p38-MAPK and CDK5,signaling pathways in neuroinflammation:a potential therapeutic intervention in Alzheimer's disease?

作     者：Vlad Ionut Viorel Ylenia Pastorello Nosherwan Bajwa Mark Slevin Vlad Ionut Viorel;Ylenia Pastorello;Nosherwan Bajwa;Mark Slevin

作者机构：George Emil Palade University of MedicinePharmacyScience and TechnologyTârgu MuresRomania Department of Anatomy and EmbryologyGeorge Emil Palade University of MedicinePharmacyScience and TechnologyTargu MuresRomania DIAKO HospitalFlensburgGermany Center for Advanced Medical and Pharmaceutical Research(CCAMF)George Emil Palade University of MedicinePharmacyScience and TechnologyTârgu MuresRomania Manchester Metropolitan UniversityManchesterUK 

出 版 物：《Neural Regeneration Research》 (中国神经再生研究（英文版）)

年 卷 期：2024年第19卷第8期

页      面：1649-1650页

核心收录：

学科分类：0710[理学-生物学] 1002[医学-临床医学] 1001[医学-基础医学(可授医学、理学学位)] 100203[医学-老年医学] 10[医学] 

主　　题：Alzheimer Aβ tau 

摘      要：Alzheimer’s disease(AD),the most common type of dementia,affects millions of people worldwide,putting a significant strain on healthcare infrastructure and societal *** is characte rized by the build-up of amyloid-beta(Aβ)plaques and neurofibrillary to ngles containing hyperphosphorylated tau protein.