Suppression of key molecules involved in the thrombogenic mechanisms of antiphospholipid syndrome by an NF-kB inhibitor
作者单位：Tokyo Medical and Dental UniversityTokyoJapan Keio UniversityYokohamaJapan Tokyo Medical and Dental UniversityTokyoJapan Keio UniversityYokohamaJapan Tokyo Medical and Dental UniversityTokyoJapan Keio UniversityYokohamaJapan Tokyo Medical and Dental UniversityTokyoJapan Keio UniversityYokohamaJapan Tokyo Medical and Dental UniversityTokyoJapan Keio UniversityYokohamaJapan Tokyo Medical and Dental UniversityTokyoJapan Keio UniversityYokohamaJapan
会议名称：《The 8th International Congress on SLE》
会议日期：2007年
学科分类：1002[医学-临床医学] 100201[医学-内科学(含：心血管病、血液病、呼吸系病、消化系病、内分泌与代谢病、肾病、风湿病、传染病)] 10[医学]
摘 要：正Purpose:Fractalkine（CX3CL1）is mainly expressed on activated vascular endothelial cells and plays a role as one of the adhesion molecules that leads to leukocyte *** addition,it is cleaved by a protease and works as a soluble chemokine to mediate ***,human