Neurochemical plasticity of Müller cells after retinal injury: overexpression of GAT-3 may potentiate excitotoxicity
Neurochemical plasticity of Müller cells after retinal injury: overexpression of GAT-3 may potentiate excitotoxicity作者机构:Laboratório de Neuroquímica Instituto de Biofísica Carlos Chagas Filho Universidade Federal do Rio de Janeiro Laboratório de Neuroanatomia Celular Instituto de Biofísica Carlos Chagas Filho Universidade Federal do Rio de Janeiro
出 版 物:《Neural Regeneration Research》 (中国神经再生研究(英文版))
年 卷 期:2015年第10卷第9期
页 面:1376-1378页
核心收录:
学科分类:0710[理学-生物学] 1002[医学-临床医学] 1001[医学-基础医学(可授医学、理学学位)] 100204[医学-神经病学] 10[医学]
基 金:supported by grants from FAPERJ CNPq(INCT-INNT) CAPES and PROLAB LARC/IBRO/CNPq
主 题:GAT Neurochemical plasticity of M ller cells after retinal injury overexpression of GAT-3 may potentiate excitotoxicity RGCs
摘 要:The retina is a multilayered tissue that develops following a central-to-peripheral gradient. Its structure derives from multipotent precursors, as shown through clonal analysis of retinal cell lineage. These progenitors generate diverse cell types, controlled by complex influences of intrinsic and extrinsic factors (Hatakevama and Kagevama, 2004).