A conserved oomycete effector RxLR23 triggers plant defense responses by targeting ERD15La to release NbNAC68
作者单位：Shandong Agricultural UniversityCollege of Plant ProtectionShandong Provincial Key Laboratory for Biology of Vegetable Diseases and Insect Pests Shandong Agricultural UniversityCollege of Life SciencesState Key Laboratory of Crop Biology Shandong Normal UniversityCollege of Life Sciences Northeast Agricultural UniversityDepartment of Horticulture China Agricultural UniversityCollege of Plant Protection Nanjing Agricultural UniversityDepartment of Plant Pathology Bowling Green State UniversityDepartment of Biological Sciences
会议名称：《中国菌物学会2023年学术年会》
会议日期：2023年
学科分类：09[农学] 0904[农学-植物保护] 090401[农学-植物病理学]
摘 要：Oomycete pathogens deliver many effectors to enhance virulence or suppress plant *** immune networks are interconnected,in which a few effectors can trigger a strong defense response when recognized by immunity-related *** effectors activate plant defense response remains poorly *** we report Phytophthora capsici effector RxLR23 can induce plant cell death and plant ***23 specifically binds to NbERD15La,a regulator of abscisic acid and salicylic acid pathway,and the binding intensity depends on the amino acid residues(K93and M320).NbNAC68,a downstream protein of ERD15La,can stimulate plant immunity that is compromised after binding with *** of NbNAC68substantially prevents the activation of plant defense ***23 binds to ERD15La,releasing NbNAC68 to activate plant immunity These findings highlight the importance of RxLR23activity as a strategy of plant defense responses.