chaeuk chung Department of Pathology Chungnam National University School of Medicine Daejeon 35015 South Korea Division of Pulmonology Department of Internal Medicine Chungnam National University 282 Munhwaro Jung-gu Daejeon 35015 South Korea Department of Internal Medicine Daejeonst Mary's Hospital College of Medicine Catholic University Daejeon 34943 South Korea ~Department of Toxicological Evaluation and Research Korea Institute of Toxicology 141 Gajeong-ro Yuseong-gu Daejeon 34114 South Korea Department of Thoracic Surgery College of Medicine Chungnam National University Daejeon 35015 South Korea Department of Radiology College of Medicine Chungnam National University Daejeon 35015 South Korea Infection Control Convergence Research Center College of Medicine Chungnam National University Jung-guMunhwa-ro 266 Daejeon 35015 South Korea

Interstitial lung disease （ILD） is a comprehensive term referring to a group of lung diseases affecting the interstitium of the lung. Idiopathic pulmonary fibrosis （IPF） is the most common idiopathic ILD, and nonspecific interstitial pneumonia （NSIP） is the second most common. As the name suggests, NSIP is diagnosed atter many other diseases are excluded. The main pathological finding in NSIP is homogeneous interstitial inflammation with or without fibrosis. NSIP can be categorized by cellular type or fibrotic type, according to the grade of inflammation and fibrosis. The cellular type has mostly inflammatory lesions with good responses to steroid, but the fibrotic type has a large proportion of fibrosis mixed with inflammatory lesions and a relatively poor response to steroid treatment So far, the exact mechanism underlying idiopathic lED has not been clarified. Determining key regulators of these ILDs will be helpful in the diagnosis and development of novel drugs for ILD.

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