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国际医学放射学杂志 2017年第1期 100-100页

作者： Y.Tanabe T.Kido T.Uetani A.Kurata T.Kono a.ogimoto 于静 Department of Radiology Ehime University Graduate School of Medicine

摘要目的通过与心脏MR(CMR)和SPECT比较,评估动态CT灌注成像参数心肌血流量发现心肌缺血与梗死的可行性。方法回顾性纳入经负荷动态CT灌注检查及SPECT

关键词： SPECT 特异度敏感度心肌缺血 CT 心肌灌注灌注成像 MR 心脏

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Analysis of value of flexibility in Japan’s power system with increased VRE

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Clean Energy 2018年第1期2卷 37-44页

作者： Sumie Nakayama Hitoshi Azuma Suguru Fukutome Kazuhiko ogimoto J-POWER/Electric Power Development Co. Ltd.6-15-1GinzaChuo-kuTokyo 104-8165Japan JP Business Service Corporation 2-2-18FukagawaKoto-kuTokyo 135-8451Japan Institute of Industrial Science Collaborative Research Center for Energy EngineeringThe University of Tokyo4-6-1KomabaMeguro-kuTokyo153-8585Japan

The growth of renewable energy has accelerated globally toward a low-carbon economy since the Paris Agreement entered into force in *** a result of the increase of variable renewable energy(VRE),namely solar PV and wind,power systems require more flexibility from conventional power plants with less power generation to regulate increased *** are sources of flexibility other than conventional power plants,including enhanced power networks,storage capacity and demand *** maximize economic utilization of VRE power generation,it is necessary to use the flexibility potential from all these *** Japan,the share of VRE has increased since the introduction of a feed-in tariff(FIT)and,in parallel,power market reform is *** has a unique power system of nine grids connected like a fish bone,making the uptake of an increasing share of VRE *** paper assesses the value of flexibility by source in Japan’s power system in *** analysis of different VRE scenarios is undertaken based on a newly developed production cost *** result of the simulation shows the quantitative impact of each source of flexibility to the generation cost and VRE curtailment and demonstrates the mechanism by which flexibility works to impact VRE curtailment.

关键词： component power demand and supply renewable energy flexibility Japan’s power system produc-tion cost model analysis

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Androgen receptor deficiency in monocytes/ macrophages does not alter adiposity or glucose homeostasis in male mice

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Asian Journal of Andrology 2018年第3期20卷 276-283页

作者： Katya B Rubinow Barbara Houston Shari Wang Leela Goodspeed Kayoko ogimoto Gregory J Morton Christopher McCarty Robert E Braun Stephanie T Page Division of Metabolism Endocrinology and Nutrition Department of Medicine University of Washington School of Medicine Seattle WA 98195 USA Jackson Laboratory Bar Harbor ME 04609 USA

Androgen deprivation in men leads to increased adiposity, but the mechanisms underlying androgen regulation of fat mass have not been fully defined. Androgen receptor （AR） is expressed in monocytes/macrophages, which are resident in key metabolic tissues and influence energy metabolism in surrounding cells. Male mice bearing a cell-specific knockout of the AR in monocytes/macrophages （M-ARKO） were generated to determine whether selective loss of androgen signaling in these cells would lead to altered body composition. Wild-type （WT） and M-ARKO mice （12-22 weeks of age, n = 12 per group） were maintained on a regular chow diet for 8 weeks and then switched to a high-fat diet for 8 additional weeks. At baseline and on both the regular chow and high-fat diets, no differences in lean mass or fat mass were observed between groups. Consistent with the absence of differential body weight or adiposity, no differences in food intake （3.0 ± 0.5 g per day for WT mice vs 2.8 ± 0.4 g per day for M-ARKO mice） or total energy expenditure （0.6 ± 0.1 Kcal h-1 for WT mice vs 0.5 ± 0.1 Kcal h-1 for M-ARKO mice） were evident between groups during high-fat feeding. Liver weight was greater in M-ARKO than that in WT mice （1.5 ± 0.1 g vs 1.3 ± 0.0 g, respectively, P = 0.02）. Finally, M-ARKO mice did not exhibit impairments in glucose tolerance or insulin sensitivity relative to WT mice at any study time point. In aggregate, these findings suggest that AR signaling specifically in monocytes/macrophages does not contribute to the regulation of systemic energy balance, adiposity, or insulin sensitivity in male mice.

关键词： androgen receptor knockout mice macrophages male hypogonadism metabolic syndrome

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