Direct Modulation of the Guard Cell Outward- Rectifying Potassium Channel （CORK） by Abscisic Acid

作     者：Amanda Ooi Fouad Lemtiri-Chfieh Aloysius Wong Christoph Gehring 

作者机构：Division of Biological and Environmental Sciences and Engineering 4700 King Abdullah University of Science and Technology Thuwa123955-6900 Kingdom of Saudi Arabia 

出 版 物：《Molecular Plant》 (分子植物（英文版）)

年 卷 期：2017年第10卷第11期

页      面：1469-1472页

核心收录：

学科分类：0710[理学-生物学] 07[理学] 071009[理学-细胞生物学] 09[农学] 0901[农学-作物学] 090102[农学-作物遗传育种] 

基　　金：supported by King Abdullah University of Science and Technology (KAUST) 

主　　题：Direct Modulation of the Guard Cell Outward- Rectifying Potassium Channel (CORK) Abscisic Acid 

摘      要：Dear Editor, Abscisic acid （ABA） induces turgot loss and hence stomatal closure by promoting rapid net K^＋ efflux from guard cells （GCs） through outward-rectifying K^＋ （K^＋out） channels （Schroeder et al., 1987; Blatt, 1990）. The mechanisms of ABA signaling in GCs are detailed elsewhere （see Munemasa et al., 2015; Weiner et al., 2010; Pandey et al., 2007）. Briefly, ABA binds to the PYR/ PYL/RCARs, a family of soluble steroidogenic acute regulatory- related lipid transfer （START） proteins, and, in turn, inactivates the downstream PP2C （type 2C protein phosphatase）, leading to the activation of SnRK2.6 （SNF1 [sucrose non-fermenting-1- related protein kinase]/OST1 [open stomata 1]） protein kinases.